Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction (2022)
- Authors:
- USP affiliated authors: IRIGOYEN, MARIA CLAUDIA COSTA - FM ; TAKANO, ANA PAULA CREMASCO - ICB ; CHAVES, MARIA LUIZA DE MORAIS BARRETO DE - ICB ; DINIZ, GABRIELA PLACONÁ - ICB ; LUNARDON, GUILHERME - ICB ; SILVA, TÁBATHA DE OLIVEIRA - ICB ; LINO, CAROLINE ANTUNES - ICB ; MIRANDA, JULIANE BRAGA - ICB ; SILVA, AMANDA DE ALMEIDA - FM
- Unidades: FM; ICB
- DOI: 10.1042/CS20220466
- Subjects: ANATOMIA; HIPERTROFIA VENTRICULAR ESQUERDA; CARDIOMEGALIA; CARDIOPATIAS; MIOCARDIOPATIAS; CÉLULAS MUSCULARES; CAMUNDONGOS; DNA MITOCONDRIAL
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Clinical Science (Online): an international journal of translational
- ISSN: 1470-8736
- Volume/Número/Paginação/Ano: v. 136, p. 1537–1554, 2022
- Status:
- Artigo aberto em periódico híbrido (Hybrid Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
LUNARDON, Guilherme et al. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, v. 136, p. 1537–1554, 2022Tradução . . Disponível em: https://doi.org/10.1042/CS20220466. Acesso em: 01 abr. 2026. -
APA
Lunardon, G., Silva, T. de O., Lino, C. A., Miranda, J. B., Silva, A. de A., Irigoyen, M. C. C., et al. (2022). Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, 136, 1537–1554. doi:10.1042/CS20220466 -
NLM
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1042/CS20220466 -
Vancouver
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1042/CS20220466 - Role of Set7 in cardiac disorders
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- Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R
- Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice
- miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction
- Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R)
- Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism
- Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor
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