Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS (2010)
- Authors:
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Instituto Israelita de Ensino e Pesquisa
- Publisher place: São Paulo
- Date published: 2010
- Source:
- Título: Einstein
- Volume/Número/Paginação/Ano: v. 8, supl 1, p. S47, res. 045, 2010
- Conference titles: Signaling in cell death, cancer and the immune system
-
ABNT
LIMA, Carina Buzzo de et al. Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS. Einstein. São Paulo: Instituto Israelita de Ensino e Pesquisa. . Acesso em: 25 jan. 2026. , 2010 -
APA
Lima, C. B. de, Campopiano, J. C., Cassado, A. dos A., Leme-Souza, R., Massis, L., Cunha, L. D. da, et al. (2010). Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS. Einstein. São Paulo: Instituto Israelita de Ensino e Pesquisa. -
NLM
Lima CB de, Campopiano JC, Cassado A dos A, Leme-Souza R, Massis L, Cunha LD da, Zamboni DS, Bortoluci KR, Amarante-Mendes JGP, Russo M. Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS. Einstein. 2010 ; 8 S47.[citado 2026 jan. 25 ] -
Vancouver
Lima CB de, Campopiano JC, Cassado A dos A, Leme-Souza R, Massis L, Cunha LD da, Zamboni DS, Bortoluci KR, Amarante-Mendes JGP, Russo M. Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS. Einstein. 2010 ; 8 S47.[citado 2026 jan. 25 ] - Regulação do CD95L por PGE2 e seu impacto na morte de linfócitos T.
- A suggestive role of NAIP5 on INOS activation
- A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages
- TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells
- A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1
- PGE2 derivada de macrófagos ou células dendríticas protege linfócitos T da AICD bloqueando a expressão de FasL
- Inhibition of interferon-'gama'-induced nitric oxide production in endotoxin-activated macrophages by cytolethal distending toxin
- Cross-talk between innate and adaptive immunity: TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3- mediated apoptosis of T cells
- Mechanism of suppression of T cell AICD mediated by macrophage- and DC-derived prostaglandin 'E IND.2'
- Macrophage- and DC-derived PGE2 protects T cells from AICD by preventing FasL upregulation
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