Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice (2022)
- Authors:
- USP affiliated authors: IRIGOYEN, MARIA CLAUDIA COSTA - FM ; DONATO JÚNIOR, JOSÉ - ICB ; CHAVES, MARIA LUIZA DE MORAIS BARRETO DE - ICB ; DINIZ, GABRIELA PLACONÁ - ICB ; MIRANDA, JULIANE BRAGA - ICB ; LUNARDON, GUILHERME - ICB ; SILVA, TÁBATHA DE OLIVEIRA - ICB ; LINO, CAROLINE ANTUNES - ICB ; LIMA, VANESSA MORAIS - ICB
- Unidades: FM; ICB
- DOI: 10.33594/000000535
- Subjects: FISIOLOGIA; ANATOMIA; DIETA ANIMAL; GLICOSE; ISQUEMIA; OBESIDADE; CAMUNDONGOS; DOENÇAS CARDIOVASCULARES EM ANIMAL; DOENÇAS METABÓLICAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Duesseldorf
- Date published: 2022
- Source:
- Título do periódico: Cellular Physiology and Biochemistry
- ISSN: 1421-9778
- Volume/Número/Paginação/Ano: v. 56, n. 3, p. 293-309, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc-nd
-
ABNT
MIRANDA, Juliane Braga et al. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, v. 56, n. 3, p. 293-309, 2022Tradução . . Disponível em: https://doi.org/10.33594/000000535. Acesso em: 23 abr. 2024. -
APA
Miranda, J. B., Lunardon, G., Lima, V. M., Silva, T. de O., Lino, C. A., Jensen, L., et al. (2022). Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, 56( 3), 293-309. doi:10.33594/000000535 -
NLM
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 abr. 23 ] Available from: https://doi.org/10.33594/000000535 -
Vancouver
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 abr. 23 ] Available from: https://doi.org/10.33594/000000535 - The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy
- Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice
- Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction
- Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia
- High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury
- Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy
- Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders
- Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R
- miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction
- Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R)
Informações sobre o DOI: 10.33594/000000535 (Fonte: oaDOI API)
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