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Artigo de periodico
Remote vs. face-to-face activities in the teaching of renal pathophysiology in the context of social isolation during the early phase of the COVID-19 pandemic (2023)
Haydar, Ahmed; Santos, Itamar de Souza ; Arcon, Luis Carlos; Martins, Milton de Arruda ; Tempski, Patricia Zen; Zatz, Roberto
Source: Advances in physiology education. Unidade: FM
Subjects: COVID-19, PANDEMIAS, EDUCAÇÃO A DISTÂNCIA, ENSINO E APRENDIZAGEM
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ABNT
HAYDAR, Ahmed et al. Remote vs. face-to-face activities in the teaching of renal pathophysiology in the context of social isolation during the early phase of the COVID-19 pandemic. Advances in physiology education, v. 47, n. 4, p. 788-795, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/57844. Acesso em: 15 nov. 2024.
APA
Haydar, A., Santos, I. de S., Arcon, L. C., Martins, M. de A., Tempski, P. Z., & Zatz, R. (2023). Remote vs. face-to-face activities in the teaching of renal pathophysiology in the context of social isolation during the early phase of the COVID-19 pandemic. Advances in physiology education, 47( 4), 788-795. doi:10.1152/advan.00257.2022
NLM
Haydar A, Santos I de S, Arcon LC, Martins M de A, Tempski PZ, Zatz R. Remote vs. face-to-face activities in the teaching of renal pathophysiology in the context of social isolation during the early phase of the COVID-19 pandemic [Internet]. Advances in physiology education. 2023 ; 47( 4): 788-795.[citado 2024 nov. 15 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57844
Vancouver
Haydar A, Santos I de S, Arcon LC, Martins M de A, Tempski PZ, Zatz R. Remote vs. face-to-face activities in the teaching of renal pathophysiology in the context of social isolation during the early phase of the COVID-19 pandemic [Internet]. Advances in physiology education. 2023 ; 47( 4): 788-795.[citado 2024 nov. 15 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57844
Artigo de periodico
Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models (2023)
Zambom, Fernanda Florencia Fregnan; Albino, Amanda Helen; Tessaro, Helena Mendonca; Foresto-Neto, Orestes; Malheiros, Denise Maria Avancini Costa ; Camara, Niels Olsen Saraiva; Zatz, Roberto
Source: American journal of physiology-renal physiology. Unidade: FM
Subjects: NEFROPATIAS, DOR CRÔNICA, RIM, MODELOS ANIMAIS, ESTRESSE OXIDATIVO
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ABNT
ZAMBOM, Fernanda Florencia Fregnan et al. Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models. American journal of physiology-renal physiology, v. 325, n. 3, p. F283-F298, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/56184. Acesso em: 15 nov. 2024.
APA
Zambom, F. F. F., Albino, A. H., Tessaro, H. M., Foresto-Neto, O., Malheiros, D. M. A. C., Camara, N. O. S., & Zatz, R. (2023). Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models. American journal of physiology-renal physiology, 325( 3), F283-F298. doi:10.1152/ajprenal.00200.2022
NLM
Zambom FFF, Albino AH, Tessaro HM, Foresto-Neto O, Malheiros DMAC, Camara NOS, Zatz R. Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models [Internet]. American journal of physiology-renal physiology. 2023 ; 325( 3): F283-F298.[citado 2024 nov. 15 ] Available from: https://observatorio.fm.usp.br/handle/OPI/56184
Vancouver
Zambom FFF, Albino AH, Tessaro HM, Foresto-Neto O, Malheiros DMAC, Camara NOS, Zatz R. Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models [Internet]. American journal of physiology-renal physiology. 2023 ; 325( 3): F283-F298.[citado 2024 nov. 15 ] Available from: https://observatorio.fm.usp.br/handle/OPI/56184
Artigo de periodico
Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload (2019)
Zambom, Fernanda Florencia Fregnan; Oliveira, Karin Carneiro; Foresto-Neto, Orestes; Faustino, Viviane Dias; Avila, Victor Ferreira; Albino, Amanda Helen; Arias, Simone Costa Alarcon; Malheiros, Denise Maria Avancini Costa ; Zatz, Roberto ; Fujihara, Clarice Kazue
Source: American journal of physiology-renal physiology. Unidade: FM
Subjects: IMUNIDADE NATURAL, INSUFICIÊNCIA RENAL CRÔNICA, HIPERTENSÃO, ÓXIDO NÍTRICO, SAL (CONDIMENTO), INFLAMAÇÃO, IMUNOHISTOQUÍMICA, RATOS WISTAR
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ABNT
ZAMBOM, Fernanda Florencia Fregnan et al. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload. American journal of physiology-renal physiology, v. 317, n. 4, p. F1058-F1067, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00251.2019. Acesso em: 15 nov. 2024.
