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Artigo de periodico
Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes (2024)
Michalani, Maria Luiza Estimo ; Passarelli, Marisa ; Machado, Ubiratan Fabres
Source: International Journal of Molecular Sciences. Unidades: FM, ICB
Subjects: FISIOLOGIA, ADIPOSIDADE, TECIDO ADIPOSO, EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, TRANSCRIÇÃO GÊNICA, DIABETES MELLITUS, GLICOSE, WESTERN BLOTTING, ATIVAÇÃO ENZIMÁTICA, INFLAMAÇÃO
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MICHALANI, Maria Luiza Estimo e PASSARELLI, Marisa e MACHADO, Ubiratan Fabres. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes. International Journal of Molecular Sciences, v. 25, n. 15, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25158242. Acesso em: 18 nov. 2024.
APA
Michalani, M. L. E., Passarelli, M., & Machado, U. F. (2024). Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes. International Journal of Molecular Sciences, 25( 15), 14 . doi:10.3390/ijms25158242
NLM
Michalani MLE, Passarelli M, Machado UF. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 15): 14 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms25158242
Vancouver
Michalani MLE, Passarelli M, Machado UF. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 15): 14 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms25158242
Artigo de periodico
Programming of metabolism by adipokines during development (2023)
Donato Junior, José
Source: Nature Reviews Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, METABOLISMO, DOENÇAS METABÓLICAS, HOMEOSTASE, GLICOSE, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO
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DONATO JUNIOR, José. Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41574-023-00828-1. Acesso em: 18 nov. 2024.
APA
Donato Junior, J. (2023). Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, 1-13. doi:10.1038/s41574-023-00828-1
NLM
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Vancouver
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Artigo de periodico
Metformin acts in the gut and induces gut-liver crosstalk (2023)
Tobar, Natália ; Rocha, Guilherme Z. ; Santos, Andrey dos ; Guadagnini, Dioze ; Assalin, Heloísa B. ; Camargo, Juliana Alves de ; Gonçalves, Any E. S. S. ; Pallis, Flavia R.; Oliveira, Alexandre G. ; Rocco, Silvana A. ; Neto, Raphael M. ; Sousa, Irene Layane de ; Alborguetti, Marcos R. ; Sforça, Mauricio L. ; Rodrigues, Patricia B.; Ludwig, Raissa G. ; Vanzela, Emerielle C. ; Brunetto, Sergio Q. ; Boer, Patricia A. ; Gontijo, José A. R. ; Geloneze, Bruno; Prada, Patricia O. ; Frolli, Franco; Curi, Rui ; Mori, Marcelo A. ; Vinolo, Marco A. R. ; Ramos, Celso D. ; Franchini, Kleber G. ; Tormena, Cláudio F. ; Saad, Mario J. A. ; Carvalho, Carla Roberta de Oliveira
Source: PNAS Nexus. Unidade: ICB
Subjects: FISIOLOGIA, CAMUNDONGOS, ANTIDIABÉTICOS (HIPOGLICEMIANTES), DIABETES MELLITUS, HIPERGLICEMIA, METFORMINA, GLICOSE, FÍGADO, INTESTINO DE ANIMAL, TOMOGRAFIA COMPUTADORIZADA DE EMISSÃO, SISTEMA GASTROINTESTINAL
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TOBAR, Natália et al. Metformin acts in the gut and induces gut-liver crosstalk. PNAS Nexus, v. 120, n. 4, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2211933120. Acesso em: 18 nov. 2024.
