Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells (2018)
- Authors:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- DOI: 10.1007/978-1-4939-8612-5_23
- Subjects: DNA; CÉLULAS EPITELIAIS
- Language: Inglês
- Imprenta:
- Publisher: Humana Press
- Publisher place: New York
- Date published: 2018
- Source:
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MAGALHÃES, Yuli Thamires et al. Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells. Rho GTPases: Methods and Protocols. Tradução . New York: Humana Press, 2018. . Disponível em: https://doi.org/10.1007/978-1-4939-8612-5_23. Acesso em: 13 fev. 2026. -
APA
Magalhães, Y. T., Farias, J. O., Monteiro, L. F., & Forti, F. L. (2018). Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells. In Rho GTPases: Methods and Protocols. New York: Humana Press. doi:10.1007/978-1-4939-8612-5_23 -
NLM
Magalhães YT, Farias JO, Monteiro LF, Forti FL. Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells [Internet]. In: Rho GTPases: Methods and Protocols. New York: Humana Press; 2018. [citado 2026 fev. 13 ] Available from: https://doi.org/10.1007/978-1-4939-8612-5_23 -
Vancouver
Magalhães YT, Farias JO, Monteiro LF, Forti FL. Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells [Internet]. In: Rho GTPases: Methods and Protocols. New York: Humana Press; 2018. [citado 2026 fev. 13 ] Available from: https://doi.org/10.1007/978-1-4939-8612-5_23 - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
- Clonagem do receptor de ACTH de células adrenocorticais Y-1 de camundongos e expressão em fivroblastos 3T3 e celulas AR-1 para elucidação de vias de transdução de sinal
- Proteomic and interactome approaches reveal PAK4, PHB-2, and 14-3-3 eta as targets of overactivated Cdc42 in cellular responses to Genomic instability
- The use of HeLa cells as a model for studying DNA damage and repair
- Revisiting the roles of VHR/DUSP3 phosphatase in human diseases
- The small GTPase RhoA signals to DNA damage repair in tumor cells
- DUSP3 Signals for proliferation and genomic stability in human cells
- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions
- Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents
Informações sobre o DOI: 10.1007/978-1-4939-8612-5_23 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