APA
Zambom, F. F. F., Oliveira, K. C., Foresto-Neto, O., Faustino, V. D., Avila, V. F., Albino, A. H., et al. (2019). Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload. American journal of physiology-renal physiology, 317( 4), F1058-F1067. doi:10.1152/ajprenal.00251.2019
NLM
Zambom FFF, Oliveira KC, Foresto-Neto O, Faustino VD, Avila VF, Albino AH, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload [Internet]. American journal of physiology-renal physiology. 2019 ; 317( 4): F1058-F1067.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00251.2019
Vancouver
Zambom FFF, Oliveira KC, Foresto-Neto O, Faustino VD, Avila VF, Albino AH, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload [Internet]. American journal of physiology-renal physiology. 2019 ; 317( 4): F1058-F1067.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00251.2019
Artigo de periodico
NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model (2018)
Foresto-neto, Orestes; Malheiros, Denise Maria Avancini Costa; Zatz, Roberto; Fujihara, Clarice Kazue
Source: Laboratory investigation. Unidade: FM
Subjects: NEFROPATIAS, RATOS WISTAR, INIBIDORES DE ENZIMAS, ESTRESSE OXIDATIVO
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ABNT
FORESTO-NETO, Orestes et al. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model. Laboratory investigation, v. 98, n. 6, p. 773-782, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41374-018-0029-4. Acesso em: 15 nov. 2024.
APA
Foresto-neto, O., Malheiros, D. M. A. C., Zatz, R., & Fujihara, C. K. (2018). NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model. Laboratory investigation, 98( 6), 773-782. doi:10.1038/s41374-018-0029-4
NLM
Foresto-neto O, Malheiros DMAC, Zatz R, Fujihara CK. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model [Internet]. Laboratory investigation. 2018 ; 98( 6): 773-782.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41374-018-0029-4
Vancouver
Foresto-neto O, Malheiros DMAC, Zatz R, Fujihara CK. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model [Internet]. Laboratory investigation. 2018 ; 98( 6): 773-782.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41374-018-0029-4
Artigo de periodico
1,5-Anhydroglucitol predicts CKD progression in macroalbuminuric diabetic kidney disease: results from non-targeted metabolomics (2018)
Tavares, Gesiane; Venturini, Gabriela; Padilha, Kallyandra; Zatz, Roberto; Pereira, Alexandre C; Thadhian, Ravi I.; Rhee, Eugene P.; Titan, Silvia M. O.
Source: Metabolomics. Unidade: FM
Subjects: VASOS CORONÁRIOS, LIPÍDEOS, DOENÇAS CARDIOVASCULARES
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ABNT
TAVARES, Gesiane et al. 1,5-Anhydroglucitol predicts CKD progression in macroalbuminuric diabetic kidney disease: results from non-targeted metabolomics. Metabolomics, v. 14, n. 4, 2018Tradução . . Disponível em: https://doi.org/10.1007/s11306-018-1337-9. Acesso em: 15 nov. 2024.
APA
Tavares, G., Venturini, G., Padilha, K., Zatz, R., Pereira, A. C., Thadhian, R. I., et al. (2018). 1,5-Anhydroglucitol predicts CKD progression in macroalbuminuric diabetic kidney disease: results from non-targeted metabolomics. Metabolomics, 14( 4). doi:10.1007/s11306-018-1337-9
NLM
Tavares G, Venturini G, Padilha K, Zatz R, Pereira AC, Thadhian RI, Rhee EP, Titan SMO. 1,5-Anhydroglucitol predicts CKD progression in macroalbuminuric diabetic kidney disease: results from non-targeted metabolomics [Internet]. Metabolomics. 2018 ; 14( 4):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s11306-018-1337-9
Vancouver
Tavares G, Venturini G, Padilha K, Zatz R, Pereira AC, Thadhian RI, Rhee EP, Titan SMO. 1,5-Anhydroglucitol predicts CKD progression in macroalbuminuric diabetic kidney disease: results from non-targeted metabolomics [Internet]. Metabolomics. 2018 ; 14( 4):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s11306-018-1337-9
Artigo de periodico
An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney (2016)
Arias, Simone Costa Alarcon; Souza, Renata Alves; Malheiros, Denise Maria Avancini Costa; Fanelli, Camilla; Fujihara, Clarice Kazue; Zatz, Roberto
Source: American Journal of Physiology. Renal Physiology. Unidade: FM
Subjects: RATOS WISTAR, RIM, HIPERTENSÃO, FÁRMACOS DO SANGUE E SISTEMA HEMATOPOÉTICO
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ABNT
ARIAS, Simone Costa Alarcon et al. An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney. American Journal of Physiology. Renal Physiology, v. 310, n. 2, p. F135-F143, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00388.2015. Acesso em: 15 nov. 2024.