APA
Tobar, N., Rocha, G. Z., Santos, A. dos, Guadagnini, D., Assalin, H. B., Camargo, J. A. de, et al. (2023). Metformin acts in the gut and induces gut-liver crosstalk. PNAS Nexus, 120( 4), 1-11. doi:10.1073/pnas.2211933120
NLM
Tobar N, Rocha GZ, Santos A dos, Guadagnini D, Assalin HB, Camargo JA de, Gonçalves AESS, Pallis FR, Oliveira AG, Rocco SA, Neto RM, Sousa IL de, Alborguetti MR, Sforça ML, Rodrigues PB, Ludwig RG, Vanzela EC, Brunetto SQ, Boer PA, Gontijo JAR, Geloneze B, Prada PO, Frolli F, Curi R, Mori MA, Vinolo MAR, Ramos CD, Franchini KG, Tormena CF, Saad MJA, Carvalho CR de O. Metformin acts in the gut and induces gut-liver crosstalk [Internet]. PNAS Nexus. 2023 ; 120( 4): 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1073/pnas.2211933120
Vancouver
Tobar N, Rocha GZ, Santos A dos, Guadagnini D, Assalin HB, Camargo JA de, Gonçalves AESS, Pallis FR, Oliveira AG, Rocco SA, Neto RM, Sousa IL de, Alborguetti MR, Sforça ML, Rodrigues PB, Ludwig RG, Vanzela EC, Brunetto SQ, Boer PA, Gontijo JAR, Geloneze B, Prada PO, Frolli F, Curi R, Mori MA, Vinolo MAR, Ramos CD, Franchini KG, Tormena CF, Saad MJA, Carvalho CR de O. Metformin acts in the gut and induces gut-liver crosstalk [Internet]. PNAS Nexus. 2023 ; 120( 4): 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1073/pnas.2211933120
Artigo de periodico
Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy (2023)
Ortiz-Silva, Milene ; Leonardi, Bianca Franco ; Castro, Érique de ; Peixoto, Albert Souza ; Gilio, Gustavo Renan ; Oliveira, Tiago E. ; Tomazelli, Caroline Aparecida ; Andrade, Maynara L.; Moreno, Mayara Franzoi ; Belchior, Thiago ; Magdalon, Juliana ; Vieira, Tayana S.; Donado-Pestana, Carlos M. ; Festuccia, William T.
Source: Molecular and Cellular Endocrinology. Unidades: ICB, FCF
Subjects: FISIOLOGIA, HOMEOSTASE, GLICOSE, LIPÍDEOS, ROEDORES, OBESIDADE, CAMUNDONGOS, DIETA ANIMAL, GORDURAS NA DIETA, ADIPOSIDADE, PESOS E MEDIDAS CORPORAIS, INSULINA, FIBROBLASTOS
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ORTIZ-SILVA, Milene et al. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, v. 578, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2023.112074. Acesso em: 18 nov. 2024.
APA
Ortiz-Silva, M., Leonardi, B. F., Castro, É. de, Peixoto, A. S., Gilio, G. R., Oliveira, T. E., et al. (2023). Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, 578, 11 . doi:10.1016/j.mce.2023.112074
NLM
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Vancouver
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Artigo de periodico
mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction (2023)
Souza, Gabriel Orefice de ; Teixeira, Pryscila Dryelle Sousa ; Camara, Niels Olsen Saraiva ; Donato Junior, José
Source: Cells. Unidade: ICB
Subjects: IMUNOLOGIA, FISIOLOGIA, HIPOTÁLAMO, PROTEÍNAS, CAMUNDONGOS, GLICOSE, ALIMENTAÇÃO ANIMAL, NEURÔNIOS, DIETA ANIMAL, PESOS E MEDIDAS CORPORAIS
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SOUZA, Gabriel Orefice de et al. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, v. 12, n. 20, p. 17 , 2023Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf. Acesso em: 18 nov. 2024.
APA
Souza, G. O. de, Teixeira, P. D. S., Camara, N. O. S., & Donato Junior, J. (2023). mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, 12( 20), 17 . doi:10.3390/cells12202442
NLM
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 nov. 18 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Vancouver
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 nov. 18 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Artigo de periodico
Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia (2023)
Yonamine, Caio Yogi ; Michalani, Maria Luiza Estimo ; Moreira, Rafael Junges ; Machado, Ubiratan Fabres
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, GLICOSE, OBESIDADE, DIABETES MELLITUS, RESISTÊNCIA À INSULINA, DOENÇA DE PARKINSON, DEMÊNCIA, ESTRESSE OXIDATIVO, HIPOCAMPO
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YONAMINE, Caio Yogi et al. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia. International Journal of Molecular Sciences, v. 24, n. 22, p. 24 , 2023Tradução . . Disponível em: https://doi.org/10.3390/ijms242216480. Acesso em: 18 nov. 2024.
