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Artigo de periodico
Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice (2022)
Veras, Katherine Maria de Araujo ; Monaco, Camila Ferraz Lucena ; Goedcke, Julia; Evangelista, Fabiana de Sant'Anna ; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Source: Frontiers in Endocrinology. Unidades: EACH, ICB
Subjects: FISIOLOGIA, PROTEÍNAS DO LEITE, DIETA, EXERCÍCIO FÍSICO, TREINAMENTO AERÓBIO, SACAROSE, ILHOTAS DE LANGERHANS, RESISTÊNCIA À INSULINA, GLICOSE, OBESIDADE, CAMUNDONGOS
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VERAS, Katherine Maria de Araujo et al. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, v. 13, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fendo.2022.881236. Acesso em: 06 nov. 2024.
APA
Veras, K. M. de A., Monaco, C. F. L., Goedcke, J., Evangelista, F. de S. 'A., Carpinelli, A. R., & Carvalho, C. R. de O. (2022). Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, 13, 1-12. doi:10.3389/fendo.2022.881236
NLM
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Vancouver
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Artigo de periodico
BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 (2022)
Jesus, Daniel Simões de; Souza, Paula Bargi ; Cruzat, Vinicius ; Yechoor, Vijay; Carpinelli, Angelo Rafael ; Garcia, Rodrigo Antonio Peliciari
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, PÂNCREAS, INSULINA, RITMOS BIOLÓGICOS, SUPERÓXIDO DISMUTASE, FATORES DE TRANSCRIÇÃO, GLICOSE, CAMUNDONGOS, PERÓXIDO DE HIDROGÊNIO, GLUTATIONA TRANSFERASE
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JESUS, Daniel Simões de et al. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, v. 555, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2022.111725. Acesso em: 06 nov. 2024.
APA
Jesus, D. S. de, Souza, P. B., Cruzat, V., Yechoor, V., Carpinelli, A. R., & Garcia, R. A. P. (2022). BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, 555, 1-8. doi:10.1016/j.mce.2022.111725
NLM
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Vancouver
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Artigo de periodico
Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro (2022)
Valle, Maíra Mello Rezende ; Vilas-Boas, Eloisa Aparecida ; Lucena, Camila Ferraz ; Teixeira, Simone Aparecida ; Muscará, Marcelo Nicolás (Orientador) ; Carpinelli, Angelo Rafael
Source: Journal of Biochemical and Molecular Toxicology. Unidade: ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, METFORMINA, ANTIOXIDANTES, DIABETES MELLITUS, INSULINA, ESTRESSE OXIDATIVO, RATOS
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VALLE, Maíra Mello Rezende et al. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro. Journal of Biochemical and Molecular Toxicology, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1002/jbt.23007. Acesso em: 06 nov. 2024.
APA
Valle, M. M. R., Vilas-Boas, E. A., Lucena, C. F., Teixeira, S. A., & Carpinelli, A. R. (2022). Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro. Journal of Biochemical and Molecular Toxicology, 1-13. doi:10.1002/jbt.23007
NLM
Valle MMR, Vilas-Boas EA, Lucena CF, Teixeira SA, Carpinelli AR. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro [Internet]. Journal of Biochemical and Molecular Toxicology. 2022 ; 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/jbt.23007
Vancouver
Valle MMR, Vilas-Boas EA, Lucena CF, Teixeira SA, Carpinelli AR. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro [Internet]. Journal of Biochemical and Molecular Toxicology. 2022 ; 1-13.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/jbt.23007
Artigo de periodico
Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival (2021)
Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher ; Nalbach, Lisa; Almeida, Davidson Correa de ; Ampofo, Emmanuel ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates ; Ortis, Fernanda
Source: Antioxidants. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, PERÓXIDO DE HIDROGÊNIO, CITOCINAS, DIABETES MELLITUS, CAMUNDONGOS, ILHOTAS DE LANGERHANS, GLICOSE
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VILAS-BOAS, Eloisa Aparecida et al. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, v. 10, n. 8, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.3390/antiox10081305. Acesso em: 06 nov. 2024.
