Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation (2016)
- Authors:
- Autor USP: ORTIS, FERNANDA - ICB
- Unidade: ICB
- DOI: 10.1530/JME-15-0306
- Assunto: HISTOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Molecular Endocrinology
- ISSN: 1479-6813
- Volume/Número/Paginação/Ano: v. 57, n. 1, p. R1-R17, 2016
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
MEYEROVICH, Kira et al. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, v. 57, n. 1, p. R1-R17, 2016Tradução . . Disponível em: https://doi.org/10.1530/JME-15-0306. Acesso em: 27 dez. 2025. -
APA
Meyerovich, K., Ortis, F., Allagnat, F., & Cardozo, A. K. (2016). Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. Journal of Molecular Endocrinology, 57( 1), R1-R17. doi:10.1530/JME-15-0306 -
NLM
Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1530/JME-15-0306 -
Vancouver
Meyerovich K, Ortis F, Allagnat F, Cardozo AK. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation [Internet]. Journal of Molecular Endocrinology. 2016 ; 57( 1): R1-R17.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1530/JME-15-0306 - The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro
- Papel de BRG1 e Brm, reguladores globais de transcrição, na reversão fenotípica de células ST1 pela ação de glicocorticóides
- Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling
- Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII
- Transporte através de membranas celulares e tráfego intracelular
- Retículo endoplasmático e Complexo de Golgi
- Membranas celulares
- The non-canonical NF-κB pathway and its contribution to β-cell failure in diabetes
- ARHGAP21 acts as an inhibitor of the glucose-stimulated insulin secretion process
- Prolactin protects against cytokine-induced beta-cell death by NFκB and JNK inhibition
Informações sobre o DOI: 10.1530/JME-15-0306 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
