Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis (2006)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; FERREIRA, SERGIO HENRIQUE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- Subjects: IMUNOLOGIA; SISTEMA IMUNE; SEPSE
- Language: Inglês
- Imprenta:
- Source:
- Título: The Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 177, p. 1264-1271, 2006
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ABNT
MORENO, Susana E. et al. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, v. 177, p. 1264-1271, 2006Tradução . . Acesso em: 02 dez. 2025. -
APA
Moreno, S. E., Alves-Filho, J. C., Rios-Santos, F., Silva, J. S. da, Ferreira, S. H., Cunha, F. de Q., & Teixeira, M. M. (2006). Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, 177, 1264-1271. -
NLM
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2025 dez. 02 ] -
Vancouver
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2025 dez. 02 ] - MIP-1 alfa[CCL3] acting on the CCR1 receptor mediates neutrophil migration in immune inflammation via sequential release of TNF-alfa and 'LTB IND 4'
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- A cascade of cytokines mediates mechanical inflammatory hipernociception in mice
- Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis
- Neutrophil migration in inflammation: nitric oxide inhibits rolling adhesion and induces apoptosis
- Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase
- Involvement of NO in the failure of neutrophil migration in sepsis induced by Staphylococcus aureus
- Endothelin-1 induces neutrophil recruitment in adaptive inflammation via TNF'alfa' and CXCL1/CXCR2 in mice
- IL-12, but not IL-18, is critical to neutrophil activation and resistance to polymicrobial sepsis induced by cecal ligation and puncture
- Molecular mechanisms involved in peripheral persistent (chronic) hypernociception
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