APA
Vilas-Boas, E. A., Carlein, C., Nalbach, L., Almeida, D. C. de, Ampofo, E., Carpinelli, A. R., et al. (2021). Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, 10( 8), 1-20. doi:10.3390/antiox10081305
NLM
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/antiox10081305
Vancouver
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/antiox10081305
Artigo de periodico
Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets (2021)
Vilas-Boas, Eloisa Aparecida ; Nalbach, Lisa; Ampofo, Emmanuel; Lucena, Camila Ferraz ; Naudet, Léa ; Morgan, Bruce ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, OBESIDADE, DIABETES MELLITUS, HÁBITOS ALIMENTARES, ÁCIDOS GRAXOS, ILHOTAS DE LANGERHANS, ESTRESSE OXIDATIVO, CAMUNDONGOS, INSULINA, HOMEOSTASE
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VILAS-BOAS, Eloisa Aparecida et al. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, v. 162, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.11.023. Acesso em: 06 nov. 2024.
APA
Vilas-Boas, E. A., Nalbach, L., Ampofo, E., Lucena, C. F., Naudet, L., Morgan, B., et al. (2021). Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, 162, 13 . doi:10.1016/j.freeradbiomed.2020.11.023
NLM
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Vancouver
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Trabalho de evento-resumo periodico
A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells (2021)
Vilas-Boas, Eloisa Aparecida ; Ortis, Fernanda ; Roma, Leticia Prates ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Conference titles: Annual Meeting Abstracts "Redox Biology in the 21. Century". Unidades: ICB, IQ
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, ILHOTAS DE LANGERHANS, ATIVAÇÃO ENZIMÁTICA, OXIDAÇÃO, INFLAMAÇÃO, ENZIMAS OXIRREDUTORAS, RADICAIS LIVRES
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VILAS-BOAS, Eloisa Aparecida et al. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.freeradbiomed.2021.08.173. Acesso em: 06 nov. 2024. , 2021
APA
Vilas-Boas, E. A., Ortis, F., Roma, L. P., & Carpinelli, A. R. (2021). A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.freeradbiomed.2021.08.173
NLM
Vilas-Boas EA, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173
Vancouver
Vilas-Boas EA, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173
Artigo de periodico
Melatonin regulates maternal pancreatic remodeling and Β‐cell function during pregnancy and lactation (2021)
Gomes, Patricia Rodrigues Lourenço ; Vilas-Boas, Eloisa Aparecida ; Leite, Eduardo de Almeida; Bonassa, Ana Cláudia Munhoz ; Monaco, Camila Ferraz Lucena ; Amaral, Fernanda Gaspar do ; Carpinelli, Angelo Rafael ; Cipolla Neto, José
Source: Journal of Pineal Research. Unidades: ICB, IQ
Subjects: FISIOLOGIA, MELATONINA, HORMÔNIO DA GLÂNDULA PINEAL, ILHOTAS DE LANGERHANS, GRAVIDEZ, LACTAÇÃO, RATOS, METABOLISMO ENERGÉTICO, METABOLISMO ANIMAL, ÍNSULA DO PÂNCREAS ANIMAL, GLÂNDULA PINEAL
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GOMES, Patricia Rodrigues Lourenço et al. Melatonin regulates maternal pancreatic remodeling and Β‐cell function during pregnancy and lactation. Journal of Pineal Research, v. 71, n. 1, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1111/jpi.12717. Acesso em: 06 nov. 2024.
APA
Gomes, P. R. L., Vilas-Boas, E. A., Leite, E. de A., Bonassa, A. C. M., Monaco, C. F. L., Amaral, F. G. do, et al. (2021). Melatonin regulates maternal pancreatic remodeling and Β‐cell function during pregnancy and lactation. Journal of Pineal Research, 71( 1), 1-15. doi:10.1111/jpi.12717
NLM
Gomes PRL, Vilas-Boas EA, Leite E de A, Bonassa ACM, Monaco CFL, Amaral FG do, Carpinelli AR, Cipolla Neto J. Melatonin regulates maternal pancreatic remodeling and Β‐cell function during pregnancy and lactation [Internet]. Journal of Pineal Research. 2021 ; 71( 1): 1-15.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1111/jpi.12717
Vancouver
Gomes PRL, Vilas-Boas EA, Leite E de A, Bonassa ACM, Monaco CFL, Amaral FG do, Carpinelli AR, Cipolla Neto J. Melatonin regulates maternal pancreatic remodeling and Β‐cell function during pregnancy and lactation [Internet]. Journal of Pineal Research. 2021 ; 71( 1): 1-15.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1111/jpi.12717
Artigo de periodico
Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress (2021)
Vilas-Boas, Eloisa Aparecida ; Almeida, Davidson Correa ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Cells. Unidades: ICB, IQ
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, LIPÍDEOS, MEMBRANA PLASMÁTICA, OBESIDADE, ÁCIDOS GRAXOS, INSULINA, GLICOSE, CÉLULAS MORTAS
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, v. 10, n. 12, p. 1-23, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10123328. Acesso em: 06 nov. 2024.
