A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages (2008)
- Authors:
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Comissão de Cultura e Extensão Universitária do ICB/USP
- Publisher place: São Paulo
- Date published: 2008
- Source:
- Título: Resumos
- Conference titles: Congresso do Instituto de Ciências Biomédicas da Universidade de São Paulo
-
ABNT
BUZZO, C. L et al. A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages. 2008, Anais.. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP, 2008. . Acesso em: 25 jan. 2026. -
APA
Buzzo, C. L., Camponiano J. C,, Leme-Souza, R., Cassado, A. A., Massis, L., Russo, M., et al. (2008). A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages. In Resumos. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP. -
NLM
Buzzo CL, Camponiano J. C, Leme-Souza R, Cassado AA, Massis L, Russo M, Amarante-Mendes JGP, Bortoluci KR. A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages. Resumos. 2008 ;[citado 2026 jan. 25 ] -
Vancouver
Buzzo CL, Camponiano J. C, Leme-Souza R, Cassado AA, Massis L, Russo M, Amarante-Mendes JGP, Bortoluci KR. A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages. Resumos. 2008 ;[citado 2026 jan. 25 ] - Regulação do CD95L por PGE2 e seu impacto na morte de linfócitos T.
- Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS
- A suggestive role of NAIP5 on INOS activation
- TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells
- A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1
- PGE2 derivada de macrófagos ou células dendríticas protege linfócitos T da AICD bloqueando a expressão de FasL
- Inhibition of interferon-'gama'-induced nitric oxide production in endotoxin-activated macrophages by cytolethal distending toxin
- Cross-talk between innate and adaptive immunity: TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3- mediated apoptosis of T cells
- Mechanism of suppression of T cell AICD mediated by macrophage- and DC-derived prostaglandin 'E IND.2'
- Macrophage- and DC-derived PGE2 protects T cells from AICD by preventing FasL upregulation
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