APA
Yonamine, C. Y., Michalani, M. L. E., Moreira, R. J., & Machado, U. F. (2023). Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia. International Journal of Molecular Sciences, 24( 22), 24 . doi:10.3390/ijms242216480
NLM
Yonamine CY, Michalani MLE, Moreira RJ, Machado UF. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 22): 24 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms242216480
Vancouver
Yonamine CY, Michalani MLE, Moreira RJ, Machado UF. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 22): 24 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms242216480
Artigo de periodico
α-cell electrophysiology and the regulation of glucagon secretion (2023)
Gao, Rui ; Acreman, Samuel ; Ma, Jinfang; Wendt, Anne ; Quan Zhang ; Abdulkader, Fernando
Source: Journal of Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, GLUCAGON, ELETROFISIOLOGIA, GLICOSE, ILHOTAS DE LANGERHANS, HIPOGLICEMIA
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GAO, Rui et al. α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, v. 26, n. 2, p. 1-20, 2023Tradução . . Disponível em: https://doi.org/10.1530/JOE-22-0295. Acesso em: 18 nov. 2024.
APA
Gao, R., Acreman, S., Ma, J., Wendt, A., Quan Zhang,, & Abdulkader, F. (2023). α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, 26( 2), 1-20. doi:10.1530/JOE-22-0295
NLM
Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JOE-22-0295
Vancouver
Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JOE-22-0295
Artigo de periodico
Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice (2023)
Valdivia, Luís Felipe Galvão ; Castro, Érique ; Eichler, Rosangela Aparecida dos Santos ; Moreno, Mayara Franzoi ; Sousa, Érica de; Jardim, Giovanna Freitas Rodrigues; Peixoto, Álbert Souza ; Moraes, Maria Nathália ; Castrucci, Ana Maria de Lauro ; Nedergaard, Jan ; Petrovic, Natasa ; Reckziegel, Patrícia ; Festuccia, William Tadeu Lara
Source: American Journal of Physiology-Endocrinology and Metabolism. Unidades: ICB, IB, FCF
Subjects: FISIOLOGIA, TERMOGÊNESE, TECIDO ADIPOSO, ACLIMATAÇÃO, GLICOSE, CAMUNDONGOS, PEROXISSOMOS, TEMPERATURA ANIMAL, PROTEÍNAS DA MEMBRANA
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VALDIVIA, Luís Felipe Galvão et al. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice. American Journal of Physiology-Endocrinology and Metabolism, v. 324, n. 4, p. E289-E373, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00217.202. Acesso em: 18 nov. 2024.
APA
Valdivia, L. F. G., Castro, É., Eichler, R. A. dos S., Moreno, M. F., Sousa, É. de, Jardim, G. F. R., et al. (2023). Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice. American Journal of Physiology-Endocrinology and Metabolism, 324( 4), E289-E373. doi:10.1152/ajpendo.00217.202
NLM
Valdivia LFG, Castro É, Eichler RA dos S, Moreno MF, Sousa É de, Jardim GFR, Peixoto ÁS, Moraes MN, Castrucci AM de L, Nedergaard J, Petrovic N, Reckziegel P, Festuccia WTL. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2023 ; 324( 4): E289-E373.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpendo.00217.202
Vancouver
Valdivia LFG, Castro É, Eichler RA dos S, Moreno MF, Sousa É de, Jardim GFR, Peixoto ÁS, Moraes MN, Castrucci AM de L, Nedergaard J, Petrovic N, Reckziegel P, Festuccia WTL. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2023 ; 324( 4): E289-E373.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpendo.00217.202
Artigo de periodico
Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation (2023)
Yonamine, Caio Yogi ; Passarelli, Marisa ; Suemoto, Claudia Kimie ; Pasqualucci, Carlos Augusto ; Jacob-Filho, Wilson ; Alves, Venâncio Avancini Ferreira ; Marie, Suely Kazue Nagahashi ; Giannella, Maria Lucia Correa ; Britto, Luiz Roberto ; Machado, Ubiratan Fabres
Source: Cells. Unidades: FM, ICB
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DIABETES MELLITUS, HIPOCAMPO, NEUROBLASTOMA, OBESIDADE, DOENÇAS NEURODEGENERATIVAS, ESTRESSE OXIDATIVO, GLICOSE, DNA POLIMERASES
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YONAMINE, Caio Yogi et al. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, v. 12, n. 9, p. 1-21, 2023Tradução . . Disponível em: https://doi.org/10.3390/cells12091250. Acesso em: 18 nov. 2024.