APA
Vilas-Boas, E. A., Almeida, D. C., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2021). Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, 10( 12), 1-23. doi:10.3390/cells10123328
NLM
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/cells10123328
Vancouver
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/cells10123328
Artigo de periodico
The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway (2020)
Costa, Ana Carolina Panveloski ; Kuwabara, Wilson Mitsuo Tatagiba ; Bonassa, Ana Cláudia Munhoz ; Monaco, Camila Ferraz Lucena ; Curi, Rui ; Carpinelli, Angelo Rafael ; Nunes, Maria Tereza
Source: Endocrine. Unidades: ICB, IQ
Subjects: FISIOLOGIA, BIOFÍSICA, HORMÔNIOS TIREOIDIANOS, RESISTÊNCIA À INSULINA, RATOS, CITOCININAS, DIABETES MELLITUS, INFLAMAÇÃO, GLICOSE, TECIDO ADIPOSO
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ABNT
COSTA, Ana Carolina Panveloski et al. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway. Endocrine, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1007/s12020-020-02208-5. Acesso em: 06 nov. 2024.
APA
Costa, A. C. P., Kuwabara, W. M. T., Bonassa, A. C. M., Monaco, C. F. L., Curi, R., Carpinelli, A. R., & Nunes, M. T. (2020). The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway. Endocrine, 9 . doi:10.1007/s12020-020-02208-5
NLM
Costa ACP, Kuwabara WMT, Bonassa ACM, Monaco CFL, Curi R, Carpinelli AR, Nunes MT. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway [Internet]. Endocrine. 2020 ;9 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s12020-020-02208-5
Vancouver
Costa ACP, Kuwabara WMT, Bonassa ACM, Monaco CFL, Curi R, Carpinelli AR, Nunes MT. The insulin resistance is reversed by exogenous 3,5,3′triiodothyronine in type 2 diabetic Goto–Kakizaki rats by an inflammatory-independent pathway [Internet]. Endocrine. 2020 ;9 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s12020-020-02208-5
Artigo de periodico
Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells (2020)
Librais, Gabriela Nunes Marsiglio ; Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher; Hoffmann, Markus Daniel Alexander; Roma, Leticia Prates ; Carpinelli, Angelo Rafael
Source: Redox Report. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, ILHOTAS DE LANGERHANS, ÁCIDOS GRAXOS, GLICOSE, MEMBRANA PLASMÁTICA, PÂNCREAS, ÁCIDO LINOLEICO
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ABNT
LIBRAIS, Gabriela Nunes Marsiglio et al. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, v. 25, n. 1, p. 41–50, 2020Tradução . . Disponível em: https://doi.org/10.1080/13510002.2020.1757877. Acesso em: 06 nov. 2024.
APA
Librais, G. N. M., Vilas-Boas, E. A., Carlein, C., Hoffmann, M. D. A., Roma, L. P., & Carpinelli, A. R. (2020). Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, 25( 1), 41–50. doi:10.1080/13510002.2020.1757877
NLM
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Vancouver
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Artigo de periodico
Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats (2020)
Silva, Flaviane de Fátima; Morais, Hely de; Silva, Milene Ortiz; Silva, Francemilson Goulart da ; Croffi, Rafael Vianna; Nascimento, Caroline Serrano ; Graciano, Maria Fernanda Rodrigues ; Carpinelli, Angelo Rafael ; Bazotte, Roberto Barbosa ; Souza, Helenir Medri de
Source: Journal of Cellular Biochemistry. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, TUMOR DE WALKER ANIMAL, CAQUEXIA, GLUTAMINA, DOENÇAS METABÓLICAS, CITOCINAS, RESISTÊNCIA À INSULINA
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ABNT
SILVA, Flaviane de Fátima et al. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats. Journal of Cellular Biochemistry, p. 1-11, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcb.29682. Acesso em: 06 nov. 2024.
