Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis (2009)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; FERREIRA, SERGIO HENRIQUE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1073/pnas.0900196106
- Subjects: NEUTRÓFILOS; SEPSE
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2009
- Source:
- Título do periódico: Proceedings of the National Academy of Sciences
- ISSN: 0027-8424
- Volume/Número/Paginação/Ano: v. 106, n. 10, p. 4018-4023, 2009
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
ALVES-FILHO, José Carlos et al. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, v. 106, n. 10, p. 4018-4023, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0900196106. Acesso em: 19 abr. 2024. -
APA
Alves-Filho, J. C., Freitas, A., Souto, F. O., Spiller, F., Paula Neto, H., Silva, J. S. da, et al. (2009). Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis. Proceedings of the National Academy of Sciences, 106( 10), 4018-4023. doi:10.1073/pnas.0900196106 -
NLM
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1073/pnas.0900196106 -
Vancouver
Alves-Filho JC, Freitas A, Souto FO, Spiller F, Paula Neto H, Silva JS da, Gazzinelli RT, Teixeira MM, Ferreira SH, Cunha F de Q. Regulation of chemokine receptor by toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis [Internet]. Proceedings of the National Academy of Sciences. 2009 ; 106( 10): 4018-4023.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1073/pnas.0900196106 - MIP-1 alfa[CCL3] acting on the CCR1 receptor mediates neutrophil migration in immune inflammation via sequential release of TNF-alfa and 'LTB IND 4'
- Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis
- Neutrophil migration in inflammation: nitric oxide inhibits rolling adhesion and induces apoptosis
- Tumour necrosis factor-alpha and leukotriene 'B ind. 4' mediate the neutrophil migration in immune inflammation
- A cascade of cytokines mediates mechanical inflammatory hipernociception in mice
- IL-12, but not IL-18, is critical to neutrophil activation and resistance to polymicrobial sepsis induced by cecal ligation and puncture
- Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase
- Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration
- Involvement of NO in the failure of neutrophil migration in sepsis induced by Staphylococcus aureus
- Endothelin-1 induces neutrophil recruitment in adaptive inflammation via TNF'alfa' and CXCL1/CXCR2 in mice
Informações sobre o DOI: 10.1073/pnas.0900196106 (Fonte: oaDOI API)
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