APA
Arias, S. C. A., Souza, R. A., Malheiros, D. M. A. C., Fanelli, C., Fujihara, C. K., & Zatz, R. (2016). An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney. American Journal of Physiology. Renal Physiology, 310( 2), F135-F143. doi:10.1152/ajprenal.00388.2015
NLM
Arias SCA, Souza RA, Malheiros DMAC, Fanelli C, Fujihara CK, Zatz R. An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 2): F135-F143.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00388.2015
Vancouver
Arias SCA, Souza RA, Malheiros DMAC, Fanelli C, Fujihara CK, Zatz R. An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 2): F135-F143.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00388.2015
Artigo de periodico
Altered KLOTHO and NF-κB-TNF-α signaling are correlated with nephrectomy-induced cognitive impairment in rats (2015)
Degaspari, Sabrina; Tzanno-Martins, Carmen; Fujihara, Clarice Kazue; Zatz, Roberto; Branco-Martins, João Paulo; Viel, Tânia Araújo; Buck, Hudson de Souza; Orellana, Ana Maria Marques; Böhmer, Ana Elisa; Lima, Larissa de Sá; Andreotti, Diana Zukas; Munhoz, Carolina Demarchi ; Scavone, Cristoforo ; Kawamoto, Elisa Mitiko
Source: PLOS ONE. Unidades: FM, EACH, ICB
Subjects: COGNIÇÃO, NEFROPATIAS, INFLAMAÇÃO, RIM, CÉREBRO
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ABNT
DEGASPARI, Sabrina et al. Altered KLOTHO and NF-κB-TNF-α signaling are correlated with nephrectomy-induced cognitive impairment in rats. PLOS ONE, v. 10, n. 5, p. e0125271-1 - e0125271-22, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0125271. Acesso em: 15 nov. 2024.
APA
Degaspari, S., Tzanno-Martins, C., Fujihara, C. K., Zatz, R., Branco-Martins, J. P., Viel, T. A., et al. (2015). Altered KLOTHO and NF-κB-TNF-α signaling are correlated with nephrectomy-induced cognitive impairment in rats. PLOS ONE, 10( 5), e0125271-1 - e0125271-22. doi:10.1371/journal.pone.0125271
NLM
Degaspari S, Tzanno-Martins C, Fujihara CK, Zatz R, Branco-Martins JP, Viel TA, Buck H de S, Orellana AMM, Böhmer AE, Lima L de S, Andreotti DZ, Munhoz CD, Scavone C, Kawamoto EM. Altered KLOTHO and NF-κB-TNF-α signaling are correlated with nephrectomy-induced cognitive impairment in rats [Internet]. PLOS ONE. 2015 ;10( 5): e0125271-1 - e0125271-22.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0125271
Vancouver
Degaspari S, Tzanno-Martins C, Fujihara CK, Zatz R, Branco-Martins JP, Viel TA, Buck H de S, Orellana AMM, Böhmer AE, Lima L de S, Andreotti DZ, Munhoz CD, Scavone C, Kawamoto EM. Altered KLOTHO and NF-κB-TNF-α signaling are correlated with nephrectomy-induced cognitive impairment in rats [Internet]. PLOS ONE. 2015 ;10( 5): e0125271-1 - e0125271-22.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0125271
Artigo de periodico
A Nonpeptide Angiotensin II Type 2 Receptor Agonist Prevents Renal Inflammation in Early Diabetes (2015)
Matavelli, Luis C.; Zatz, Roberto; Siragay, Helmy M.
Source: Journal of Cardiovascular Pharmacology. Unidade: FM
Subjects: RIM (FISIOPATOLOGIA), DIABETES MELLITUS, INFLAMAÇÃO, ANGIOTENSINA II, ESTRESSE OXIDATIVO
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ABNT
MATAVELLI, Luis C. e ZATZ, Roberto e SIRAGAY, Helmy M. A Nonpeptide Angiotensin II Type 2 Receptor Agonist Prevents Renal Inflammation in Early Diabetes. Journal of Cardiovascular Pharmacology, v. 65, n. 4, p. 371-376, 2015Tradução . . Disponível em: https://doi.org/10.1097/FJC.0000000000000207. Acesso em: 15 nov. 2024.