APA
Silva, F. de F., Morais, H. de, Silva, M. O., Silva, F. G. da, Croffi, R. V., Nascimento, C. S., et al. (2020). Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats. Journal of Cellular Biochemistry, 1-11. doi:10.1002/jcb.29682
NLM
Silva F de F, Morais H de, Silva MO, Silva FG da, Croffi RV, Nascimento CS, Graciano MFR, Carpinelli AR, Bazotte RB, Souza HM de. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats [Internet]. Journal of Cellular Biochemistry. 2020 ; 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/jcb.29682
Vancouver
Silva F de F, Morais H de, Silva MO, Silva FG da, Croffi RV, Nascimento CS, Graciano MFR, Carpinelli AR, Bazotte RB, Souza HM de. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats [Internet]. Journal of Cellular Biochemistry. 2020 ; 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/jcb.29682
Artigo de periodico
Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats (2020)
Bonassa, Ana Cláudia Munhoz ; Vilas-Boas, Eloisa Aparecida ; Costa, Ana Carolina Panveloski ; Leite, Jaqueline Santos Moreira ; Monaco, Camila Ferraz Lucena ; Patrício, Patrícia Riva; Emilio, Henriette; Carpinelli, Angelo Rafael
Source: Nutrients. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, ILHOTAS DE LANGERHANS, JEJUNO DE ANIMAL, RATOS WISTAR, GLICOSE, PLASMA DE ANIMAL
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BONASSA, Ana Cláudia Munhoz et al. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats. Nutrients, v. 12, n. 4, p. 24 , 2020Tradução . . Disponível em: https://doi.org/10.3390/nu12041029. Acesso em: 06 nov. 2024.
APA
Bonassa, A. C. M., Vilas-Boas, E. A., Costa, A. C. P., Leite, J. S. M., Monaco, C. F. L., Patrício, P. R., et al. (2020). Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats. Nutrients, 12( 4), 24 . doi:10.3390/nu12041029
NLM
Bonassa ACM, Vilas-Boas EA, Costa ACP, Leite JSM, Monaco CFL, Patrício PR, Emilio H, Carpinelli AR. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats [Internet]. Nutrients. 2020 ; 12( 4): 24 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/nu12041029
Vancouver
Bonassa ACM, Vilas-Boas EA, Costa ACP, Leite JSM, Monaco CFL, Patrício PR, Emilio H, Carpinelli AR. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats [Internet]. Nutrients. 2020 ; 12( 4): 24 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/nu12041029
Artigo de periodico
Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function (2020)
Vilas-Boas, Eloisa Aparecida ; Karabacz, Noémie; Librais, Gabriela Nunes Marsiglio ; Valle, Maíra Mello Rezende ; Nalbach, Lisa; Ampofo, Emmanuel; Morgan, Bruce ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates
Source: Pharmacological Reports. Unidade: ICB
Subjects: FISIOLOGIA, ÁCIDOS GRAXOS, INSULINA, ESTRESSE OXIDATIVO, PÂNCREAS, APOPTOSE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function. Pharmacological Reports, v. 72, n. 6, p. 1725-1737, 2020Tradução . . Disponível em: https://doi.org/10.1007/s43440-020-00101-6. Acesso em: 06 nov. 2024.
APA
Vilas-Boas, E. A., Karabacz, N., Librais, G. N. M., Valle, M. M. R., Nalbach, L., Ampofo, E., et al. (2020). Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function. Pharmacological Reports, 72( 6), 1725-1737. doi:10.1007/s43440-020-00101-6
NLM
Vilas-Boas EA, Karabacz N, Librais GNM, Valle MMR, Nalbach L, Ampofo E, Morgan B, Carpinelli AR, Roma LP. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function [Internet]. Pharmacological Reports. 2020 ; 72( 6): 1725-1737.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s43440-020-00101-6
Vancouver
Vilas-Boas EA, Karabacz N, Librais GNM, Valle MMR, Nalbach L, Ampofo E, Morgan B, Carpinelli AR, Roma LP. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function [Internet]. Pharmacological Reports. 2020 ; 72( 6): 1725-1737.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s43440-020-00101-6
Artigo de periodico
Effects of lixisenatide treatment on mild cachexia and related metabolic abnormalities in Walker‐256 tumour‐bearing rats (2020)
Quintilhano, Débora L.; Miksza, Daniele R. ; Riazi, Giuliana R.; Frasson-Uemura, Isabele G. ; Graciano, Maria Fernanda R. ; Mazzuco, Tânia L. ; Souza, Helenir M. de ; Gisele L. Bertolini ; Carpinelli, Angelo Rafael
Source: Cell Biochemistry & Function. Unidade: ICB
Subjects: CAQUEXIA, METABOLISMO, RATOS, TUMOR DE WALKER ANIMAL, DIABETES MELLITUS, RESISTÊNCIA À INSULINA, GLUCAGON, INSULINA, CÉLULAS CULTIVADAS DE TUMOR, PERDA DE PESO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUINTILHANO, Débora L. et al. Effects of lixisenatide treatment on mild cachexia and related metabolic abnormalities in Walker‐256 tumour‐bearing rats. Cell Biochemistry & Function, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1002/cbf.3588. Acesso em: 06 nov. 2024.
