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Artigo de periodico
Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase (2017)
Dal Secco, Daniela; DalBó, Silvia; Lautherbach, Natalia Ennes da Silva; Gava, Fábio N.; Celes, Mara Rúbia Nunes; Benedet, Patricia de OLiveira; Souza, Adriana Helena de; Akinaga, Juliana; Lima, Vanessa; Silva, Katiussia P.; Kiguti, Luiz Ricardo A.; Rossi, Marcos Antonio; Kettelhut, Isis do Carmo; Pupo, André Sampaio; Cunha, Fernando de Queiroz; Assreuy, Jamil
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidade: FMRP
Subjects: ATIVAÇÃO ENZIMÁTICA, INSUFICIÊNCIA CARDÍACA, ÓXIDO NÍTRICO, SEPSE, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
DAL SECCO, Daniela et al. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, v. 313, n. 1, p. H149-H163, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00052.2016. Acesso em: 09 nov. 2024.
APA
Dal Secco, D., DalBó, S., Lautherbach, N. E. da S., Gava, F. N., Celes, M. R. N., Benedet, P. de O. L., et al. (2017). Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, 313( 1), H149-H163. doi:10.1152/ajpheart.00052.2016
NLM
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Vancouver
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Artigo de periodico
The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice (2014)
Canavaci, Adriana Monte Cassiano; Sorgi, Carlos Artério; Martins, Vicente de Paulo; Morais, Fabiana Rosseto; Sousa, Érika V. G. de; Trindade, Bruno Caetano; Cunha, Fernando de Queiróz; Rossi, Marcos Antonio; Aronoff, David M.; Faccioli, Lúcia Helena; Nomizo, Auro
Source: Mediators of Inflammation. Unidades: FCFRP, FMRP
Subjects: TRYPANOSOMA CRUZI, INTERLEUCINAS, ANTIPARASITÁRIOS (FARMACOLOGIA)
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ABNT
CANAVACI, Adriana Monte Cassiano et al. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/893634. Acesso em: 09 nov. 2024.
APA
Canavaci, A. M. C., Sorgi, C. A., Martins, V. de P., Morais, F. R., Sousa, É. V. G. de, Trindade, B. C., et al. (2014). The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, 2014. doi:10.1155/2014/893634
NLM
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2014/893634
Vancouver
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 nov. 09 ] Available from: https://doi.org/10.1155/2014/893634
Artigo de periodico
CCR5 controls immune and metabolic functions during Toxoplasma gondii infection (2014)
Bonfá, Giuliano; Souza, Maria do Carmo; Fonseca, Denise Morais da ; Mineo, Tiago Wilson Patriarca; Rossi, Marcos Antonio; Silva, Neide Maria; Silva, João Santana; Cardoso, Cristina Ribeiro de Barros
Source: PLOs One. Unidades: FMRP, FCFRP
Subjects: CITOCINAS (METABOLISMO), CITOMETRIA DE FLUXO, ÍLEO (IMUNOLOGIA), NEUTRÓFILOS, TOXOPLASMOSE
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ABNT
BONFÁ, Giuliano et al. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection. PLOs One, v. 9, n. 8, p. e104736-1 - e104736-13, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0104736. Acesso em: 09 nov. 2024.
APA
Bonfá, G., Souza, M. do C., Fonseca, D. M. da, Mineo, T. W. P., Rossi, M. A., Silva, N. M., et al. (2014). CCR5 controls immune and metabolic functions during Toxoplasma gondii infection. PLOs One, 9( 8), e104736-1 - e104736-13. doi:10.1371/journal.pone.0104736
NLM
Bonfá G, Souza M do C, Fonseca DM da, Mineo TWP, Rossi MA, Silva NM, Silva JS, Cardoso CR de B. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection [Internet]. PLOs One. 2014 ; 9( 8): e104736-1 - e104736-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0104736
Vancouver
Bonfá G, Souza M do C, Fonseca DM da, Mineo TWP, Rossi MA, Silva NM, Silva JS, Cardoso CR de B. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection [Internet]. PLOs One. 2014 ; 9( 8): e104736-1 - e104736-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0104736
Artigo de periodico
Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats (2013)
Cáu, Stêfany Bruno de Assis; Barato, Renan C.; Celes, Mara Rúbia Nunes; Muniz, Jaqueline J.; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo
Source: Cardiovascular Drugs and Therapy. Unidade: FMRP
Subjects: EMBOLIA PULMONAR, DOENÇAS CARDIOVASCULARES, METALOPROTEINASES, MODELOS ANIMAIS
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ABNT
CÁU, Stêfany Bruno de Assis et al. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, v. 27, n. 4, p. 259-267, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10557-013-6458-9. Acesso em: 09 nov. 2024.