APA
Matavelli, L. C., Zatz, R., & Siragay, H. M. (2015). A Nonpeptide Angiotensin II Type 2 Receptor Agonist Prevents Renal Inflammation in Early Diabetes. Journal of Cardiovascular Pharmacology, 65( 4), 371-376. doi:10.1097/FJC.0000000000000207
NLM
Matavelli LC, Zatz R, Siragay HM. A Nonpeptide Angiotensin II Type 2 Receptor Agonist Prevents Renal Inflammation in Early Diabetes [Internet]. Journal of Cardiovascular Pharmacology. 2015 ; 65( 4): 371-376.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1097/FJC.0000000000000207
Vancouver
Matavelli LC, Zatz R, Siragay HM. A Nonpeptide Angiotensin II Type 2 Receptor Agonist Prevents Renal Inflammation in Early Diabetes [Internet]. Journal of Cardiovascular Pharmacology. 2015 ; 65( 4): 371-376.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1097/FJC.0000000000000207
Artigo de periodico-carta/editorial
Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial] (2013)
Zatz, Roberto; Fujihara, Clarice Kazue
Source: American Journal of Physiology. Renal Physiology. Unidade: FM
Subjects: TRATO URINÁRIO (FISIOPATOLOGIA), TRANSPLANTE DE RIM, EXPRESSÃO GÊNICA, INSUFICIÊNCIA RENAL CRÔNICA, NEFROPATIAS, HIDRÓLISE
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ABNT
ZATZ, Roberto e FUJIHARA, Clarice Kazue. Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial]. American Journal of Physiology. Renal Physiology. Bethesda: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1152/ajprenal.00504.2013. Acesso em: 15 nov. 2024. , 2013
APA
Zatz, R., & Fujihara, C. K. (2013). Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial]. American Journal of Physiology. Renal Physiology. Bethesda: Faculdade de Medicina, Universidade de São Paulo. doi:10.1152/ajprenal.00504.2013
NLM
Zatz R, Fujihara CK. Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial] [Internet]. American Journal of Physiology. Renal Physiology. 2013 ; 305( 2): F1544-F1545.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00504.2013
Vancouver
Zatz R, Fujihara CK. Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial] [Internet]. American Journal of Physiology. Renal Physiology. 2013 ; 305( 2): F1544-F1545.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00504.2013
Artigo de periodico
NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy (2013)
Okabe, Cristiene; Borges, Raquel Lerner; Almeida, Danilo Candido de; Fanelli, Camilla; Barlette, Grasiela Pedreira; Machado, Flavia Gomes; Arias, Simone Costa Alarcon; Malheiros, Denise Maria Avancini Costa; Câmara, Niels Olsen Saraiva ; Zatz, Roberto; Fujihara, Clarice Kazue
Source: American Journal of Physiology. Renal Physiology. Unidades: FM, ICB
Subjects: NEFROPATIAS, TÉCNICAS E PROCEDIMENTOS DE LABORATÓRIO, RIM (PATOLOGIA), RATOS WISTAR, MODELOS ANIMAIS, INSUFICIÊNCIA RENAL ANIMAL
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ABNT
OKABE, Cristiene et al. NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy. American Journal of Physiology. Renal Physiology, v. 305, n. 2, p. F155-F163, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00491.2012. Acesso em: 15 nov. 2024.
APA
Okabe, C., Borges, R. L., Almeida, D. C. de, Fanelli, C., Barlette, G. P., Machado, F. G., et al. (2013). NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy. American Journal of Physiology. Renal Physiology, 305( 2), F155-F163. doi:10.1152/ajprenal.00491.2012
NLM
Okabe C, Borges RL, Almeida DC de, Fanelli C, Barlette GP, Machado FG, Arias SCA, Malheiros DMAC, Câmara NOS, Zatz R, Fujihara CK. NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy [Internet]. American Journal of Physiology. Renal Physiology. 2013 ; 305( 2): F155-F163.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00491.2012
Vancouver
Okabe C, Borges RL, Almeida DC de, Fanelli C, Barlette GP, Machado FG, Arias SCA, Malheiros DMAC, Câmara NOS, Zatz R, Fujihara CK. NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy [Internet]. American Journal of Physiology. Renal Physiology. 2013 ; 305( 2): F155-F163.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00491.2012
Artigo de periodico
Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy (2013)
Arias, Simone Costa Alarcon; Valente, Carla Perez; Machado, Flavia Gomes; Fanelli, Camilla; Origassa, Clarice Silvia Taemi; Brito, Thales de; Câmara, Niels Olsen Saraiva ; Malheiros, Denise Maria Avancini Costa; Zatz, Roberto; Clarice Kazue Fujihara
Source: PLOS ONE. Unidades: FM, ICB
Subjects: ANIMAIS DE LABORATÓRIO, EXPERIMENTOS ANIMAIS, IMUNOHISTOQUÍMICA (MÉTODOS), INSUFICIÊNCIA RENAL CRÔNICA, GLOMÉRULOS RENAIS
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ABNT
ARIAS, Simone Costa Alarcon et al. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy. PLOS ONE, v. 8, n. 2, p. 16 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0056215. Acesso em: 15 nov. 2024.
