The small GTPase RhoA signals to DNA damage repair in tumor cells (2016)
- Authors:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- Subjects: DANO AO DNA; NEOPLASIAS
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Biologia Celular (SBBC)
- Publisher place: São Paulo
- Date published: 2016
- Source:
- Título: Resumos
- Conference titles: Congresso da Sociedade Brasileira de Biologia Celular
-
ABNT
MAGALHÃES, Yuli Thamires e FORTI, Fabio Luis. The small GTPase RhoA signals to DNA damage repair in tumor cells. 2016, Anais.. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC), 2016. . Acesso em: 05 fev. 2026. -
APA
Magalhães, Y. T., & Forti, F. L. (2016). The small GTPase RhoA signals to DNA damage repair in tumor cells. In Resumos. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC). -
NLM
Magalhães YT, Forti FL. The small GTPase RhoA signals to DNA damage repair in tumor cells. Resumos. 2016 ;[citado 2026 fev. 05 ] -
Vancouver
Magalhães YT, Forti FL. The small GTPase RhoA signals to DNA damage repair in tumor cells. Resumos. 2016 ;[citado 2026 fev. 05 ] - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
- Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells
- Clonagem do receptor de ACTH de células adrenocorticais Y-1 de camundongos e expressão em fivroblastos 3T3 e celulas AR-1 para elucidação de vias de transdução de sinal
- Proteomic and interactome approaches reveal PAK4, PHB-2, and 14-3-3 eta as targets of overactivated Cdc42 in cellular responses to Genomic instability
- The use of HeLa cells as a model for studying DNA damage and repair
- Revisiting the roles of VHR/DUSP3 phosphatase in human diseases
- DUSP3 Signals for proliferation and genomic stability in human cells
- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions
- Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents
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