Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress (2021)
- Authors:
- USP affiliated authors: ORTIS, FERNANDA - ICB ; CARPINELLI, ANGELO RAFAEL - ICB ; BÔAS, ELOÍSA APARECIDA VILAS - IQ ; ALMEIDA, DAVIDSON CORREA DE - ICB
- Unidades: ICB; IQ
- DOI: 10.3390/cells10123328
- Subjects: FISIOLOGIA; BIOLOGIA CELULAR; ESTRESSE OXIDATIVO; ILHOTAS DE LANGERHANS; DIABETES MELLITUS; LIPÍDEOS; MEMBRANA PLASMÁTICA; OBESIDADE; ÁCIDOS GRAXOS; INSULINA; GLICOSE; CÉLULAS MORTAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
BOAS, Eloisa Aparecida Vilas et al. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, v. 10, n. 12, p. 1-23, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10123328. Acesso em: 23 abr. 2024. -
APA
Boas, E. A. V., Almeida, D. C., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2021). Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, 10( 12), 1-23. doi:10.3390/cells10123328 -
NLM
Boas EAV, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 abr. 23 ] Available from: https://doi.org/10.3390/cells10123328 -
Vancouver
Boas EAV, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 abr. 23 ] Available from: https://doi.org/10.3390/cells10123328 - Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival
- Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets
- A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells
- Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS
- Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells
- Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function
- Expressão, localização e modulação de NADPH oxidases por citocinas pró-inflamatórias na célula β pancreática
- Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling
- Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII
- The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro
Informações sobre o DOI: 10.3390/cells10123328 (Fonte: oaDOI API)
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