IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection (2015)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; MILANEZI, CRISTIANE MARIA - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.4049/jimmunol.1402321
- Subjects: SISTEMA IMUNE; FUNGOS; INFLAMAÇÃO; MICOSES; PARACOCCIDIOIDES BRASILIENSIS
- Language: Inglês
- Imprenta:
- Source:
- Título: The Journal of Immunology
- ISSN: 1550-6606
- Volume/Número/Paginação/Ano: v. 194, n. 9, p. 4507-4517, 2015
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
KETELUT-CARNEIRO, Natália et al. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, v. 194, n. 9, p. 4507-4517, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1402321. Acesso em: 30 dez. 2025. -
APA
Ketelut-Carneiro, N., Silva, G. K., Rocha, F. A., Milanezi, C. M., Cavalcanti-Neto, F. F., Zamboni, D. S., & Silva, J. S. da. (2015). IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, 194( 9), 4507-4517. doi:10.4049/jimmunol.1402321 -
NLM
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2025 dez. 30 ] Available from: https://doi.org/10.4049/jimmunol.1402321 -
Vancouver
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2025 dez. 30 ] Available from: https://doi.org/10.4049/jimmunol.1402321 - Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs
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Informações sobre o DOI: 10.4049/jimmunol.1402321 (Fonte: oaDOI API)
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