CDC42 gtpase activation affects hela cell DNA repair and proliferation following UV radiation-induced genotoxic stress (2015)
- Authors:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- DOI: 10.1002/jcb.25166
- Subjects: PROLIFERAÇÃO CELULAR; DANO AO DNA; RADIAÇÃO ULTRAVIOLETA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Cellular Biochemistry
- ISSN: 0730-2312
- Volume/Número/Paginação/Ano: v. 116, n. 9, p. 2086-2097, 2015
- Status:
- Nenhuma versão em acesso aberto identificada
-
ABNT
ASCER, Liv Goldstein et al. CDC42 gtpase activation affects hela cell DNA repair and proliferation following UV radiation-induced genotoxic stress. Journal of Cellular Biochemistry, v. 116, n. 9, p. 2086-2097, 2015Tradução . . Disponível em: https://doi.org/10.1002/jcb.25166. Acesso em: 18 mar. 2026. -
APA
Ascer, L. G., Magalhães, Y. T., Espinha, G. T. da S., Osaki, J. H., Souza, R. C. de, & Forti, F. L. (2015). CDC42 gtpase activation affects hela cell DNA repair and proliferation following UV radiation-induced genotoxic stress. Journal of Cellular Biochemistry, 116( 9), 2086-2097. doi:10.1002/jcb.25166 -
NLM
Ascer LG, Magalhães YT, Espinha GT da S, Osaki JH, Souza RC de, Forti FL. CDC42 gtpase activation affects hela cell DNA repair and proliferation following UV radiation-induced genotoxic stress [Internet]. Journal of Cellular Biochemistry. 2015 ; 116( 9): 2086-2097.[citado 2026 mar. 18 ] Available from: https://doi.org/10.1002/jcb.25166 -
Vancouver
Ascer LG, Magalhães YT, Espinha GT da S, Osaki JH, Souza RC de, Forti FL. CDC42 gtpase activation affects hela cell DNA repair and proliferation following UV radiation-induced genotoxic stress [Internet]. Journal of Cellular Biochemistry. 2015 ; 116( 9): 2086-2097.[citado 2026 mar. 18 ] Available from: https://doi.org/10.1002/jcb.25166 - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
- Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells
- Proteomic and interactome approaches reveal PAK4, PHB-2, and 14-3-3 eta as targets of overactivated Cdc42 in cellular responses to Genomic instability
- The use of HeLa cells as a model for studying DNA damage and repair
- Revisiting the roles of VHR/DUSP3 phosphatase in human diseases
- The small GTPase RhoA signals to DNA damage repair in tumor cells
- DUSP3 Signals for proliferation and genomic stability in human cells
- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions
- Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents
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