APA
Arias, S. C. A., Valente, C. P., Machado, F. G., Fanelli, C., Origassa, C. S. T., Brito, T. de, et al. (2013). Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy. PLOS ONE, 8( 2), 16 . doi:10.1371/journal.pone.0056215
NLM
Arias SCA, Valente CP, Machado FG, Fanelli C, Origassa CST, Brito T de, Câmara NOS, Malheiros DMAC, Zatz R, Clarice Kazue Fujihara. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy [Internet]. PLOS ONE. 2013 ; 8( 2): 16 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0056215
Vancouver
Arias SCA, Valente CP, Machado FG, Fanelli C, Origassa CST, Brito T de, Câmara NOS, Malheiros DMAC, Zatz R, Clarice Kazue Fujihara. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy [Internet]. PLOS ONE. 2013 ; 8( 2): 16 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0056215
Artigo de periodico
Urinary MCP-1 and RBP: Independent predictors of renal outcome in macroalbuminuric diabetic nephropathy Original (2012)
Titan, S. M.; Vieira Jr., J. M.; Dominguez, W. V.; Moreira, S. R. S.; Pereira, A. B.; Barros, R. T.; Zatz, R.
Source: Journal of Diabetes and its Complications. Unidade: FM
Subjects: NEFROPATIAS (DIAGNÓSTICO), BIOMARCADORES, SEGUIMENTOS, INSUFICIÊNCIA RENAL CRÔNICA, DIABETES MELLITUS, ALBUMINÚRIA (URINA)
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ABNT
TITAN, S. M. et al. Urinary MCP-1 and RBP: Independent predictors of renal outcome in macroalbuminuric diabetic nephropathy Original. Journal of Diabetes and its Complications, v. 26, n. 5, p. 546-553, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2012.06.006. Acesso em: 15 nov. 2024.
APA
Titan, S. M., Vieira Jr., J. M., Dominguez, W. V., Moreira, S. R. S., Pereira, A. B., Barros, R. T., & Zatz, R. (2012). Urinary MCP-1 and RBP: Independent predictors of renal outcome in macroalbuminuric diabetic nephropathy Original. Journal of Diabetes and its Complications, 26( 5), 546-553. doi:10.1016/j.jdiacomp.2012.06.006
NLM
Titan SM, Vieira Jr. JM, Dominguez WV, Moreira SRS, Pereira AB, Barros RT, Zatz R. Urinary MCP-1 and RBP: Independent predictors of renal outcome in macroalbuminuric diabetic nephropathy Original [Internet]. Journal of Diabetes and its Complications. 2012 ; 26( 5): 546-553.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.jdiacomp.2012.06.006
Vancouver
Titan SM, Vieira Jr. JM, Dominguez WV, Moreira SRS, Pereira AB, Barros RT, Zatz R. Urinary MCP-1 and RBP: Independent predictors of renal outcome in macroalbuminuric diabetic nephropathy Original [Internet]. Journal of Diabetes and its Complications. 2012 ; 26( 5): 546-553.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.jdiacomp.2012.06.006
Artigo de periodico
Pivotal role of toll-like receptors 2 and 4, its adaptor molecule MyD88, and inflammasome complex in experimental tubule-interstitial nephritis (2011)
Correa-Costa, Matheus; Braga, Tarcio Teodoro; Semedo, Patricia; Hayashida, Caroline Yuri; Bechara, Luiz Roberto Grassmann; Elias, Rosa Maria; Barreto, claudiene Rodrigues; Silva-Cunha, Claudia; Hyane, Meire Ioshie; Gonçalves, Giselle Martins; Brum, Patricia Chakur; Fujihara, Clarice; Zats, Roberto; Pacheco-Silva, Álvaro; Zamboni, Dario Simões; Câmara, Niels Olsen Saraiva
Source: PLOS One. Unidades: FMRP, EEFE, FM, ICB
Subjects: TÚBULOS RENAIS, INFLAMAÇÃO, ADENOSINA, RECEPTORES (DESEMPENHO)
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ABNT
CORREA-COSTA, Matheus et al. Pivotal role of toll-like receptors 2 and 4, its adaptor molecule MyD88, and inflammasome complex in experimental tubule-interstitial nephritis. PLOS One, v. 6, n. 12, p. 1-7, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0029004. Acesso em: 15 nov. 2024.
