Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS (2019)
- Authors:
- USP affiliated authors: ORTIS, FERNANDA - ICB ; CARPINELLI, ANGELO RAFAEL - ICB ; BÔAS, ELOÍSA APARECIDA VILAS - ICB ; ROMA, LETICIA PRATES - ICB
- Unidade: ICB
- DOI: 10.1007/s00125-019-4946-6
- Subjects: FISIOLOGIA; BIOLOGIA CELULAR; CITOCINAS; INFLAMAÇÃO; CÉLULAS MORTAS; PÂNCREAS; INSULINA; MITOCÔNDRIAS; CAMUNDONGOS; CITOMETRIA DE FLUXO; WESTERN BLOTTING; ILHOTAS DE LANGERHANS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Springer
- Publisher place: Heidelberg
- Date published: 2019
- Source:
- Título: Diabetologia
- ISSN: 1432-0428
- Volume/Número/Paginação/Ano: v. 62, S1, p. S221-S222, res. 450, 2019
- Conference titles: EASD Annual Meeting of the European Association for the Study of Diabetes
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
VILAS-BOAS, Eloisa Aparecida et al. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS. Diabetologia. Heidelberg: Springer. Disponível em: https://doi.org/10.1007/s00125-019-4946-6. Acesso em: 11 fev. 2026. , 2019 -
APA
Vilas-Boas, E. A., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2019). Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS. Diabetologia. Heidelberg: Springer. doi:10.1007/s00125-019-4946-6 -
NLM
Vilas-Boas EA, Roma LP, Ortis F, Carpinelli AR. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS [Internet]. Diabetologia. 2019 ; 62 S221-S222.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1007/s00125-019-4946-6 -
Vancouver
Vilas-Boas EA, Roma LP, Ortis F, Carpinelli AR. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS [Internet]. Diabetologia. 2019 ; 62 S221-S222.[citado 2026 fev. 11 ] Available from: https://doi.org/10.1007/s00125-019-4946-6 - Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets
- A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells
- Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress
- Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells
- Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function
- Expression, localization and regulation of NADPH oxidases in pancreatic beta cells
- Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival
- The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro
- Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation
- The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes
Informações sobre o DOI: 10.1007/s00125-019-4946-6 (Fonte: oaDOI API)
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