APA
Cáu, S. B. de A., Barato, R. C., Celes, M. R. N., Muniz, J. J., Rossi, M. A., & Tanus-Santos, J. E. (2013). Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, 27( 4), 259-267. doi:10.1007/s10557-013-6458-9
NLM
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Vancouver
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Artigo de periodico
Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis (2013)
Czaikoski, Paula Giselle; Nascimento, Daniele Carvalho; Sônego, Fabiane; Freitas, Andressa de; Turato, Walter Miguel; Carvalho, Michel A. de; Santos, Raquel Souza; Oliveira, Gisele Pena de; Samary, Cynthia dos Santos; Tefe-Silva, Cristiane; Alves Filho, José Carlos Farias; Ferreira, Sérgio Henrique; Rossi, Marcos Antonio; Rocco, Patricia Rieken Macedo; Spiller, Fernando; Cunha, Fernando de Queiroz
Source: Shock. Unidades: FCF, FMRP
Subjects: OXIGENASE, INIBIDORES DE ENZIMAS, NEUTRÓFILOS, SEPSE, PNEUMONIA
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ABNT
CZAIKOSKI, Paula Giselle et al. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, v. 39, n. 4, p. 389-396, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e31828bbcf9. Acesso em: 09 nov. 2024.
APA
Czaikoski, P. G., Nascimento, D. C., Sônego, F., Freitas, A. de, Turato, W. M., Carvalho, M. A. de, et al. (2013). Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, 39( 4), 389-396. doi:10.1097/SHK.0b013e31828bbcf9
NLM
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Vancouver
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Artigo de periodico
Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols (2013)
Kuga, Milton Carlos ; Faria, Gisele ; Rossi, Marcos Antonio; Monteiro, Jardel Camilo do Carmo ; Bonetti-Filho, Idomeo; Berbert, Fábio Luiz Camargo Vilella; Keine, Kátia Cristina ; Só, Marcus Vinicius Reis
Source: Scanning. Unidade: FMRP
Subjects: MATERIAIS DENTÁRIOS (ANÁLISE), CIMENTOS DENTÁRIOS (REMOÇÃO), OBTURAÇÃO DO CANAL RADICULAR
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ABNT
KUGA, Milton Carlos et al. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols. Scanning, v. 35, n. 1, p. 17-21, 2013Tradução . . Disponível em: https://doi.org/10.1002/sca.21030. Acesso em: 09 nov. 2024.
APA
Kuga, M. C., Faria, G., Rossi, M. A., Monteiro, J. C. do C., Bonetti-Filho, I., Berbert, F. L. C. V., et al. (2013). Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols. Scanning, 35( 1), 17-21. doi:10.1002/sca.21030
NLM
Kuga MC, Faria G, Rossi MA, Monteiro JC do C, Bonetti-Filho I, Berbert FLCV, Keine KC, Só MVR. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols [Internet]. Scanning. 2013 ; 35( 1): 17-21.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/sca.21030
Vancouver
Kuga MC, Faria G, Rossi MA, Monteiro JC do C, Bonetti-Filho I, Berbert FLCV, Keine KC, Só MVR. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols [Internet]. Scanning. 2013 ; 35( 1): 17-21.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/sca.21030
Artigo de periodico
Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy (2013)
Rizzi, Elen; Ceron, Carla S.; Guimarães, Danielle A.; Prado, Cibele Maria; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda; Santos, José Eduardo Tanus dos
Source: Experimental and Molecular Pathology. Unidades: FMRP, FORP
Subjects: METALOPROTEINASES, HIPERTENSÃO, DOENÇAS CARDIOVASCULARES
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ABNT
RIZZI, Elen et al. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, v. 94, n. 1, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.yexmp.2012.10.010. Acesso em: 09 nov. 2024.