APA
Correa-Costa, M., Braga, T. T., Semedo, P., Hayashida, C. Y., Bechara, L. R. G., Elias, R. M., et al. (2011). Pivotal role of toll-like receptors 2 and 4, its adaptor molecule MyD88, and inflammasome complex in experimental tubule-interstitial nephritis. PLOS One, 6( 12), 1-7. doi:10.1371/journal.pone.0029004
NLM
Correa-Costa M, Braga TT, Semedo P, Hayashida CY, Bechara LRG, Elias RM, Barreto claudiene R, Silva-Cunha C, Hyane MI, Gonçalves GM, Brum PC, Fujihara C, Zats R, Pacheco-Silva Á, Zamboni DS, Câmara NOS. Pivotal role of toll-like receptors 2 and 4, its adaptor molecule MyD88, and inflammasome complex in experimental tubule-interstitial nephritis [Internet]. PLOS One. 2011 ; 6( 12): 1-7.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0029004
Vancouver
Correa-Costa M, Braga TT, Semedo P, Hayashida CY, Bechara LRG, Elias RM, Barreto claudiene R, Silva-Cunha C, Hyane MI, Gonçalves GM, Brum PC, Fujihara C, Zats R, Pacheco-Silva Á, Zamboni DS, Câmara NOS. Pivotal role of toll-like receptors 2 and 4, its adaptor molecule MyD88, and inflammasome complex in experimental tubule-interstitial nephritis [Internet]. PLOS One. 2011 ; 6( 12): 1-7.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0029004
Artigo de periodico-carta/editorial
Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] (2011)
Fanelli, Camilla; Zatz, Roberto
Source: Hypertension. Unidade: FM
Subjects: ESTRESSE OXIDATIVO, SISTEMA RENINA-ANGIOTENSINA, HIPERTENSÃO (FISIOPATOLOGIA), EDITORIAL (COMENTÁRIOS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FANELLI, Camilla e ZATZ, Roberto. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html. Acesso em: 15 nov. 2024. , 2011
APA
Fanelli, C., & Zatz, R. (2011). Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]. Hypertension. Dallas: Faculdade de Medicina, Universidade de São Paulo. doi:10.1161/HYPERTENSIONAHA.110.167775
NLM
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 nov. 15 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
Vancouver
Fanelli C, Zatz R. Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial] [Internet]. Hypertension. 2011 ; 57( 3): 373-374.[citado 2024 nov. 15 ] Available from: http://hyper.ahajournals.org/content/57/3/373.full.pdf+html
Artigo de periodico
FGF-23 as a predictor of renal outcome in diabetic nephropathy (2011)
Titan, Silvia M.; Zatz, Roberto; Graciolli, Fabiana G.; Reis, Luciene M. dos; Barros, Rui Toledo; Jorgetti, Vanda; Moyse's, Rosa M.A.
Source: Clinical journal of the american society of nephrology. Unidade: FM
Subjects: NEFROPATIAS (COMPLICAÇÕES;EPIDEMIOLOGIA), ALBUMINAS (ANÁLISE), DIABETES MELLITUS, PLACEBOS, EVOLUÇÃO CLÍNICA, MÉTODO DUPLO-CEGO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TITAN, Silvia M. et al. FGF-23 as a predictor of renal outcome in diabetic nephropathy. Clinical journal of the american society of nephrology, v. 6, n. 2, p. 241-247, 2011Tradução . . Disponível em: http://cjasn.asnjournals.org/content/6/2/241.full.pdf+html. Acesso em: 15 nov. 2024.
APA
Titan, S. M., Zatz, R., Graciolli, F. G., Reis, L. M. dos, Barros, R. T., Jorgetti, V., & Moyse's, R. M. A. (2011). FGF-23 as a predictor of renal outcome in diabetic nephropathy. Clinical journal of the american society of nephrology, 6( 2), 241-247. Recuperado de http://cjasn.asnjournals.org/content/6/2/241.full.pdf+html
NLM
Titan SM, Zatz R, Graciolli FG, Reis LM dos, Barros RT, Jorgetti V, Moyse's RMA. FGF-23 as a predictor of renal outcome in diabetic nephropathy [Internet]. Clinical journal of the american society of nephrology. 2011 ; 6( 2): 241-247.[citado 2024 nov. 15 ] Available from: http://cjasn.asnjournals.org/content/6/2/241.full.pdf+html
Vancouver
Titan SM, Zatz R, Graciolli FG, Reis LM dos, Barros RT, Jorgetti V, Moyse's RMA. FGF-23 as a predictor of renal outcome in diabetic nephropathy [Internet]. Clinical journal of the american society of nephrology. 2011 ; 6( 2): 241-247.[citado 2024 nov. 15 ] Available from: http://cjasn.asnjournals.org/content/6/2/241.full.pdf+html
Artigo de periodico
Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation (2011)
Fanelli, C.; Fernandes, B. H. V.; Machado, F. G.; Okabe, C.; Malheiros, D. M. A. C.; Fujihara, C. K.; Zatz, R.
