Aggressive adrenocortical carcinoma producer estrogen and inhibin with a select supression of FSH (2001)
- Authors:
- Autor USP: MENDONCA, BERENICE BILHARINHO DE - FM
- Unidade: FM
- Subjects: CARCINOMA; ENDOCRINOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Program & Abstracts
- Conference titles: Annual Meeting of the Endocrine Society's
-
ABNT
FRAGOSO, M. B. et al. Aggressive adrenocortical carcinoma producer estrogen and inhibin with a select supression of FSH. 2001, Anais.. Denver: Faculdade de Medicina, Universidade de São Paulo, 2001. . Acesso em: 07 out. 2024. -
APA
Fragoso, M. B., Martin, R. M., Kohek, M. F., Domenice, S., & Mendonça, B. B. (2001). Aggressive adrenocortical carcinoma producer estrogen and inhibin with a select supression of FSH. In Program & Abstracts. Denver: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Fragoso MB, Martin RM, Kohek MF, Domenice S, Mendonça BB. Aggressive adrenocortical carcinoma producer estrogen and inhibin with a select supression of FSH. Program & Abstracts. 2001 ;[citado 2024 out. 07 ] -
Vancouver
Fragoso MB, Martin RM, Kohek MF, Domenice S, Mendonça BB. Aggressive adrenocortical carcinoma producer estrogen and inhibin with a select supression of FSH. Program & Abstracts. 2001 ;[citado 2024 out. 07 ] - Molecular analysis of SRY gene in Brazilian 46,XX sex reversed patients: absense of SRY sequence in gonadal tissue
- Variable ACTH-stimulated 17-Hydroxyprogesterone values in 21-hydroxylase deficiency carriers are not related to the different CYP21 gene mutations
- Absense of duplication on DAX1 gene in patients with 46,XY sex reversal
- Crescimento linear e estatura final em crianças com tumores adrenais predominantemente virilizantes
- Absense of inactivating mutations and deletions of DAX1 gene in SRY negative 46,XX sex-reversed patients
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- Clinical, hormonal, behavioral, and genetic characteristics of androgen insensitibity syndrome in a brazilian cohort: five novel mutations in the androgen receptor gene
- Reavaliação na idade adulta do diagnóstico da deficiência de GH (DGH) determinado na infância
- Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty
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