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Santos, J. E. T. dos. (2013). Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, 94( 1), 1-9. doi:10.1016/j.yexmp.2012.10.010
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Artigo de periodico
Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure (2013)
Lataro, Renata Maria; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens; Rossi, Marcos Antonio; Prado, Cibele Maria; Godinho, Rosely Oliveira; Salgado, Helio Cesar
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: CORAÇÃO (FISIOPATOLOGIA;EFEITOS DE DROGAS), INIBIDORES DE ENZIMAS (FARMACOLOGIA), SISTEMA NERVOSO PARASSIMPÁTICO, VENTRÍCULO CARDÍACO
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ABNT
LATARO, Renata Maria et al. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 305, n. 8, p. R908-R916, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00102.2013. Acesso em: 09 nov. 2024.
APA
Lataro, R. M., Silva, C. A. A. da, Fazan Júnior, R., Rossi, M. A., Prado, C. M., Godinho, R. O., & Salgado, H. C. (2013). Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 305( 8), R908-R916. doi:10.1152/ajpregu.00102.2013
NLM
Lataro RM, Silva CAA da, Fazan Júnior R, Rossi MA, Prado CM, Godinho RO, Salgado HC. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 305( 8): R908-R916.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpregu.00102.2013
Vancouver
Lataro RM, Silva CAA da, Fazan Júnior R, Rossi MA, Prado CM, Godinho RO, Salgado HC. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 305( 8): R908-R916.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpregu.00102.2013
Artigo de periodico
Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis (2013)
Celes, Mara R. N.; Malvestio, Lygia M.; Suadicani, Sylvia O.; Prado, Cibele Maria; Figueiredo, Maria J.; Campos, Erica C.; Freitas, Ana C. S.; Spray, David C.; Tanowitz, Herbert B.; Silva, João Santana da; Rossi, Marcos Antonio
Source: PLOS ONE. Unidade: FMRP
Subjects: CARDIOPATIAS, SEPSE, EXPERIMENTOS ANIMAIS
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ABNT
CELES, Mara R. N. et al. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, v. 8, n. 7, p. e68809-1-e68809-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0068809. Acesso em: 09 nov. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Suadicani, S. O., Prado, C. M., Figueiredo, M. J., Campos, E. C., et al. (2013). Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, 8( 7), e68809-1-e68809-13. doi:10.1371/journal.pone.0068809
NLM
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Vancouver
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Artigo de periodico
5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection (2013)
Tristão, Fabrine Sales Massafera; Rocha, Fernanda Agostini; Moreira, Ana Paula; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio; Silva, João Santana da
Source: Infection and Immunity. Unidade: FMRP
Assunto: PARACOCCIDIOIDES BRASILIENSIS (IMUNOLOGIA)
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ABNT
TRISTÃO, Fabrine Sales Massafera et al. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection. Infection and Immunity, v. 81, n. 4, p. 1256-1266, 2013Tradução . . Disponível em: https://doi.org/10.1128/IAI.01209-12. Acesso em: 09 nov. 2024.
