DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress (2020)
- Authors:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- DOI: 10.1158/1538-7445.AM2020-2372
- Subjects: PROTEÍNAS NUCLEARES; GENÔMICA
- Language: Inglês
- Imprenta:
- Publisher: American Association for Cancer Research (AACR)
- Publisher place: Philadelphia
- Date published: 2020
- Source:
- Título: Cancer Research
- ISSN: 0008-5472
- Volume/Número/Paginação/Ano: v. 80, n. 16, Suppl., res. 2372, 2020
- Conference titles: AACR Annual Meeting
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
FORTI, Fabio Luis e RUSSO, Lilian Cristina. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress. Cancer Research. Philadelphia: American Association for Cancer Research (AACR). Disponível em: https://doi.org/10.1158/1538-7445.AM2020-2372. Acesso em: 18 fev. 2026. , 2020 -
APA
Forti, F. L., & Russo, L. C. (2020). DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress. Cancer Research. Philadelphia: American Association for Cancer Research (AACR). doi:10.1158/1538-7445.AM2020-2372 -
NLM
Forti FL, Russo LC. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress [Internet]. Cancer Research. 2020 ; 80( 16):[citado 2026 fev. 18 ] Available from: https://doi.org/10.1158/1538-7445.AM2020-2372 -
Vancouver
Forti FL, Russo LC. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress [Internet]. Cancer Research. 2020 ; 80( 16):[citado 2026 fev. 18 ] Available from: https://doi.org/10.1158/1538-7445.AM2020-2372 - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
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- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions
Informações sobre o DOI: 10.1158/1538-7445.AM2020-2372 (Fonte: oaDOI API)
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