APA
Yonamine, C. Y., Passarelli, M., Suemoto, C. K., Pasqualucci, C. A., Jacob-Filho, W., Alves, V. A. F., et al. (2023). Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, 12( 9), 1-21. doi:10.3390/cells12091250
NLM
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells12091250
Vancouver
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells12091250
Artigo de periodico
Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice (2022)
Veras, Katherine Maria de Araujo ; Monaco, Camila Ferraz Lucena ; Goedcke, Julia; Evangelista, Fabiana de Sant'Anna ; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Source: Frontiers in Endocrinology. Unidades: EACH, ICB
Subjects: FISIOLOGIA, PROTEÍNAS DO LEITE, DIETA, EXERCÍCIO FÍSICO, TREINAMENTO AERÓBIO, SACAROSE, ILHOTAS DE LANGERHANS, RESISTÊNCIA À INSULINA, GLICOSE, OBESIDADE, CAMUNDONGOS
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VERAS, Katherine Maria de Araujo et al. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, v. 13, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fendo.2022.881236. Acesso em: 18 nov. 2024.
APA
Veras, K. M. de A., Monaco, C. F. L., Goedcke, J., Evangelista, F. de S. 'A., Carpinelli, A. R., & Carvalho, C. R. de O. (2022). Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, 13, 1-12. doi:10.3389/fendo.2022.881236
NLM
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Vancouver
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Artigo de periodico
Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice (2022)
Miranda, Juliane Braga ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Jensen, Leonardo ; Irigoyen, Maria Cláudia Costa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: FISIOLOGIA, ANATOMIA, DIETA ANIMAL, GLICOSE, ISQUEMIA, OBESIDADE, CAMUNDONGOS, DOENÇAS CARDIOVASCULARES EM ANIMAL, DOENÇAS METABÓLICAS
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MIRANDA, Juliane Braga et al. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, v. 56, n. 3, p. 293-309, 2022Tradução . . Disponível em: https://doi.org/10.33594/000000535. Acesso em: 18 nov. 2024.
APA
Miranda, J. B., Lunardon, G., Lima, V. M., Silva, T. de O., Lino, C. A., Jensen, L., et al. (2022). Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, 56( 3), 293-309. doi:10.33594/000000535
NLM
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 18 ] Available from: https://doi.org/10.33594/000000535
Vancouver
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 18 ] Available from: https://doi.org/10.33594/000000535
Artigo de periodico
SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism (2022)
Pedroso, João Alfredo Bolivar ; Silva, Ivson Bruno da ; Zampieri, Thais Tessari ; Totola, Leonardo Tedesco; Moreira, Thiago dos Santos ; Taniguti, Ana Paula Tiemi; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, CARDIOPATIAS, HIPOTÁLAMO, LEPTINA, OBESIDADE, ENVELHECIMENTO, CAMUNDONGOS, METABOLISMO DE PROTEÍNA, METABOLISMO ENERGÉTICO, NEURÔNIOS, CITOCININAS, GLICOSE
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ABNT
PEDROSO, João Alfredo Bolivar et al. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, v. 23, n. 12, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23126484. Acesso em: 18 nov. 2024.