APA
Tristão, F. S. M., Rocha, F. A., Moreira, A. P., Cunha, F. de Q., Rossi, M. A., & Silva, J. S. da. (2013). 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection. Infection and Immunity, 81( 4), 1256-1266. doi:10.1128/IAI.01209-12
NLM
Tristão FSM, Rocha FA, Moreira AP, Cunha F de Q, Rossi MA, Silva JS da. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2013 ; 81( 4): 1256-1266.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1128/IAI.01209-12
Vancouver
Tristão FSM, Rocha FA, Moreira AP, Cunha F de Q, Rossi MA, Silva JS da. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2013 ; 81( 4): 1256-1266.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1128/IAI.01209-12
Trabalho de evento-resumo periodico
Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy (2012)
Campos, Erica C; Malvestio, L. M.; Romano, M. D.; Celes, M. N.; Blefari, V.; Prado, Cibele Maria; Rossi, Marco Antônio
Source: The Journal of Heart Disease. Conference titles: World Congress on Heart Disease. Unidade: FMRP
Subjects: MIOCARDIOPATIAS, FÁRMACOS
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ABNT
CAMPOS, Erica C et al. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2012
APA
Campos, E. C., Malvestio, L. M., Romano, M. D., Celes, M. N., Blefari, V., Prado, C. M., & Rossi, M. A. (2012). Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Campos EC, Malvestio LM, Romano MD, Celes MN, Blefari V, Prado CM, Rossi MA. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. The Journal of Heart Disease. 2012 ; 9( 1): 1.[citado 2024 nov. 09 ]
Vancouver
Campos EC, Malvestio LM, Romano MD, Celes MN, Blefari V, Prado CM, Rossi MA. Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy. The Journal of Heart Disease. 2012 ; 9( 1): 1.[citado 2024 nov. 09 ]
Artigo de periodico
Response of mice connective tissue to intracanal dressings containing chlorhexidine (2012)
Pereira, Maristela Soares Swerts; Faria, Gisele; Silva, Léa Assed Bezzerra da; Tanomaru Filho, Mario; Kuga, Milton Carlos; Rossi, Marcos Antonio
Source: Microscopy Research and Technique. Unidades: FORP, FMRP
Subjects: MICROSCÓPIO ÓTICO, TECIDO CONJUNTIVO SUBCUTÂNEO, HIDRÓXIDO DE CÁLCIO
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ABNT
PEREIRA, Maristela Soares Swerts et al. Response of mice connective tissue to intracanal dressings containing chlorhexidine. Microscopy Research and Technique, v. 75, n. 12, p. 1653-1658, 2012Tradução . . Disponível em: https://doi.org/10.1002/jemt.22112. Acesso em: 09 nov. 2024.
APA
Pereira, M. S. S., Faria, G., Silva, L. A. B. da, Tanomaru Filho, M., Kuga, M. C., & Rossi, M. A. (2012). Response of mice connective tissue to intracanal dressings containing chlorhexidine. Microscopy Research and Technique, 75( 12), 1653-1658. doi:10.1002/jemt.22112
NLM
Pereira MSS, Faria G, Silva LAB da, Tanomaru Filho M, Kuga MC, Rossi MA. Response of mice connective tissue to intracanal dressings containing chlorhexidine [Internet]. Microscopy Research and Technique. 2012 ; 75( 12): 1653-1658.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/jemt.22112
Vancouver
Pereira MSS, Faria G, Silva LAB da, Tanomaru Filho M, Kuga MC, Rossi MA. Response of mice connective tissue to intracanal dressings containing chlorhexidine [Internet]. Microscopy Research and Technique. 2012 ; 75( 12): 1653-1658.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/jemt.22112
Trabalho de evento-resumo
Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy (2012)
Rizzi, Elen; Ceron, Carla Speroni; Guimarães, Danielle Aparecida; Prado, Cibele Maria; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Source: Final Program. Conference titles: High Blood Pressure Research Scientific Sessions (HBPR). Unidades: FMRP, FORP
Subjects: ESTRESSE OXIDATIVO, METALOPROTEINASES, HIPERTROFIA VENTRICULAR ESQUERDA
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ABNT
RIZZI, Elen et al. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 09 nov. 2024.
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Tanus-Santos, J. E. (2012). Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2024 nov. 09 ]
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2024 nov. 09 ]
Artigo de periodico
The vasculature in Chagas Disease (2011)
Prado, Cibele M.; Jelicks, Linda A.; Weiss, Louis M.; Factor, Stephen M.; Tanowitz, Herbert B.; Rossi, Marcos Antonio
Source: Advances in Parasitology. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI, MIOCARDIOPATIA CHAGÁSICA, CITOCINAS, TROMBOXANOS, VASCULITE, ENDOTELINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, Cibele M. et al. The vasculature in Chagas Disease. Advances in Parasitology, v. 76, p. 83-99, 2011Tradução . . Disponível em: https://doi.org/10.1016/B978-0-12-385895-5.00004-9. Acesso em: 09 nov. 2024.