Source: American Journal of Physiology. Renal Physiology. Unidade: FM
Subjects: NEFROPATIAS (TERAPIA), ANGIOTENSINA II, INSUFICIÊNCIA RENAL CRÔNICA (TERAPIA;CONTROLE)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FANELLI, C. et al. Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation. American Journal of Physiology. Renal Physiology, v. 301, n. 3, p. F580-F587, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00042.2011. Acesso em: 15 nov. 2024.
APA
Fanelli, C., Fernandes, B. H. V., Machado, F. G., Okabe, C., Malheiros, D. M. A. C., Fujihara, C. K., & Zatz, R. (2011). Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation. American Journal of Physiology. Renal Physiology, 301( 3), F580-F587. doi:10.1152/ajprenal.00042.2011
NLM
Fanelli C, Fernandes BHV, Machado FG, Okabe C, Malheiros DMAC, Fujihara CK, Zatz R. Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation [Internet]. American Journal of Physiology. Renal Physiology. 2011 ; 301( 3): F580-F587.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00042.2011
Vancouver
Fanelli C, Fernandes BHV, Machado FG, Okabe C, Malheiros DMAC, Fujihara CK, Zatz R. Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation [Internet]. American Journal of Physiology. Renal Physiology. 2011 ; 301( 3): F580-F587.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00042.2011
Artigo de periodico
Early Brief Treatment with Losartan plus Mycophenolate Mofetil Provides Lasting Renoprotection in a Renal Ablation Model (2010)
Fujihara, Clarice Kazue; Vieira Jr., Jose Mauro; Sena, Claudia Ramos; Ventura, Bianca Helena; Malheiros, Denise Maria; Zatz, Roberto
Source: American Journal of Nephrology. Unidade: FM
Subjects: ANGIOTENSINA II, DOENÇA CRÔNICA, EVOLUÇÃO CLÍNICA, MODELOS ANIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FUJIHARA, Clarice Kazue et al. Early Brief Treatment with Losartan plus Mycophenolate Mofetil Provides Lasting Renoprotection in a Renal Ablation Model. American Journal of Nephrology, v. 32, n. 2, p. 95-102, 2010Tradução . . Disponível em: https://doi.org/10.1159/000315137. Acesso em: 15 nov. 2024.
APA
Fujihara, C. K., Vieira Jr., J. M., Sena, C. R., Ventura, B. H., Malheiros, D. M., & Zatz, R. (2010). Early Brief Treatment with Losartan plus Mycophenolate Mofetil Provides Lasting Renoprotection in a Renal Ablation Model. American Journal of Nephrology, 32( 2), 95-102. doi:10.1159/000315137
NLM
Fujihara CK, Vieira Jr. JM, Sena CR, Ventura BH, Malheiros DM, Zatz R. Early Brief Treatment with Losartan plus Mycophenolate Mofetil Provides Lasting Renoprotection in a Renal Ablation Model [Internet]. American Journal of Nephrology. 2010 ; 32( 2): 95-102.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1159/000315137
Vancouver
Fujihara CK, Vieira Jr. JM, Sena CR, Ventura BH, Malheiros DM, Zatz R. Early Brief Treatment with Losartan plus Mycophenolate Mofetil Provides Lasting Renoprotection in a Renal Ablation Model [Internet]. American Journal of Nephrology. 2010 ; 32( 2): 95-102.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1159/000315137
Artigo de periodico
Demonstration of Epithelial-Mesenchymal Transition in Kidney: The Contribution of a Coupled Histochemical and Immunohistochemical Staining With Periodic Acid-Thionin Schiff (2008)
Santos, Maria Carmen Lopes Ferreira Silva; Musso, Carlos; Alves, Venâncio Avancini Ferreira; Zatz, Roberto; Fujihara, Clarice kazue; Malheiros, Denise Maria Avancini Costa
Source: Applied immunohistochemistry & molecular morphology. Unidade: FM
Subjects: TÚBULOS RENAIS (IMUNOLOGIA), RIM (PATOLOGIA), ÁCIDOS (CLASSIFICAÇÃO), IMUNOHISTOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Maria Carmen Lopes Ferreira Silva et al. Demonstration of Epithelial-Mesenchymal Transition in Kidney: The Contribution of a Coupled Histochemical and Immunohistochemical Staining With Periodic Acid-Thionin Schiff. Applied immunohistochemistry & molecular morphology, v. 16, n. 2, 2008Tradução . . Disponível em: https://doi.org/10.1097/PAI.0b013e31804d680f. Acesso em: 15 nov. 2024.