APA
Pedroso, J. A. B., Silva, I. B. da, Zampieri, T. T., Totola, L. T., Moreira, T. dos S., Taniguti, A. P. T., et al. (2022). SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, 23( 12), 1-19. doi:10.3390/ijms23126484
NLM
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms23126484
Vancouver
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms23126484
Artigo de periodico
Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study (2022)
Faria, Daniele de Paula ; Souza, Larissa Estessi de ; Duran, Fabio Luis de Souza ; Buchpiguel, Carlos Alberto ; Britto, Luiz Roberto Giorgetti de ; Crippa, José Alexandre de Souza ; Busatto Filho, Geraldo ; Gregório, Caroline Cristiano Real
Source: International Journal of Molecular Sciences. Unidades: FM, ICB, FMRP
Subjects: FISIOLOGIA, CANABINOIDES, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, ANTINEOPLÁSICOS, MEMÓRIA ANIMAL, GLICOSE
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FARIA, Daniele de Paula et al. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, v. 23. n. 3, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23031076. Acesso em: 18 nov. 2024.
APA
Faria, D. de P., Souza, L. E. de, Duran, F. L. de S., Buchpiguel, C. A., Britto, L. R. G. de, Crippa, J. A. de S., et al. (2022). Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, 23. n. 3, 1-10. doi:10.3390/ijms23031076
NLM
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms23031076
Vancouver
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms23031076
Artigo de periodico
BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 (2022)
Jesus, Daniel Simões de; Souza, Paula Bargi ; Cruzat, Vinicius ; Yechoor, Vijay; Carpinelli, Angelo Rafael ; Garcia, Rodrigo Antonio Peliciari
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, PÂNCREAS, INSULINA, RITMOS BIOLÓGICOS, SUPERÓXIDO DISMUTASE, FATORES DE TRANSCRIÇÃO, GLICOSE, CAMUNDONGOS, PERÓXIDO DE HIDROGÊNIO, GLUTATIONA TRANSFERASE
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ABNT
JESUS, Daniel Simões de et al. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, v. 555, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2022.111725. Acesso em: 18 nov. 2024.
APA
Jesus, D. S. de, Souza, P. B., Cruzat, V., Yechoor, V., Carpinelli, A. R., & Garcia, R. A. P. (2022). BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, 555, 1-8. doi:10.1016/j.mce.2022.111725
NLM
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Vancouver
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Artigo de periodico
AGEs-induced and endoplasmic reticulum stress/inflammation-mediated regulation of GLUT4 expression and atherogenesis in diabetes mellitus (2022)
Passarelli, Marisa ; Machado, Ubiratan Fabres
Source: Cells. Unidades: FM, ICB
Subjects: FISIOLOGIA, GLICOSE, RETÍCULO ENDOPLASMÁTICO, GORDURAS, DOENÇAS CARDIOVASCULARES, DIABETES MELLITUS, HIPERGLICEMIA, ARTERIOSCLEROSE
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ABNT
PASSARELLI, Marisa e MACHADO, Ubiratan Fabres. AGEs-induced and endoplasmic reticulum stress/inflammation-mediated regulation of GLUT4 expression and atherogenesis in diabetes mellitus. Cells, v. 11, n. 1, p. 1-20, 2022Tradução . . Disponível em: https://doi.org/10.3390/cells11010104. Acesso em: 18 nov. 2024.
APA
Passarelli, M., & Machado, U. F. (2022). AGEs-induced and endoplasmic reticulum stress/inflammation-mediated regulation of GLUT4 expression and atherogenesis in diabetes mellitus. Cells, 11( 1), 1-20. doi:10.3390/cells11010104
NLM
Passarelli M, Machado UF. AGEs-induced and endoplasmic reticulum stress/inflammation-mediated regulation of GLUT4 expression and atherogenesis in diabetes mellitus [Internet]. Cells. 2022 ; 11( 1): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells11010104
Vancouver
Passarelli M, Machado UF. AGEs-induced and endoplasmic reticulum stress/inflammation-mediated regulation of GLUT4 expression and atherogenesis in diabetes mellitus [Internet]. Cells. 2022 ; 11( 1): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells11010104
Artigo de periodico
Long-term supplementation with phenolic compounds from jaboticaba (Plinia jaboticaba (Vell.) Berg) reduces adiposophaty and improves glucose, lipid, and energy metabolism (2021)
Moura, Márcio Hercules Caldas ; Pestana, Carlos M. Donado ; Rodrigues, Larissa ; Pessoa, Érika Vicencia Monteiro ; Silva, Rafaela Rossi e; Festuccia, William Tadeu Lara ; Genovese, Maria Inês
Source: Food Research International. Unidades: ICB, FCF, NUTRIÇÃO HUMANA
Subjects: JABUTICABA, COMPOSTOS FENÓLICOS, RESISTÊNCIA À INSULINA, OBESIDADE, FISIOLOGIA, LIPÍDEOS, POLIFENÓIS, GLICOSE
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ABNT
MOURA, Márcio Hercules Caldas et al. Long-term supplementation with phenolic compounds from jaboticaba (Plinia jaboticaba (Vell.) Berg) reduces adiposophaty and improves glucose, lipid, and energy metabolism. Food Research International, v. 143, p. 1-17 art. 110302, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.foodres.2021.110302. Acesso em: 18 nov. 2024.