APA
Prado, C. M., Jelicks, L. A., Weiss, L. M., Factor, S. M., Tanowitz, H. B., & Rossi, M. A. (2011). The vasculature in Chagas Disease. Advances in Parasitology, 76, 83-99. doi:10.1016/B978-0-12-385895-5.00004-9
NLM
Prado CM, Jelicks LA, Weiss LM, Factor SM, Tanowitz HB, Rossi MA. The vasculature in Chagas Disease [Internet]. Advances in Parasitology. 2011 ; 76 83-99.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/B978-0-12-385895-5.00004-9
Vancouver
Prado CM, Jelicks LA, Weiss LM, Factor SM, Tanowitz HB, Rossi MA. The vasculature in Chagas Disease [Internet]. Advances in Parasitology. 2011 ; 76 83-99.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/B978-0-12-385895-5.00004-9
Artigo de periodico
Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms (2011)
Oliveira, Marcela S.; Floriano, Elaine Medeiros; Mazin, Suleimy Cristina; Martinez, Edson Zangiacomi; Vicente, Walter Villela de Andrade; Peres, Luiz Cesar; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Source: Cardiovascular Pathology. Unidade: FMRP
Subjects: ISQUEMIA MIOCÁRDICA, CARDIOPATIAS CONGÊNITAS, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Marcela S. et al. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, v. 20, n. 1, p. e43-e52, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.carpath.2010.01.012. Acesso em: 09 nov. 2024.
APA
Oliveira, M. S., Floriano, E. M., Mazin, S. C., Martinez, E. Z., Vicente, W. V. de A., Peres, L. C., et al. (2011). Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, 20( 1), e43-e52. doi:10.1016/j.carpath.2010.01.012
NLM
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Vancouver
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Trabalho de evento-resumo periodico
Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice (2011)
Celes, M. R. N; Prado, C. M.; Campos, E C; Malvestio, L. M.; Blefari, V; Cunha, F Q; Tanowitz, H. B; Rossi, Marcos Antonio
Source: The Journal of Heart Disease. Conference titles: World Congress on Heart Disease. Unidade: FMRP
Subjects: SEPSE, FÁRMACOS, MIOCARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, M. R. N et al. Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2011
APA
Celes, M. R. N., Prado, C. M., Campos, E. C., Malvestio, L. M., Blefari, V., Cunha, F. Q., et al. (2011). Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Celes MRN, Prado CM, Campos EC, Malvestio LM, Blefari V, Cunha FQ, Tanowitz HB, Rossi MA. Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2024 nov. 09 ]
Vancouver
Celes MRN, Prado CM, Campos EC, Malvestio LM, Blefari V, Cunha FQ, Tanowitz HB, Rossi MA. Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2024 nov. 09 ]
Artigo de periodico
Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis (2011)
Cavassani, Karen A.; Tristao, Fabrine S. M.; Oliveira, Leandro L.; Rocha, Fernanda A.; Vancim, Jaqueline O.; Moreira, Ana Paula; Campanelli, Ana Paula; Panagio, Luciano A.; Milanezi, Cristiane M.; Martinez, Roberto; Rossi, Marcos A.; Silva, João Santana
Source: PLos ONE. Unidades: FOB, FMRP
Subjects: INTERLEUCINA 4, PARACOCCIDIOIDES BRASILIENSIS, PARACOCCIDIOIDOMICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAVASSANI, Karen A. et al. Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis. PLos ONE, v. 6, n. 6, p. art. e17614 1-9, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0021423. Acesso em: 09 nov. 2024.
