NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease (2020)
- Authors:
- USP affiliated authors: MALHEIROS, DENISE MARIA AVANCINI COSTA - FM ; FUJIHARA, CLARICE KAZUE - FM ; ZATZ, ROBERTO - FM
- Unidade: FM
- DOI: 10.3389/fphys.2020.00084
- Subjects: INFLAMAÇÃO; RECEPTORES; PREVENÇÃO DE DOENÇAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in physiology
- ISSN: 1664-042X
- Volume/Número/Paginação/Ano: v. 11, article ID 84, 11p, 2020
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FORESTO-NETO, Orestes et al. NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease. Frontiers in physiology, v. 11, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2020.00084. Acesso em: 14 fev. 2026. -
APA
Foresto-Neto, O., Albino, A. H., Arias, S. C. A., Faustino, V. D., Zambom, F. F. F., Cenedeze, M. A., et al. (2020). NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease. Frontiers in physiology, 11. doi:10.3389/fphys.2020.00084 -
NLM
Foresto-Neto O, Albino AH, Arias SCA, Faustino VD, Zambom FFF, Cenedeze MA, Elias RM, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease [Internet]. Frontiers in physiology. 2020 ; 11[citado 2026 fev. 14 ] Available from: https://doi.org/10.3389/fphys.2020.00084 -
Vancouver
Foresto-Neto O, Albino AH, Arias SCA, Faustino VD, Zambom FFF, Cenedeze MA, Elias RM, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease [Internet]. Frontiers in physiology. 2020 ; 11[citado 2026 fev. 14 ] Available from: https://doi.org/10.3389/fphys.2020.00084 - Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload
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Informações sobre o DOI: 10.3389/fphys.2020.00084 (Fonte: oaDOI API)
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