APA
Moura, M. H. C., Pestana, C. M. D., Rodrigues, L., Pessoa, É. V. M., Silva, R. R. e, Festuccia, W. T. L., & Genovese, M. I. (2021). Long-term supplementation with phenolic compounds from jaboticaba (Plinia jaboticaba (Vell.) Berg) reduces adiposophaty and improves glucose, lipid, and energy metabolism. Food Research International, 143, 1-17 art. 110302. doi:10.1016/j.foodres.2021.110302
NLM
Moura MHC, Pestana CMD, Rodrigues L, Pessoa ÉVM, Silva RR e, Festuccia WTL, Genovese MI. Long-term supplementation with phenolic compounds from jaboticaba (Plinia jaboticaba (Vell.) Berg) reduces adiposophaty and improves glucose, lipid, and energy metabolism [Internet]. Food Research International. 2021 ; 143 1-17 art. 110302.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.foodres.2021.110302
Vancouver
Moura MHC, Pestana CMD, Rodrigues L, Pessoa ÉVM, Silva RR e, Festuccia WTL, Genovese MI. Long-term supplementation with phenolic compounds from jaboticaba (Plinia jaboticaba (Vell.) Berg) reduces adiposophaty and improves glucose, lipid, and energy metabolism [Internet]. Food Research International. 2021 ; 143 1-17 art. 110302.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.foodres.2021.110302
Artigo de periodico
Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival (2021)
Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher ; Nalbach, Lisa; Almeida, Davidson Correa de ; Ampofo, Emmanuel ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates ; Ortis, Fernanda
Source: Antioxidants. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, PERÓXIDO DE HIDROGÊNIO, CITOCINAS, DIABETES MELLITUS, CAMUNDONGOS, ILHOTAS DE LANGERHANS, GLICOSE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, v. 10, n. 8, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.3390/antiox10081305. Acesso em: 18 nov. 2024.
APA
Vilas-Boas, E. A., Carlein, C., Nalbach, L., Almeida, D. C. de, Ampofo, E., Carpinelli, A. R., et al. (2021). Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, 10( 8), 1-20. doi:10.3390/antiox10081305
NLM
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/antiox10081305
Vancouver
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/antiox10081305
Artigo de periodico
New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia (2021)
Silva, Renée de Nazaré Oliveira da ; Llanos, Ricardo Pariona ; Eichler, Rosangela Aparecida dos Santos ; Oliveira, Thiago Belchior de ; Gozzo, Fábio Cesar ; Festuccia, William Tadeu Lara ; Ferro, Emer Suavinho
Source: Pharmaceutics. Unidade: ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, PEPTÍDEOS, INSULINA, DIABETES MELLITUS, GLICOSE, GLICEMIA, HEMOGLOBINAS, CAMUNDONGOS, CÉLULAS MUSCULARES, EXPRESSÃO GÊNICA, MEMBRANA PLASMÁTICA, TECIDO ADIPOSO
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ABNT
SILVA, Renée de Nazaré Oliveira da et al. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia. Pharmaceutics, v. 13, n. 12, p. 1-18, 2021Tradução . . Disponível em: https://doi.org/10.3390/pharmaceutics13122175. Acesso em: 18 nov. 2024.