APA
Cavassani, K. A., Tristao, F. S. M., Oliveira, L. L., Rocha, F. A., Vancim, J. O., Moreira, A. P., et al. (2011). Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis. PLos ONE, 6( 6), art. e17614 1-9. doi:10.1371/journal.pone.0021423
NLM
Cavassani KA, Tristao FSM, Oliveira LL, Rocha FA, Vancim JO, Moreira AP, Campanelli AP, Panagio LA, Milanezi CM, Martinez R, Rossi MA, Silva JS. Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis [Internet]. PLos ONE. 2011 ; 6( 6): art. e17614 1-9.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0021423
Vancouver
Cavassani KA, Tristao FSM, Oliveira LL, Rocha FA, Vancim JO, Moreira AP, Campanelli AP, Panagio LA, Milanezi CM, Martinez R, Rossi MA, Silva JS. Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis [Internet]. PLos ONE. 2011 ; 6( 6): art. e17614 1-9.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0021423
Trabalho de evento-resumo periodico
Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy (2011)
Prado, C. M.; Celes, M. R. N; Malvestio, L. M.; Silva, J S; Jelicks, L. A; Tanowitz, H. B; Rossi, Marcos Antonio
Source: The Journal of Heart Disease. Conference titles: World Congress on Heart Disease. Unidade: FMRP
Subjects: MIOCARDIOPATIA CHAGÁSICA, FÁRMACOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, C. M. et al. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2011
APA
Prado, C. M., Celes, M. R. N., Malvestio, L. M., Silva, J. S., Jelicks, L. A., Tanowitz, H. B., & Rossi, M. A. (2011). Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Prado CM, Celes MRN, Malvestio LM, Silva JS, Jelicks LA, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2024 nov. 09 ]
Vancouver
Prado CM, Celes MRN, Malvestio LM, Silva JS, Jelicks LA, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2024 nov. 09 ]
Trabalho de evento-resumo periodico
Effect of pyridostigmine on the sympathovagal balance and cardiac mophometry in heart failure (2010)
Lataro, Renata M.; Silva, Carlos Alberto Aguiar da; Prado, Cibele M.; Rossi, Marcos Antonio; Fazan Júnior, Rubens; Salgado, Hélio Cesar
Source: The FASEB Journal. Conference titles: Experimental Biology. Unidade: FMRP
Subjects: FISIOLOGIA, MORFOMETRIA, CORAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LATARO, Renata M. et al. Effect of pyridostigmine on the sympathovagal balance and cardiac mophometry in heart failure. The FASEB Journal. Bethesda: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2010
APA
Lataro, R. M., Silva, C. A. A. da, Prado, C. M., Rossi, M. A., Fazan Júnior, R., & Salgado, H. C. (2010). Effect of pyridostigmine on the sympathovagal balance and cardiac mophometry in heart failure. The FASEB Journal. Bethesda: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Lataro RM, Silva CAA da, Prado CM, Rossi MA, Fazan Júnior R, Salgado HC. Effect of pyridostigmine on the sympathovagal balance and cardiac mophometry in heart failure. The FASEB Journal. 2010 ; 24[citado 2024 nov. 09 ]
Vancouver
Lataro RM, Silva CAA da, Prado CM, Rossi MA, Fazan Júnior R, Salgado HC. Effect of pyridostigmine on the sympathovagal balance and cardiac mophometry in heart failure. The FASEB Journal. 2010 ; 24[citado 2024 nov. 09 ]
Artigo de periodico
Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension (2010)
Rizzi, Élen; Castro, Michele M.; Prado, Cibele M.; Silva, Carlos A.; Fazan Junior, Rubens; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo; Gerlach, Raquel Fernanda
Source: Journal of Cardiac Failure. Unidades: FMRP, FORP
Subjects: HIPERTROFIA VENTRICULAR ESQUERDA, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIZZI, Élen et al. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, v. 16, n. 7, p. 599-608, 2010Tradução . . Acesso em: 09 nov. 2024.
APA
Rizzi, É., Castro, M. M., Prado, C. M., Silva, C. A., Fazan Junior, R., Rossi, M. A., et al. (2010). Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, 16( 7), 599-608.
NLM
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 nov. 09 ]
Vancouver
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 nov. 09 ]

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