IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis (2010)
- Authors:
- USP affiliated authors: MILANEZI, CRISTIANE MARIA - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: TRYPANOSOMA CRUZI; CITOCINAS (PRODUÇÃO); MIOCARDITE
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2010
- Source:
- Título do periódico: PLoS Neglected Tropical Diseases
- ISSN: 1935-2727
- Volume/Número/Paginação/Ano: v. 4, n. 2, p. e604-1-e604-11, 2010
-
ABNT
GUEDES, Paulo Marcos da Matta et al. IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis. PLoS Neglected Tropical Diseases, v. 4, n. 2, p. e604-1-e604-11, 2010Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/5a5baee3-81f0-42fe-ad3b-9fccd0075104/002128995.pdf. Acesso em: 19 abr. 2024. -
APA
Guedes, P. M. da M., Gutierrez, F. R. S., Maia, F. L., Milanezi, C. M., Silva, G. K., Pavanelli, W. R., & Silva, J. S. da. (2010). IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis. PLoS Neglected Tropical Diseases, 4( 2), e604-1-e604-11. Recuperado de https://repositorio.usp.br/directbitstream/5a5baee3-81f0-42fe-ad3b-9fccd0075104/002128995.pdf -
NLM
Guedes PM da M, Gutierrez FRS, Maia FL, Milanezi CM, Silva GK, Pavanelli WR, Silva JS da. IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis [Internet]. PLoS Neglected Tropical Diseases. 2010 ; 4( 2): e604-1-e604-11.[citado 2024 abr. 19 ] Available from: https://repositorio.usp.br/directbitstream/5a5baee3-81f0-42fe-ad3b-9fccd0075104/002128995.pdf -
Vancouver
Guedes PM da M, Gutierrez FRS, Maia FL, Milanezi CM, Silva GK, Pavanelli WR, Silva JS da. IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis [Internet]. PLoS Neglected Tropical Diseases. 2010 ; 4( 2): e604-1-e604-11.[citado 2024 abr. 19 ] Available from: https://repositorio.usp.br/directbitstream/5a5baee3-81f0-42fe-ad3b-9fccd0075104/002128995.pdf - Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs
- CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system
- Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis
- Toxoplasma gondii: the severity of toxoplasmic encephalitis in C57BL/6 mice is associated with increased ALCAM and VCAM-1 expression in the central nervous system and higher blood-brain barrier permeability
- Heme oxygenase-1 activity is involved in the control of Toxoplasma gondii infection in the lung of BALB/c and C57BL/6 and in the small intestine of C57BL/6 mice
- Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis
- IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection
- NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses
- Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth
- BALB/c mice infected with antimony treatment refractory isolate of Leishmania braziliensis present severe lesions due to IL-4 production
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