APA
Silva, R. de N. O. da, Llanos, R. P., Eichler, R. A. dos S., Oliveira, T. B. de, Gozzo, F. C., Festuccia, W. T. L., & Ferro, E. S. (2021). New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia. Pharmaceutics, 13( 12), 1-18. doi:10.3390/pharmaceutics13122175
NLM
Silva R de NO da, Llanos RP, Eichler RA dos S, Oliveira TB de, Gozzo FC, Festuccia WTL, Ferro ES. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia [Internet]. Pharmaceutics. 2021 ; 13( 12): 1-18.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/pharmaceutics13122175
Vancouver
Silva R de NO da, Llanos RP, Eichler RA dos S, Oliveira TB de, Gozzo FC, Festuccia WTL, Ferro ES. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia [Internet]. Pharmaceutics. 2021 ; 13( 12): 1-18.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/pharmaceutics13122175
Artigo de periodico
Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice (2021)
Castro, Érique de ; Vieira, Thayna dos Santos ; Oliveira, Tiago E.; Silva, Milene Ortiz ; Andrade, Maynara Lucca; Tomazelli, Caroline Aparecida ; Peixoto, Albert Souza ; Sobrinho, Cleyton Roberto ; Moreno, Mayara Franzoi ; Gilio, Gustavo Renan ; Moreira, Rafael Junges ; Guimarães, Raphael Campos ; Perandini, Luiz Augusto Buoro ; Chimin, Patricia ; Reckziegel, Patricia ; Moretti, Eduardo Hermógenes ; Steiner, Alexandre Alarcon ; Laplante, Mathieu ; Festuccia, William Tadeu Lara
Source: AJP: Endocrinology and Metabolism. Unidade: ICB
Subjects: IMUNOLOGIA, FISIOLOGIA, FÁRMACOS IMUNOSSUPRESSORES, TECIDO ADIPOSO, METABOLISMO ENERGÉTICO, EXPRESSÃO GÊNICA, GLICOSE, CAMUNDONGOS, TERMOGÊNESE, GENES, GORDURAS
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ABNT
CASTRO, Érique de et al. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, v. 321, p. E592–E605, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00587.2020. Acesso em: 18 nov. 2024.
APA
Castro, É. de, Vieira, T. dos S., Oliveira, T. E., Silva, M. O., Andrade, M. L., Tomazelli, C. A., et al. (2021). Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, 321, E592–E605. doi:10.1152/ajpendo.00587.2020
NLM
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020
Vancouver
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020
Artigo de periodico
Effects of growth hormone receptor ablation in corticotropin-releasing hormone cells (2021)
Santos, Willian O. dos ; Gusmão, Daniela O. ; Wasinski, Frederick ; List, Edward O. ; Kopchick, John J. ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIOS GLICOCORTICOIDES, HORMÔNIO DO CRESCIMENTO, HIPOTÁLAMO, HORMÔNIOS DO CÓRTEX ADRENAL, NEUROENDOCRINOLOGIA, CAMUNDONGOS, PESOS E MEDIDAS CORPORAIS, INGESTÃO, GLICOSE
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ABNT
SANTOS, Willian O. dos et al. Effects of growth hormone receptor ablation in corticotropin-releasing hormone cells. International Journal of Molecular Sciences, v. 22, n. 18, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.3390/ijms22189908. Acesso em: 18 nov. 2024.
APA
Santos, W. O. dos, Gusmão, D. O., Wasinski, F., List, E. O., Kopchick, J. J., & Donato Junior, J. (2021). Effects of growth hormone receptor ablation in corticotropin-releasing hormone cells. International Journal of Molecular Sciences, 22( 18), 1-15. doi:10.3390/ijms22189908
NLM
Santos WO dos, Gusmão DO, Wasinski F, List EO, Kopchick JJ, Donato Junior J. Effects of growth hormone receptor ablation in corticotropin-releasing hormone cells [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 18): 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms22189908
Vancouver
Santos WO dos, Gusmão DO, Wasinski F, List EO, Kopchick JJ, Donato Junior J. Effects of growth hormone receptor ablation in corticotropin-releasing hormone cells [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 18): 1-15.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/ijms22189908

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