TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress (2011)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SANTOS, JOSE EDUARDO TANUS DOS - FMRP ; GERLACH, RAQUEL FERNANDA - FORP
- Unidades: FMRP; FORP
- Subjects: HIPERTENSÃO; ESTRESSE OXIDATIVO
- Language: Inglês
- Imprenta:
- Source:
- Título: Programa
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE)
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ABNT
RIZZI, E. et al. TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress. 2011, Anais.. São Paulo: FeSBE, 2011. . Acesso em: 28 jan. 2026. -
APA
Rizzi, E., Castro, M. M., Ceron, C. S., Neto-Neves, E. M., Prado, C. M., Rossi, M. A., et al. (2011). TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress. In Programa. São Paulo: FeSBE. -
NLM
Rizzi E, Castro MM, Ceron CS, Neto-Neves EM, Prado CM, Rossi MA, Tanus-Santos JE, Gerlach RF. TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress. Programa. 2011 ;[citado 2026 jan. 28 ] -
Vancouver
Rizzi E, Castro MM, Ceron CS, Neto-Neves EM, Prado CM, Rossi MA, Tanus-Santos JE, Gerlach RF. TGF-Beta and matrix metalloproteinases upregulation contribute to cardiac alterations in 2K1C hypertension via oxidative stress. Programa. 2011 ;[citado 2026 jan. 28 ] - Tempol inhibits TGF-'beta' and MMPs upregulation and prevents cardiac hypertensive changes
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- Nebivolol attenuates prooxidant and profibrotic mechanisms involving TGF-β and MMPs, and decreases vascular remodeling in renovascular hypertension
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- Nitrite or sildenafil, but not BAY 41-2272, blunt acute pulmonary embolism-induced increases in circulating matrix metalloproteinase-9 and oxidative stress
- Chronic treatment with nebivolol attenuates the vascular changes in the 2-kidney, 1-clip model of renovascular hypertension
- Circulating matrix metalloproteinases and their inhibitors in hypertension
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