APA
Quintilhano, D. L., Miksza, D. R., Riazi, G. R., Frasson-Uemura, I. G., Graciano, M. F. R., Mazzuco, T. L., et al. (2020). Effects of lixisenatide treatment on mild cachexia and related metabolic abnormalities in Walker‐256 tumour‐bearing rats. Cell Biochemistry & Function, 9 . doi:10.1002/cbf.3588
NLM
Quintilhano DL, Miksza DR, Riazi GR, Frasson-Uemura IG, Graciano MFR, Mazzuco TL, Souza HM de, Gisele L. Bertolini, Carpinelli AR. Effects of lixisenatide treatment on mild cachexia and related metabolic abnormalities in Walker‐256 tumour‐bearing rats [Internet]. Cell Biochemistry & Function. 2020 ;9 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/cbf.3588
Vancouver
Quintilhano DL, Miksza DR, Riazi GR, Frasson-Uemura IG, Graciano MFR, Mazzuco TL, Souza HM de, Gisele L. Bertolini, Carpinelli AR. Effects of lixisenatide treatment on mild cachexia and related metabolic abnormalities in Walker‐256 tumour‐bearing rats [Internet]. Cell Biochemistry & Function. 2020 ;9 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1002/cbf.3588
Trabalho de evento-resumo periodico
Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS (2019)
Vilas-Boas, Eloisa Aparecida ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Diabetologia. Conference titles: EASD Annual Meeting of the European Association for the Study of Diabetes. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, CITOCINAS, INFLAMAÇÃO, CÉLULAS MORTAS, PÂNCREAS, INSULINA, MITOCÔNDRIAS, CAMUNDONGOS, CITOMETRIA DE FLUXO, WESTERN BLOTTING, ILHOTAS DE LANGERHANS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VILAS-BOAS, Eloisa Aparecida et al. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS. Diabetologia. Heidelberg: Springer. Disponível em: https://doi.org/10.1007/s00125-019-4946-6. Acesso em: 06 nov. 2024. , 2019
APA
Vilas-Boas, E. A., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2019). Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS. Diabetologia. Heidelberg: Springer. doi:10.1007/s00125-019-4946-6
NLM
Vilas-Boas EA, Roma LP, Ortis F, Carpinelli AR. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS [Internet]. Diabetologia. 2019 ; 62 S221-S222.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s00125-019-4946-6
Vancouver
Vilas-Boas EA, Roma LP, Ortis F, Carpinelli AR. Pro-inflammatory cytokines and beta cells death: role of cytosolic ROS [Internet]. Diabetologia. 2019 ; 62 S221-S222.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s00125-019-4946-6
Artigo de periodico
Effects of metformin on insulin resistance and metabolic disorders in tumor-bearing rats with advanced cachexia (2018)
Silva, Flaviane de Fatima ; Silva, Milene Ortiz ; Galia, Winny Beatriz de Souza; Cassolla, Priscila; Silva, Francemilson Goulart da ; Graciano, Maria Fernanda Rodrigues ; Carpinelli, Angelo Rafael ; Souza, Helenir Medri de
Source: Canadian Journal of Physiology and Pharmacology. Unidade: ICB
Subjects: FISIOLOGIA, CAQUEXIA, INSULINA, RESISTÊNCIA À INSULINA, NEOPLASIAS, TUMOR DE WALKER ANIMAL, METABOLISMO ANIMAL, TRANSDUÇÃO DE SINAL CELULAR, CÉLULAS CULTIVADAS DE TUMOR, ANTINEOPLÁSICOS, METFORMINA, DIABETES MELLITUS, RATOS, ILHOTAS DE LANGERHANS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Flaviane de Fatima et al. Effects of metformin on insulin resistance and metabolic disorders in tumor-bearing rats with advanced cachexia. Canadian Journal of Physiology and Pharmacology, v. 96, n. 5, p. 1-8, 2018Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2017-0171. Acesso em: 06 nov. 2024.
