A suggestive role of NAIP5 on INOS activation (2008)
- Authors:
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2008
- Source:
- Título do periódico: Resumos
- Conference titles: Congress of the Brazilian Society for Immunology - SBI
-
ABNT
BUZZO, Carina L. et al. A suggestive role of NAIP5 on INOS activation. 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 02 ago. 2024. -
APA
Buzzo, C. L., Campopiano, J. C., Souza, R. L. C., Cassado, A., Massis, L., Russo, M., et al. (2008). A suggestive role of NAIP5 on INOS activation. In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Buzzo CL, Campopiano JC, Souza RLC, Cassado A, Massis L, Russo M, Amarante-Mendes JGP, Bortoluci KR. A suggestive role of NAIP5 on INOS activation. Resumos. 2008 ;[citado 2024 ago. 02 ] -
Vancouver
Buzzo CL, Campopiano JC, Souza RLC, Cassado A, Massis L, Russo M, Amarante-Mendes JGP, Bortoluci KR. A suggestive role of NAIP5 on INOS activation. Resumos. 2008 ;[citado 2024 ago. 02 ] - A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages
- TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells
- Regulação do CD95L por PGE2 e seu impacto na morte de linfócitos T.
- Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS
- A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1
- Inhibition of interferon-'gama'-induced nitric oxide production in endotoxin-activated macrophages by cytolethal distending toxin
- Cross-talk between innate and adaptive immunity: TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3- mediated apoptosis of T cells
- A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes
- FASL upregulation and T cell death by AICD are prevented by macrophage- and DC-derived PGE2
- PGE2 derivada de macrófagos ou células dendríticas protege linfócitos T da AICD bloqueando a expressão de FasL
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