Combined experimental and bioinformatics analysis for the prediction and identification of VHR/DUSP3 nuclear targets related to DNA damage and repair (2015)
- Autor:
- Autor USP: FORTI, FÁBIO LUÍS - IQ
- Unidade: IQ
- DOI: 10.1039/c4ib00186a
- Subjects: BIOINFORMÁTICA; DANO AO DNA
- Language: Inglês
- Imprenta:
- Source:
- Título: Integrative Biology
- ISSN: 1757-9694
- Volume/Número/Paginação/Ano: v. 7, n. 1, p. 73-89, 2015
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
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ABNT
FORTI, Fabio Luis. Combined experimental and bioinformatics analysis for the prediction and identification of VHR/DUSP3 nuclear targets related to DNA damage and repair. Integrative Biology, v. 7, n. 1, p. 73-89, 2015Tradução . . Disponível em: https://doi.org/10.1039/c4ib00186a. Acesso em: 18 mar. 2026. -
APA
Forti, F. L. (2015). Combined experimental and bioinformatics analysis for the prediction and identification of VHR/DUSP3 nuclear targets related to DNA damage and repair. Integrative Biology, 7( 1), 73-89. doi:10.1039/c4ib00186a -
NLM
Forti FL. Combined experimental and bioinformatics analysis for the prediction and identification of VHR/DUSP3 nuclear targets related to DNA damage and repair [Internet]. Integrative Biology. 2015 ; 7( 1): 73-89.[citado 2026 mar. 18 ] Available from: https://doi.org/10.1039/c4ib00186a -
Vancouver
Forti FL. Combined experimental and bioinformatics analysis for the prediction and identification of VHR/DUSP3 nuclear targets related to DNA damage and repair [Internet]. Integrative Biology. 2015 ; 7( 1): 73-89.[citado 2026 mar. 18 ] Available from: https://doi.org/10.1039/c4ib00186a - Investigação estrutural da via de transdução de sinal da proteína SRC: interação de CSK com caveolina-1 depende do estado redox do domínio SH2
- Measuring the contributions of the Rho pathway to the DNA damage response in tumor epithelial cells
- Proteomic and interactome approaches reveal PAK4, PHB-2, and 14-3-3 eta as targets of overactivated Cdc42 in cellular responses to Genomic instability
- The use of HeLa cells as a model for studying DNA damage and repair
- Revisiting the roles of VHR/DUSP3 phosphatase in human diseases
- The small GTPase RhoA signals to DNA damage repair in tumor cells
- DUSP3 Signals for proliferation and genomic stability in human cells
- DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins
- Non-canonical role of Cdc42 GTPase and its novel interaction partners in genomic instability conditions
- Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents
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