APA
Silva, F. de F., Silva, M. O., Galia, W. B. de S., Cassolla, P., Silva, F. G. da, Graciano, M. F. R., et al. (2018). Effects of metformin on insulin resistance and metabolic disorders in tumor-bearing rats with advanced cachexia. Canadian Journal of Physiology and Pharmacology, 96( 5), 1-8. doi:10.1139/cjpp-2017-0171
NLM
Silva F de F, Silva MO, Galia WB de S, Cassolla P, Silva FG da, Graciano MFR, Carpinelli AR, Souza HM de. Effects of metformin on insulin resistance and metabolic disorders in tumor-bearing rats with advanced cachexia [Internet]. Canadian Journal of Physiology and Pharmacology. 2018 ; 96( 5): 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1139/cjpp-2017-0171
Vancouver
Silva F de F, Silva MO, Galia WB de S, Cassolla P, Silva FG da, Graciano MFR, Carpinelli AR, Souza HM de. Effects of metformin on insulin resistance and metabolic disorders in tumor-bearing rats with advanced cachexia [Internet]. Canadian Journal of Physiology and Pharmacology. 2018 ; 96( 5): 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1139/cjpp-2017-0171
Artigo de periodico
Short-term high glucose culture potentiates pancreatic beta cell function (2018)
Oliveira, Eduardo Rebelato Lopes de; Santos, Laila Romagueira Bichara dos; Carpinelli, Angelo Rafael ; Rorsman, Patrik; Abdulkader, Fernando Rodrigues de Moraes
Source: Scientific reports. Unidade: ICB
Subjects: ILHOTAS DE LANGERHANS, GLICOSE, INSULINA, PÂNCREAS, ÍNSULA DO PÂNCREAS ANIMAL, ROEDORES, METABOLISMO ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Eduardo Rebelato Lopes de et al. Short-term high glucose culture potentiates pancreatic beta cell function. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-31325-5. Acesso em: 06 nov. 2024.
APA
Oliveira, E. R. L. de, Santos, L. R. B. dos, Carpinelli, A. R., Rorsman, P., & Abdulkader, F. R. de M. (2018). Short-term high glucose culture potentiates pancreatic beta cell function. Scientific reports, 8. doi:10.1038/s41598-018-31325-5
NLM
Oliveira ERL de, Santos LRB dos, Carpinelli AR, Rorsman P, Abdulkader FR de M. Short-term high glucose culture potentiates pancreatic beta cell function [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41598-018-31325-5
Vancouver
Oliveira ERL de, Santos LRB dos, Carpinelli AR, Rorsman P, Abdulkader FR de M. Short-term high glucose culture potentiates pancreatic beta cell function [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41598-018-31325-5
Artigo de periodico
Chronic treatment with dexamethasone alters clock gene expression and melatonin synthesis in rat pineal gland at night (2018)
Santos, Daniela Meneses; Buonfiglio, Daniella do Carmo ; Garcia, Rodrigo Antonio Peliciari ; Lobo, Angela Maria Ramos ; Souza, Divanízia do Nascimento ; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira ; Sertie, Rogério Antonio Laurato ; Andreotti, Sandra ; Lima, Fabio Bessa ; Afeche, Solange Castro ; Fioretto, Emerson Ticona ; Cipolla Neto, José ; Marçal, Anderson Carlos
Source: Nature and Science of Sleep. Unidade: ICB
Subjects: MELATONINA, METABOLISMO ENERGÉTICO, RITMOS CIRCADIANOS ANIMAL, RATOS, HORMÔNIOS GLICOCORTICOIDES, HIPOTÁLAMO, GLICEMIA, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Daniela Meneses et al. Chronic treatment with dexamethasone alters clock gene expression and melatonin synthesis in rat pineal gland at night. Nature and Science of Sleep, v. 10, p. 203-215, 2018Tradução . . Disponível em: https://doi.org/10.2147/NSS.S158602. Acesso em: 06 nov. 2024.
APA
Santos, D. M., Buonfiglio, D. do C., Garcia, R. A. P., Lobo, A. M. R., Souza, D. do N., Carpinelli, A. R., et al. (2018). Chronic treatment with dexamethasone alters clock gene expression and melatonin synthesis in rat pineal gland at night. Nature and Science of Sleep, 10, 203-215. doi:10.2147/NSS.S158602
NLM
Santos DM, Buonfiglio D do C, Garcia RAP, Lobo AMR, Souza D do N, Carpinelli AR, Carvalho CR de O, Sertie RAL, Andreotti S, Lima FB, Afeche SC, Fioretto ET, Cipolla Neto J, Marçal AC. Chronic treatment with dexamethasone alters clock gene expression and melatonin synthesis in rat pineal gland at night [Internet]. Nature and Science of Sleep. 2018 ; 10 203-215.[citado 2024 nov. 06 ] Available from: https://doi.org/10.2147/NSS.S158602
Vancouver
Santos DM, Buonfiglio D do C, Garcia RAP, Lobo AMR, Souza D do N, Carpinelli AR, Carvalho CR de O, Sertie RAL, Andreotti S, Lima FB, Afeche SC, Fioretto ET, Cipolla Neto J, Marçal AC. Chronic treatment with dexamethasone alters clock gene expression and melatonin synthesis in rat pineal gland at night [Internet]. Nature and Science of Sleep. 2018 ; 10 203-215.[citado 2024 nov. 06 ] Available from: https://doi.org/10.2147/NSS.S158602
Artigo de periodico
NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice (2017)
Souza, Arnaldo H. de; Santos, Laila R. B.; Roma, Leticia P.; Bensellam, Mohammed; Carpinelli, Angelo Rafael ; Jonas, Jean-Christophe
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Arnaldo H. de et al. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, v. 439, p. 354-362, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.09.022. Acesso em: 06 nov. 2024.
APA
Souza, A. H. de, Santos, L. R. B., Roma, L. P., Bensellam, M., Carpinelli, A. R., & Jonas, J. -C. (2017). NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, 439, 354-362. doi:10.1016/j.mce.2016.09.022
NLM
Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
Vancouver
Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
Artigo de periodico
Pioglitazone improves insulin sensitivity and reduces weight loss in Walker-256 tumor-bearing rats (2017)
Silva, Flaviane de Fatima; Ortiz-Silva, Milene; Galia, Winny Beatriz de Souza; Cassolla, Priscila; Graciano, Maria Fernanda Rodrigue; Zaia, Cassia Thaïs Bussamra Vieira; Zaia, Dimas; Carpinelli, Angelo Rafael ; Silva, Francemilson Goulart da ; Souza, Helenir Medri de
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Flaviane de Fatima et al. Pioglitazone improves insulin sensitivity and reduces weight loss in Walker-256 tumor-bearing rats. Life Sciences, v. 171, p. 68-74, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2016.12.016. Acesso em: 06 nov. 2024.
APA
Silva, F. de F., Ortiz-Silva, M., Galia, W. B. de S., Cassolla, P., Graciano, M. F. R., Zaia, C. T. B. V., et al. (2017). Pioglitazone improves insulin sensitivity and reduces weight loss in Walker-256 tumor-bearing rats. Life Sciences, 171, 68-74. doi:10.1016/j.lfs.2016.12.016
NLM
Silva F de F, Ortiz-Silva M, Galia WB de S, Cassolla P, Graciano MFR, Zaia CTBV, Zaia D, Carpinelli AR, Silva FG da, Souza HM de. Pioglitazone improves insulin sensitivity and reduces weight loss in Walker-256 tumor-bearing rats [Internet]. Life Sciences. 2017 ; 171 68-74.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.lfs.2016.12.016
Vancouver
Silva F de F, Ortiz-Silva M, Galia WB de S, Cassolla P, Graciano MFR, Zaia CTBV, Zaia D, Carpinelli AR, Silva FG da, Souza HM de. Pioglitazone improves insulin sensitivity and reduces weight loss in Walker-256 tumor-bearing rats [Internet]. Life Sciences. 2017 ; 171 68-74.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.lfs.2016.12.016

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