Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions (2019)
- Authors:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- Assunto: GENÔMICA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq
- Publisher place: São Paulo
- Date published: 2019
- Source:
- Título: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq
-
ABNT
SILVA, Luiz Eduardo e SOUZA, Renan de e FORTI, Fabio Luis. Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions. 2019, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq, 2019. Disponível em: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf. Acesso em: 06 fev. 2026. -
APA
Silva, L. E., Souza, R. de, & Forti, F. L. (2019). Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq. Recuperado de http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf -
NLM
Silva LE, Souza R de, Forti FL. Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions [Internet]. Abstracts. 2019 ;[citado 2026 fev. 06 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf -
Vancouver
Silva LE, Souza R de, Forti FL. Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions [Internet]. Abstracts. 2019 ;[citado 2026 fev. 06 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
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- Proteomic and interactome approaches reveal PAK4, PHB-2, and 14-3-3 eta as targets of overactivated Cdc42 in cellular responses to Genomic instability
- The use of HeLa cells as a model for studying DNA damage and repair
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- The small GTPase RhoA signals to DNA damage repair in tumor cells
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- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents
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