Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase (2017)
- Authors:
- Dal Secco, Daniela
- DalBó, Silvia
- Lautherbach, Natalia Ennes da Silva
- Gava, Fábio N.
- Celes, Mara Rúbia Nunes
- Benedet, Patricia de OLiveira
- Souza, Adriana Helena de
- Akinaga, Juliana
- Lima, Vanessa
- Silva, Katiussia P.
- Kiguti, Luiz Ricardo A.
- Rossi, Marcos Antonio
- Kettelhut, Isis do Carmo
- Pupo, André Sampaio
- Cunha, Fernando de Queiroz
- Assreuy, Jamil
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; KETTELHUT, ISIS DO CARMO - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1152/ajpheart.00052.2016
- Subjects: ATIVAÇÃO ENZIMÁTICA; INSUFICIÊNCIA CARDÍACA; ÓXIDO NÍTRICO; SEPSE; TRANSDUÇÃO DE SINAL CELULAR
- Keywords: Adrenergic receptors; G protein receptor kinase; Inducible nitric oxide synthase; Nitric oxide; Sepsis
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: American Journal of Physiology-Heart and Circulatory Physiology
- ISSN: 0363-6135
- Volume/Número/Paginação/Ano: v. 313, n. 1, p. H149-H163, 2017
- Este artigo possui versão em acesso aberto
- URL de acesso aberto
- PDF de acesso aberto
- Versão do Documento: Versão publicada (Published version)
-
Status: Artigo possui acesso gratuito no site do editor (Bronze Open Access) -
ABNT
DAL SECCO, Daniela et al. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, v. 313, n. 1, p. H149-H163, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00052.2016. Acesso em: 14 mar. 2026. -
APA
Dal Secco, D., DalBó, S., Lautherbach, N. E. da S., Gava, F. N., Celes, M. R. N., Benedet, P. de O. L., et al. (2017). Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, 313( 1), H149-H163. doi:10.1152/ajpheart.00052.2016 -
NLM
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2026 mar. 14 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016 -
Vancouver
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2026 mar. 14 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016 - Cardiac changes in experimental sepsis
- Sepsis and the heart
- Verapamil ameliorates septic cardiomyopathy induced by cecal ligation and puncture in mice
- Sepsis and the heart
- Reduction of gap junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to sepsis
- Reduction of GAP junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to sepsis
- Sepsis and the heart
- Aorta remodeling due to stretch or turbulent flow/low wall shear stress: distinctly different biological phenomena ?
- Intercalated disc and dystrophin-glycoprotein complex remodeling in the hearts of mice submitted to severe sepsis
- Cardiac changes in experimental sepsis
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