Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload (2019)
- Authors:
- USP affiliated authors: MALHEIROS, DENISE MARIA AVANCINI COSTA - FM ; ZATZ, ROBERTO - FM ; FUJIHARA, CLARICE KAZUE - FM
- Unidade: FM
- DOI: 10.1152/ajprenal.00251.2019
- Subjects: IMUNIDADE NATURAL; INSUFICIÊNCIA RENAL CRÔNICA; HIPERTENSÃO; ÓXIDO NÍTRICO; SAL (CONDIMENTO); INFLAMAÇÃO; IMUNOHISTOQUÍMICA; RATOS WISTAR
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American journal of physiology-renal physiology
- ISSN: 1931-857X
- Volume/Número/Paginação/Ano: v. 317, n. 4, p. F1058-F1067, 2019
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
ZAMBOM, Fernanda Florencia Fregnan et al. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload. American journal of physiology-renal physiology, v. 317, n. 4, p. F1058-F1067, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00251.2019. Acesso em: 24 abr. 2024. -
APA
Zambom, F. F. F., Oliveira, K. C., Foresto-Neto, O., Faustino, V. D., Avila, V. F., Albino, A. H., et al. (2019). Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload. American journal of physiology-renal physiology, 317( 4), F1058-F1067. doi:10.1152/ajprenal.00251.2019 -
NLM
Zambom FFF, Oliveira KC, Foresto-Neto O, Faustino VD, Avila VF, Albino AH, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload [Internet]. American journal of physiology-renal physiology. 2019 ; 317( 4): F1058-F1067.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1152/ajprenal.00251.2019 -
Vancouver
Zambom FFF, Oliveira KC, Foresto-Neto O, Faustino VD, Avila VF, Albino AH, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK. Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload [Internet]. American journal of physiology-renal physiology. 2019 ; 317( 4): F1058-F1067.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1152/ajprenal.00251.2019 - TLR2 and TLR4 play opposite role in autophagy associated with cisplatin-induced acute kidney injury
- NF-kappa B System Is Chronically Activated and Promotes Glomerular Injury in Experimental Type 1 Diabetic Kidney Disease
- NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model
- Simultaneous activation of innate and adaptive immunity participates in the development of renal injury in a model of heavy proteinuria
- An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney
- Renal and myocardial histopathology and morphometry in rats with adenine induced chronic renal failure: Influence of gum acacia
- Role of glomerular hypertension: glomerular hypertrophy and lipid deposition in the genesis of glomerular sclerosis (gs) of experimental diabetes (dm)
- Glomerular abnormalities in long-term experimental diabetes
- Effects of an endothelin et receptor antagonist on experimental fk506 induced acute renal dysfunction
- An extremely high dose of losartan affords superior renoprotection in the remnant model
Informações sobre o DOI: 10.1152/ajprenal.00251.2019 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
