Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy (2012)
- Authors:
- USP affiliated authors: SANTOS, JOSE EDUARDO TANUS DOS - FMRP ; GERLACH, RAQUEL FERNANDA - FORP ; ROSSI, MARCOS ANTONIO - FMRP
- Unidades: FMRP; FORP
- Subjects: ESTRESSE OXIDATIVO; METALOPROTEINASES; HIPERTROFIA VENTRICULAR ESQUERDA
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2012
- Source:
- Título: Final Program
- Conference titles: High Blood Pressure Research Scientific Sessions (HBPR)
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ABNT
RIZZI, Elen et al. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 01 dez. 2025. -
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Tanus-Santos, J. E. (2012). Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2025 dez. 01 ] -
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2025 dez. 01 ] - TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress
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- Inverse relationship between markers of nitric oxide formation and plasma matrix metalloproteinase-9 levels in healthy volunteers
- Circulating cell-free DNA levels in plasma increase with severity in experimental acute pulmonary thromboembolism
- Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension
- A role for matrix metalloproteinase-9 in the hemodynamic changes following acute pulmonary embolism
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