Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy (2013)
- Authors:
- USP affiliated authors: ZATZ, ROBERTO - FM ; FANELLI, CAMILLA - FM ; BRITO, THALES DE - FM ; CÂMARA, NIELS OLSEN SARAIVA - ICB ; FUJIHARA, CLARICE KAZUE - FM
- Unidades: FM; ICB
- DOI: 10.1371/journal.pone.0056215
- Subjects: ANIMAIS DE LABORATÓRIO; EXPERIMENTOS ANIMAIS; IMUNOHISTOQUÍMICA (MÉTODOS); INSUFICIÊNCIA RENAL CRÔNICA; GLOMÉRULOS RENAIS
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2013
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
ARIAS, Simone Costa Alarcon et al. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy. PLOS ONE, v. 8, n. 2, p. 16 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0056215. Acesso em: 29 dez. 2025. -
APA
Arias, S. C. A., Valente, C. P., Machado, F. G., Fanelli, C., Origassa, C. S. T., Brito, T. de, et al. (2013). Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy. PLOS ONE, 8( 2), 16 . doi:10.1371/journal.pone.0056215 -
NLM
Arias SCA, Valente CP, Machado FG, Fanelli C, Origassa CST, Brito T de, Câmara NOS, Malheiros DMAC, Zatz R, Clarice Kazue Fujihara. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy [Internet]. PLOS ONE. 2013 ; 8( 2): 16 .[citado 2025 dez. 29 ] Available from: https://doi.org/10.1371/journal.pone.0056215 -
Vancouver
Arias SCA, Valente CP, Machado FG, Fanelli C, Origassa CST, Brito T de, Câmara NOS, Malheiros DMAC, Zatz R, Clarice Kazue Fujihara. Regression of Albuminuria and Hypertension and Arrest of Severe Renal Injury by a Losartan-Hydrochlorothiazide Association in a Model of Very Advanced Nephropathy [Internet]. PLOS ONE. 2013 ; 8( 2): 16 .[citado 2025 dez. 29 ] Available from: https://doi.org/10.1371/journal.pone.0056215 - NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy
- Losartan (L) and hydrochlorotiazide (H) association (LH) arrests renal inhury in a model of very advanced chronic kidney disease (CKD)
- Losartan (L) and Hydrochlorothiazide (H)Treatment (LH) arrests renal and cardiac injury in advanced chronic kidney disease by mechanisms not entireky dependent on blood pressure
- Renoprotective effect of two dosages of Losartan (L) in a model of crhonic kidney disease (CDK) by nitric oxide inhibition and and salt overload
- Mechanisms of progressive renal injury in adult rats treated with losartan during lactation
- Effects of losartan, in monotherapy or in association with hydrochlorothiazide, in chronic nephropathy resulting from losartan treatment during lactation
- Associação terapêutica de losartan (L) e hidroclorotiazida (H) previne progressão da lesão renal em modelo de nefropatia crônica resultante da administração de (L) durante a lactação
- Furosemide (F) is less effective than hydroclorothiazide (H) as adjuvant to losartan (L) in arresting the nephropathy associated with the 5/6 renal ablation model (NX)
- Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension [Editorial]
- An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney
Informações sobre o DOI: 10.1371/journal.pone.0056215 (Fonte: oaDOI API)
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