NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model (2018)
- Authors:
- USP affiliated authors: MALHEIROS, DENISE MARIA AVANCINI COSTA - FM ; ZATZ, ROBERTO - FM ; FUJIHARA, CLARICE KAZUE - FM
- Unidade: FM
- DOI: 10.1038/s41374-018-0029-4
- Subjects: NEFROPATIAS; RATOS WISTAR; INIBIDORES DE ENZIMAS; ESTRESSE OXIDATIVO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Laboratory investigation
- ISSN: 0023-6837
- Volume/Número/Paginação/Ano: v. 98, n. 6, p. 773-782, 2018
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FORESTO-NETO, Orestes et al. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model. Laboratory investigation, v. 98, n. 6, p. 773-782, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41374-018-0029-4. Acesso em: 14 fev. 2026. -
APA
Foresto-neto, O., Malheiros, D. M. A. C., Zatz, R., & Fujihara, C. K. (2018). NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model. Laboratory investigation, 98( 6), 773-782. doi:10.1038/s41374-018-0029-4 -
NLM
Foresto-neto O, Malheiros DMAC, Zatz R, Fujihara CK. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model [Internet]. Laboratory investigation. 2018 ; 98( 6): 773-782.[citado 2026 fev. 14 ] Available from: https://doi.org/10.1038/s41374-018-0029-4 -
Vancouver
Foresto-neto O, Malheiros DMAC, Zatz R, Fujihara CK. NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model [Internet]. Laboratory investigation. 2018 ; 98( 6): 773-782.[citado 2026 fev. 14 ] Available from: https://doi.org/10.1038/s41374-018-0029-4 - Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload
- TLR2 and TLR4 play opposite role in autophagy associated with cisplatin-induced acute kidney injury
- Simultaneous activation of innate and adaptive immunity participates in the development of renal injury in a model of heavy proteinuria
- NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease
- An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney
- Renal and myocardial histopathology and morphometry in rats with adenine induced chronic renal failure: Influence of gum acacia
- Chronic environmental hypoxia attenuates innate immunity activation and renal injury in two CKD models
- The inflammatory component in progressive renal disease: are interventions possible
- Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial]
- Acute renal failure (arf) after intravenous (iv) cyclosporine (csa) administration role of the vehicle
Informações sobre o DOI: 10.1038/s41374-018-0029-4 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas