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Artigo de periodico
Acetylcholine, fatty acids, and lipid mediators are linked to COVID-19 severity (2022)
Pérez, Malena Martínez; Pimentel, Vinícius Eduardo; Fuzo, Carlos Alessandro; Silva Neto, Pedro Vieira da; Toro, Diana Mota; Silva, Thais Fernanda de Campos Fraga da; Gardinassi, Luiz Gustavo Araujo ; Oliveira, Camilla Narjara Simão; Souza, Camila O. S.; Torre Neto, Nicola T.; Carvalho, Jonatan Constança Silva de; Leo, Thais Canassa de ; Takahashi, Viviani Nardini ; Feitosa, Marley Ribeiro; Parra, Rogério Serafim; Rocha, José Joaquim Ribeiro da ; Féres, Omar; Vilar, Fernando Crivelenti; Gaspar, Gilberto Gambero; Constant, Leticia F.; Ostini, Fátima M.; Degiovani, Augusto Marcussi; Amorim, Alessandro P.; Viana, Angelina Lettiere ; Fernandes, Ana Paula Morais ; Maruyama, Sandra Regina; Russo, Elisa Maria de Sousa ; Santos, Isabel Kinney Ferreira de Miranda; Bonato, Vânia Luiza Deperon; Cardoso, Cristina Ribeiro de Barros; Sorgi, Carlos Arterio ; Dias-Baruffi, Marcelo; Faccioli, Lucia Helena ; Carmona-Garcia, Ingryd; Milanezi, Cristiane Maria; Zaparoli, Kamila; Garbato, Caroline Tempesta; Argolo, Jamille Guedes Malta
Fonte: The Journal of Immunology. Unidades: FCFRP, FMRP, EERP, FFCLRP, EE/EERP
Assuntos: ACETILCOLINA, ÁCIDOS ARAQUIDÔNICOS, COVID-19, ÁCIDOS GRAXOS
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ABNT
PÉREZ, Malena Martínez et al. Acetylcholine, fatty acids, and lipid mediators are linked to COVID-19 severity. The Journal of Immunology, v. 209, n. 2, p. 250-261, 2022Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2200079. Acesso em: 04 nov. 2024.
APA
Pérez, M. M., Pimentel, V. E., Fuzo, C. A., Silva Neto, P. V. da, Toro, D. M., Silva, T. F. de C. F. da, et al. (2022). Acetylcholine, fatty acids, and lipid mediators are linked to COVID-19 severity. The Journal of Immunology, 209( 2), 250-261. doi:10.4049/jimmunol.2200079
NLM
Pérez MM, Pimentel VE, Fuzo CA, Silva Neto PV da, Toro DM, Silva TF de CF da, Gardinassi LGA, Oliveira CNS, Souza COS, Torre Neto NT, Carvalho JCS de, Leo TC de, Takahashi VN, Feitosa MR, Parra RS, Rocha JJR da, Féres O, Vilar FC, Gaspar GG, Constant LF, Ostini FM, Degiovani AM, Amorim AP, Viana AL, Fernandes APM, Maruyama SR, Russo EM de S, Santos IKF de M, Bonato VLD, Cardoso CR de B, Sorgi CA, Dias-Baruffi M, Faccioli LH, Carmona-Garcia I, Milanezi CM, Zaparoli K, Garbato CT, Argolo JGM. Acetylcholine, fatty acids, and lipid mediators are linked to COVID-19 severity [Internet]. The Journal of Immunology. 2022 ; 209( 2): 250-261.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.2200079
Vancouver
Pérez MM, Pimentel VE, Fuzo CA, Silva Neto PV da, Toro DM, Silva TF de CF da, Gardinassi LGA, Oliveira CNS, Souza COS, Torre Neto NT, Carvalho JCS de, Leo TC de, Takahashi VN, Feitosa MR, Parra RS, Rocha JJR da, Féres O, Vilar FC, Gaspar GG, Constant LF, Ostini FM, Degiovani AM, Amorim AP, Viana AL, Fernandes APM, Maruyama SR, Russo EM de S, Santos IKF de M, Bonato VLD, Cardoso CR de B, Sorgi CA, Dias-Baruffi M, Faccioli LH, Carmona-Garcia I, Milanezi CM, Zaparoli K, Garbato CT, Argolo JGM. Acetylcholine, fatty acids, and lipid mediators are linked to COVID-19 severity [Internet]. The Journal of Immunology. 2022 ; 209( 2): 250-261.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.2200079
Artigo de periodico
Local sustained delivery of anti-IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis (2021)
Pacheco, Cinthia Mara da Fonseca ; Maltos, Katia L. M. ; Shehabeldin, Mostafa S. ; Thomas, Laura L. ; Zhuang, Zhe ; Yoshizawa, Sayuri; Verdelis, Konstantinos; Gaffen, Sarah L. ; Garlet, Gustavo Pompermaier ; Little, Steven R.; Sfeir, Charles
Fonte: The Journal of Immunology. Unidade: FOB
Assuntos: PERIODONTITE, DENSIDADE ÓSSEA, INFLAMAÇÃO, REABSORÇÃO ÓSSEA ALVEOLAR
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ABNT
PACHECO, Cinthia Mara da Fonseca et al. Local sustained delivery of anti-IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis. The Journal of Immunology, v. 206, n. 10, p. 2386-2392, 2021Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2001432. Acesso em: 04 nov. 2024.
APA
Pacheco, C. M. da F., Maltos, K. L. M., Shehabeldin, M. S., Thomas, L. L., Zhuang, Z., Yoshizawa, S., et al. (2021). Local sustained delivery of anti-IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis. The Journal of Immunology, 206( 10), 2386-2392. doi:10.4049/jimmunol.2001432
NLM
Pacheco CM da F, Maltos KLM, Shehabeldin MS, Thomas LL, Zhuang Z, Yoshizawa S, Verdelis K, Gaffen SL, Garlet GP, Little SR, Sfeir C. Local sustained delivery of anti-IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis [Internet]. The Journal of Immunology. 2021 ; 206( 10): 2386-2392.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.2001432
Vancouver
Pacheco CM da F, Maltos KLM, Shehabeldin MS, Thomas LL, Zhuang Z, Yoshizawa S, Verdelis K, Gaffen SL, Garlet GP, Little SR, Sfeir C. Local sustained delivery of anti-IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis [Internet]. The Journal of Immunology. 2021 ; 206( 10): 2386-2392.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.2001432
Trabalho de evento-resumo periodico
CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis (2021)
Souza, Camila O. S.; Oliveira, Jefferson Elias ; Fontanari, Caroline ; Rodriguez, Vanderlei; Gardinassi, Luiz; Faccioli, Lucia Helena
Fonte: The Journal of Immunology. Nome do evento: Immunology 2021™ Meeting Abstracts. Unidades: FCFRP, FMRP
Assuntos: SCHISTOSOMA MANSONI, DOENÇAS PARASITÁRIAS, MACRÓFAGOS, ESQUISTOSSOMOSE
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ABNT
SOUZA, Camila O. S. et al. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis. The Journal of Immunology. Rockville: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.4049/jimmunol.206.Supp.99.29. Acesso em: 04 nov. 2024. , 2021
APA
Souza, C. O. S., Oliveira, J. E., Fontanari, C., Rodriguez, V., Gardinassi, L., & Faccioli, L. H. (2021). CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis. The Journal of Immunology. Rockville: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. doi:10.4049/jimmunol.206.Supp.99.29
NLM
Souza COS, Oliveira JE, Fontanari C, Rodriguez V, Gardinassi L, Faccioli LH. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis [Internet]. The Journal of Immunology. 2021 ; 206[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.206.Supp.99.29
Vancouver
Souza COS, Oliveira JE, Fontanari C, Rodriguez V, Gardinassi L, Faccioli LH. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis [Internet]. The Journal of Immunology. 2021 ; 206[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.206.Supp.99.29
Artigo de periodico
Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development (2020)
Sesti-Costa, Renata; Cervantes-Barragan, Luisa; Swiecki, Melissa K.; Fachi, José Luís; Cella, Marina; Gilfillan, Susan; Silva, João Santana; Colonna, Marco
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: ANIMAIS, INTERFERONS, RATOS, IMUNOLOGIA
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ABNT
SESTI-COSTA, Renata et al. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development. The Journal of Immunology, v. 204, n. 8, p. 2257-2268, 2020Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1900604. Acesso em: 04 nov. 2024.
APA
Sesti-Costa, R., Cervantes-Barragan, L., Swiecki, M. K., Fachi, J. L., Cella, M., Gilfillan, S., et al. (2020). Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development. The Journal of Immunology, 204( 8), 2257-2268. doi:10.4049/jimmunol.1900604
NLM
Sesti-Costa R, Cervantes-Barragan L, Swiecki MK, Fachi JL, Cella M, Gilfillan S, Silva JS, Colonna M. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development [Internet]. The Journal of Immunology. 2020 ; 204( 8): 2257-2268.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1900604
Vancouver
Sesti-Costa R, Cervantes-Barragan L, Swiecki MK, Fachi JL, Cella M, Gilfillan S, Silva JS, Colonna M. Leukemia inhibitory factor inhibits plasmacytoid dendritic cell function and development [Internet]. The Journal of Immunology. 2020 ; 204( 8): 2257-2268.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1900604
Artigo de periodico
Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus (2017)
Silveira, Eduardo Lani Volpe da ; Rogers, Kenneth A; Gumber, Sanjeev; Amancha, Praveen; Xiao, Peng; Woollard, Shawna M; Byrareddy, Siddappa N; Teixeira, Mauro Martins; Villinger, Francois
Fonte: The Journal of Immunology. Unidade: FCF
Assuntos: ZIKA VÍRUS, MACACOS
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ABNT
SILVEIRA, Eduardo Lani Volpe da et al. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus. The Journal of Immunology, v. 199, n. 3, p. 1003-1011, 2017Tradução . . Disponível em: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256. Acesso em: 04 nov. 2024.
APA
Silveira, E. L. V. da, Rogers, K. A., Gumber, S., Amancha, P., Xiao, P., Woollard, S. M., et al. (2017). Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus. The Journal of Immunology, 199( 3), 1003-1011. doi:10.4049/jimmunol.1700256
NLM
Silveira ELV da, Rogers KA, Gumber S, Amancha P, Xiao P, Woollard SM, Byrareddy SN, Teixeira MM, Villinger F. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus [Internet]. The Journal of Immunology. 2017 ; 199( 3): 1003-1011.[citado 2024 nov. 04 ] Available from: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256
Vancouver
Silveira ELV da, Rogers KA, Gumber S, Amancha P, Xiao P, Woollard SM, Byrareddy SN, Teixeira MM, Villinger F. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus [Internet]. The Journal of Immunology. 2017 ; 199( 3): 1003-1011.[citado 2024 nov. 04 ] Available from: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256
Artigo de periodico
Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication (2017)
Lima Júnior, Djalma de Souza; Mineo, Tiago Wilson Patriarca; Calich, Vera Lúcia Garcia; Zamboni, Dario Simões
Fonte: The Journal of Immunology. Unidades: ICB, FMRP
Assuntos: LEISHMANIA, FAGOCITOSE, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
LIMA JÚNIOR, Djalma de Souza et al. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, v. 199, n. 6, p. 2055-2068, 2017Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1700258. Acesso em: 04 nov. 2024.
APA
Lima Júnior, D. de S., Mineo, T. W. P., Calich, V. L. G., & Zamboni, D. S. (2017). Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, 199( 6), 2055-2068. doi:10.4049/jimmunol.1700258
NLM
Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1700258
Vancouver
Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1700258
Artigo de periodico
Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo (2015)
Cerqueira, Daiane M.; Pereira, Marcelo S. F.; Silva, Alexandre L. N.; Cunha, Larissa D.; Zamboni, Dario Simões
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: BACTÉRIAS GRAM-NEGATIVAS, PNEUMONIA, MACRÓFAGOS, PROTEÍNAS DE LIGAÇÃO DO CÁLCIO, APOPTOSE
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ABNT
CERQUEIRA, Daiane M. et al. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, v. 195, n. 5, p. 2303-2311, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1501223. Acesso em: 04 nov. 2024.
APA
Cerqueira, D. M., Pereira, M. S. F., Silva, A. L. N., Cunha, L. D., & Zamboni, D. S. (2015). Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, 195( 5), 2303-2311. doi:10.4049/jimmunol.1501223
NLM
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1501223
Vancouver
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1501223
Artigo de periodico
IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection (2015)
Ketelut-Carneiro, Natália; Silva, Grace Kelly; Rocha, Fernanda Agostini; Milanezi, Cristiane Maria; Cavalcanti-Neto, Florêncio Figueiredo; Zamboni, Dario Simões; Silva, João Santana da
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: SISTEMA IMUNE, FUNGOS, INFLAMAÇÃO, MICOSES, PARACOCCIDIOIDES BRASILIENSIS
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ABNT
KETELUT-CARNEIRO, Natália et al. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, v. 194, n. 9, p. 4507-4517, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1402321. Acesso em: 04 nov. 2024.
APA
Ketelut-Carneiro, N., Silva, G. K., Rocha, F. A., Milanezi, C. M., Cavalcanti-Neto, F. F., Zamboni, D. S., & Silva, J. S. da. (2015). IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, 194( 9), 4507-4517. doi:10.4049/jimmunol.1402321
NLM
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1402321
Vancouver
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2024 nov. 04 ] Available from: https://doi.org/10.4049/jimmunol.1402321
Artigo de periodico
CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression (2008)
Moreira, Ana Paula; Cavassani, Karen Angelica; Tristão, Fabrine Sales Massafera; Campanelli, Ana Paula; Martinez, Roberto; Rossi, Marcos Antonio; Silva, João S.
Fonte: The Journal of Immunology. Unidades: FOB, FMRP
Assuntos: IMUNOSSUPRESSÃO, FUNGOS, PARACOCCIDIOIDES BRASILIENSIS, LEUCÓCITOS
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ABNT
MOREIRA, Ana Paula et al. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression. The Journal of Immunology, v. 180, n. 5, p. 3049-3056, 2008Tradução . . Disponível em: http://www.jimmunol.org/cgi/content/full/180/5/3049. Acesso em: 04 nov. 2024.
APA
Moreira, A. P., Cavassani, K. A., Tristão, F. S. M., Campanelli, A. P., Martinez, R., Rossi, M. A., & Silva, J. S. (2008). CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression. The Journal of Immunology, 180( 5), 3049-3056. Recuperado de http://www.jimmunol.org/cgi/content/full/180/5/3049
NLM
Moreira AP, Cavassani KA, Tristão FSM, Campanelli AP, Martinez R, Rossi MA, Silva JS. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression [Internet]. The Journal of Immunology. 2008 ; 180( 5): 3049-3056.[citado 2024 nov. 04 ] Available from: http://www.jimmunol.org/cgi/content/full/180/5/3049
Vancouver
Moreira AP, Cavassani KA, Tristão FSM, Campanelli AP, Martinez R, Rossi MA, Silva JS. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression [Internet]. The Journal of Immunology. 2008 ; 180( 5): 3049-3056.[citado 2024 nov. 04 ] Available from: http://www.jimmunol.org/cgi/content/full/180/5/3049
Artigo de periodico
Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease (2006)
Faé, Kellen C.; Silva, Danielle D. da; Oshiro, Sandra E.; Tanaka, Ana C.; Pomerantzeff, Pablo; Douay, Cornine; Charron, Dominique; Toubert, Antoine; Cunningham, Madeleine; Kalil Filho, J.; Guilherme, Ana L.
Fonte: The Journal of Immunology. Unidade: FM
Assuntos: MIOSINA, PEPTÍDEOS
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ABNT
FAÉ, Kellen C. et al. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology, v. 176, p. 5662-5670, 2006Tradução . . Acesso em: 04 nov. 2024.
APA
Faé, K. C., Silva, D. D. da, Oshiro, S. E., Tanaka, A. C., Pomerantzeff, P., Douay, C., et al. (2006). Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology, 176, 5662-5670.
NLM
Faé KC, Silva DD da, Oshiro SE, Tanaka AC, Pomerantzeff P, Douay C, Charron D, Toubert A, Cunningham M, Kalil Filho J, Guilherme AL. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology. 2006 ; 176 5662-5670.[citado 2024 nov. 04 ]
Vancouver
Faé KC, Silva DD da, Oshiro SE, Tanaka AC, Pomerantzeff P, Douay C, Charron D, Toubert A, Cunningham M, Kalil Filho J, Guilherme AL. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology. 2006 ; 176 5662-5670.[citado 2024 nov. 04 ]
Artigo de periodico
Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis (2006)
Moreno, Susana E.; Alves-Filho, José C.; Rios-Santos, Fabrício; Silva, João Santana da; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Teixeira, Mauro M.
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: IMUNOLOGIA, SISTEMA IMUNE, SEPSE
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ABNT
MORENO, Susana E. et al. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, v. 177, p. 1264-1271, 2006Tradução . . Acesso em: 04 nov. 2024.
APA
Moreno, S. E., Alves-Filho, J. C., Rios-Santos, F., Silva, J. S. da, Ferreira, S. H., Cunha, F. de Q., & Teixeira, M. M. (2006). Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, 177, 1264-1271.
NLM
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2024 nov. 04 ]
Vancouver
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2024 nov. 04 ]
Artigo de periodico
Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis (2005)
Machado, Eleuza Rodrigues; Ueta, Marlene T.; Lourenço, Elaine Vicente; Anibal, Fernanda F.; Sorgi, Carlos Artério; Soares, Edson Garcia; Roque-Barreira, Maria Cristina; Medeiros, Alexandra Ivo; Faccioli, Lúcia Helena
Fonte: The Journal of Immunology. Unidades: FCFRP, FMRP
Assunto: IMUNOLOGIA
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ABNT
MACHADO, Eleuza Rodrigues et al. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology, v. 175, p. 3892-3899, 2005Tradução . . Acesso em: 04 nov. 2024.
APA
Machado, E. R., Ueta, M. T., Lourenço, E. V., Anibal, F. F., Sorgi, C. A., Soares, E. G., et al. (2005). Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology, 175, 3892-3899.
NLM
Machado ER, Ueta MT, Lourenço EV, Anibal FF, Sorgi CA, Soares EG, Roque-Barreira MC, Medeiros AI, Faccioli LH. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology. 2005 ; 175 3892-3899.[citado 2024 nov. 04 ]
Vancouver
Machado ER, Ueta MT, Lourenço EV, Anibal FF, Sorgi CA, Soares EG, Roque-Barreira MC, Medeiros AI, Faccioli LH. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology. 2005 ; 175 3892-3899.[citado 2024 nov. 04 ]
Artigo de periodico
Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi (2004)
Michailowsky, Vladimir; Celes, Mara Rúbia Nunes; Marino, Ana P.; Silva, Andréa A.; Vieira, Leda Q.; Rossi, Marcos Antonio; Gazzinelli, Ricardo T.; Lannes-Vieira, Joseli; Silva, João Santana da
Fonte: The Journal of Immunology. Unidade: FMRP
Assunto: TRYPANOSOMA CRUZI (IMUNOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MICHAILOWSKY, Vladimir et al. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, v. 173, p. 463-470, 2004Tradução . . Acesso em: 04 nov. 2024.
APA
Michailowsky, V., Celes, M. R. N., Marino, A. P., Silva, A. A., Vieira, L. Q., Rossi, M. A., et al. (2004). Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, 173, 463-470.
NLM
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470.[citado 2024 nov. 04 ]
Vancouver
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470.[citado 2024 nov. 04 ]
Artigo de periodico
Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity (2003)
Rodríguez, Dunia; Keller, Alexandre de Castro; Faquim-Mauro, Eliana Lima; Macedo, Mahasti Sahihi de; Cunha, Fernando de Queiroz; Lefort, Jean; Vargaftig, B. Boris; Russo, Momtchilo
Fonte: The Journal of Immunology. Unidades: FMRP, ICB
Assunto: ASMA (IMUNOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRÍGUEZ, Dunia et al. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology, v. 171, n. 2, p. 1001-1008, 2003Tradução . . Acesso em: 04 nov. 2024.
APA
Rodríguez, D., Keller, A. de C., Faquim-Mauro, E. L., Macedo, M. S. de, Cunha, F. de Q., Lefort, J., et al. (2003). Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology, 171( 2), 1001-1008.
NLM
Rodríguez D, Keller A de C, Faquim-Mauro EL, Macedo MS de, Cunha F de Q, Lefort J, Vargaftig BB, Russo M. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology. 2003 ; 171( 2): 1001-1008.[citado 2024 nov. 04 ]
Vancouver
Rodríguez D, Keller A de C, Faquim-Mauro EL, Macedo MS de, Cunha F de Q, Lefort J, Vargaftig BB, Russo M. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology. 2003 ; 171( 2): 1001-1008.[citado 2024 nov. 04 ]
Artigo de periodico
IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1' (2003)
Cannetti, Claudio A.; Leung, Bernard P.; Culshaw, Shauna; McInnes, Iain B.; Cunha, Fernando de Queiroz; Liew, Foo Y.
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: IMUNOLOGIA, FARMACOLOGIA EXPERIMENTAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CANNETTI, Claudio A. et al. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology, v. 171, n. 2, p. 1009-1015, 2003Tradução . . Acesso em: 04 nov. 2024.
APA
Cannetti, C. A., Leung, B. P., Culshaw, S., McInnes, I. B., Cunha, F. de Q., & Liew, F. Y. (2003). IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology, 171( 2), 1009-1015.
NLM
Cannetti CA, Leung BP, Culshaw S, McInnes IB, Cunha F de Q, Liew FY. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology. 2003 ; 171( 2): 1009-1015.[citado 2024 nov. 04 ]
Vancouver
Cannetti CA, Leung BP, Culshaw S, McInnes IB, Cunha F de Q, Liew FY. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology. 2003 ; 171( 2): 1009-1015.[citado 2024 nov. 04 ]
Artigo de periodico
A Role for IL-18 un neutrophil activation (2001)
Leung, Bernard P.; Culshaw, Shauna; Gracie, J. Alastair; Hunter, David; Canetti, Cláudio de Azevedo; Campbell, Carol; Cunha, Fernando de Queiroz; Liew, Foo Y.; McInnes, Iain B.
Fonte: The Journal of Immunology. Unidade: FMRP
Assuntos: FARMACOLOGIA, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEUNG, Bernard P. et al. A Role for IL-18 un neutrophil activation. The Journal of Immunology, v. 167, p. 2879-2886, 2001Tradução . . Acesso em: 04 nov. 2024.
APA
Leung, B. P., Culshaw, S., Gracie, J. A., Hunter, D., Canetti, C. de A., Campbell, C., et al. (2001). A Role for IL-18 un neutrophil activation. The Journal of Immunology, 167, 2879-2886.
NLM
Leung BP, Culshaw S, Gracie JA, Hunter D, Canetti C de A, Campbell C, Cunha F de Q, Liew FY, McInnes IB. A Role for IL-18 un neutrophil activation. The Journal of Immunology. 2001 ; 167 2879-2886.[citado 2024 nov. 04 ]
Vancouver
Leung BP, Culshaw S, Gracie JA, Hunter D, Canetti C de A, Campbell C, Cunha F de Q, Liew FY, McInnes IB. A Role for IL-18 un neutrophil activation. The Journal of Immunology. 2001 ; 167 2879-2886.[citado 2024 nov. 04 ]
Artigo de periodico
Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction (2001)
Eaves-Pyles, Tonyia; Murthy, Kanneganti; Liaudet, Lucas; Virág, László; Ross, Gary; Szabó, Csaba; Salzman, Andrew; Soriano, Francisco Garcia
Fonte: The Journal of Immunology. Unidade: FM
Assuntos: ÓXIDO NÍTRICO, INFLAMAÇÃO, SALMONELLA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
EAVES-PYLES, Tonyia et al. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology, p. 1248-1259, 2001Tradução . . Acesso em: 04 nov. 2024.
APA
Eaves-Pyles, T., Murthy, K., Liaudet, L., Virág, L., Ross, G., Szabó, C., et al. (2001). Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology, 1248-1259.
NLM
Eaves-Pyles T, Murthy K, Liaudet L, Virág L, Ross G, Szabó C, Salzman A, Soriano FG. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology. 2001 ; 1248-1259.[citado 2024 nov. 04 ]
Vancouver
Eaves-Pyles T, Murthy K, Liaudet L, Virág L, Ross G, Szabó C, Salzman A, Soriano FG. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology. 2001 ; 1248-1259.[citado 2024 nov. 04 ]

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