APA
Santos, M. C. L. F. S., Musso, C., Alves, V. A. F., Zatz, R., Fujihara, C. kazue, & Malheiros, D. M. A. C. (2008). Demonstration of Epithelial-Mesenchymal Transition in Kidney: The Contribution of a Coupled Histochemical and Immunohistochemical Staining With Periodic Acid-Thionin Schiff. Applied immunohistochemistry & molecular morphology, 16( 2). doi:10.1097/PAI.0b013e31804d680f
NLM
Santos MCLFS, Musso C, Alves VAF, Zatz R, Fujihara C kazue, Malheiros DMAC. Demonstration of Epithelial-Mesenchymal Transition in Kidney: The Contribution of a Coupled Histochemical and Immunohistochemical Staining With Periodic Acid-Thionin Schiff [Internet]. Applied immunohistochemistry & molecular morphology. 2008 ; 16( 2):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1097/PAI.0b013e31804d680f
Vancouver
Santos MCLFS, Musso C, Alves VAF, Zatz R, Fujihara C kazue, Malheiros DMAC. Demonstration of Epithelial-Mesenchymal Transition in Kidney: The Contribution of a Coupled Histochemical and Immunohistochemical Staining With Periodic Acid-Thionin Schiff [Internet]. Applied immunohistochemistry & molecular morphology. 2008 ; 16( 2):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1097/PAI.0b013e31804d680f
Artigo de periodico
Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model (2007)
Fujihara, Clarice Kazue; Malheiros, Denise M. A. C.; Zatz, Roberto
Source: American Journal of Physiology. Renal Physiology. Unidade: FM
Subjects: PRESSÃO SANGUÍNEA, MODELOS ANIMAIS, SEGUIMENTOS, CATETRISMO, NEFRECTOMIA, INSUFICIÊNCIA RENAL CRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FUJIHARA, Clarice Kazue e MALHEIROS, Denise M. A. C. e ZATZ, Roberto. Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model. American Journal of Physiology. Renal Physiology, v. 292, n. 6, p. F1810-F1818, 2007Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00521.2006. Acesso em: 15 nov. 2024.
APA
Fujihara, C. K., Malheiros, D. M. A. C., & Zatz, R. (2007). Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model. American Journal of Physiology. Renal Physiology, 292( 6), F1810-F1818. doi:10.1152/ajprenal.00521.2006
NLM
Fujihara CK, Malheiros DMAC, Zatz R. Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model [Internet]. American Journal of Physiology. Renal Physiology. 2007 ; 292( 6): F1810-F1818.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00521.2006
Vancouver
Fujihara CK, Malheiros DMAC, Zatz R. Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model [Internet]. American Journal of Physiology. Renal Physiology. 2007 ; 292( 6): F1810-F1818.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00521.2006
Artigo de periodico
Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model (2007)
Fujihara, Clarice Kazue; Antunes, Gláucia R.; Mattar, Ana Lúcia; Malheiros, Denise M. A. C.; Vieira, José M.; Zatz, Roberto
Source: American Journal Physiology Renal Physiology. Unidade: FM
Subjects: NEFROPATIAS, INFLAMAÇÃO, GLOMÉRULOS RENAIS (PATOLOGIA), ANGIOTENSINA II, DOENÇA CRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FUJIHARA, Clarice Kazue et al. Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model. American Journal Physiology Renal Physiology, n. 292, p. F92-F99, 2007Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00184.2006. Acesso em: 15 nov. 2024.
APA
Fujihara, C. K., Antunes, G. R., Mattar, A. L., Malheiros, D. M. A. C., Vieira, J. M., & Zatz, R. (2007). Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model. American Journal Physiology Renal Physiology, ( 292), F92-F99. doi:10.1152/ajprenal.00184.2006
NLM
Fujihara CK, Antunes GR, Mattar AL, Malheiros DMAC, Vieira JM, Zatz R. Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model [Internet]. American Journal Physiology Renal Physiology. 2007 ;( 292): F92-F99.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00184.2006
Vancouver
Fujihara CK, Antunes GR, Mattar AL, Malheiros DMAC, Vieira JM, Zatz R. Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model [Internet]. American Journal Physiology Renal Physiology. 2007 ;( 292): F92-F99.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1152/ajprenal.00184.2006

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