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Artigo de periodico
Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility (2021)
Gil, Noemi Lourenço ; Azevedo, Gabriela A. ; Balbino, Aleksandro Martins ; Silva, Marina M. ; Carvalho, Maria Helena Catelli ; Akamine, Eliana Hiromi ; Keller, Alexandre C. ; Landgraf, Richardt G. ; Landgraf, Maristella de Almeida Vitta
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, RATOS WISTAR, LIPOPOLISSACARÍDEOS, CRESCIMENTO ANIMAL, INFLAMAÇÃO, HOMEOSTASE, PERDA DE PESO, INTERLEUCINA 6, NEUTRÓFILOS, MACRÓFAGOS, EXPRESSÃO GÊNICA, PROSTAGLANDINAS, LEUCOTRIENOS B
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GIL, Noemi Lourenço et al. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility. Life Sciences, v. 281, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119764. Acesso em: 28 jun. 2024.
APA
Gil, N. L., Azevedo, G. A., Balbino, A. M., Silva, M. M., Carvalho, M. H. C., Akamine, E. H., et al. (2021). Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility. Life Sciences, 281, 1-10. doi:10.1016/j.lfs.2021.119764
NLM
Gil NL, Azevedo GA, Balbino AM, Silva MM, Carvalho MHC, Akamine EH, Keller AC, Landgraf RG, Landgraf M de AV. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility [Internet]. Life Sciences. 2021 ; 281 1-10.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.lfs.2021.119764
Vancouver
Gil NL, Azevedo GA, Balbino AM, Silva MM, Carvalho MHC, Akamine EH, Keller AC, Landgraf RG, Landgraf M de AV. Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility [Internet]. Life Sciences. 2021 ; 281 1-10.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.lfs.2021.119764
Artigo de periodico
Gold nanoparticles reduce inflammation in cerebral microvessels of mice with sepsis (2021)
Bella, Davide Di ; Ferreira, João P. S.; Silva, Renée de Nazaré Oliveira da ; Pereira, Cinthya Echem de Souza; Milan, Aline ; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de ; Rodrigues, Stephen Fernandes de Paula
Source: Journal of Nanobiotechnology. Unidade: ICB
Subjects: FARMACOLOGIA, DOENÇAS CEREBRAIS, SEPSE, SEPTICEMIA, NANOPARTÍCULAS, MOLÉCULAS DE ADESÃO CELULAR, RATOS, ELETROENCEFALOGRAFIA, CAMUNDONGOS
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BELLA, Davide Di et al. Gold nanoparticles reduce inflammation in cerebral microvessels of mice with sepsis. Journal of Nanobiotechnology, v. 19, p. 15 , 2021Tradução . . Disponível em: https://doi.org/10.1186/s12951-021-00796-6. Acesso em: 28 jun. 2024.
APA
Bella, D. D., Ferreira, J. P. S., Silva, R. de N. O. da, Pereira, C. E. de S., Milan, A., Akamine, E. H., et al. (2021). Gold nanoparticles reduce inflammation in cerebral microvessels of mice with sepsis. Journal of Nanobiotechnology, 19, 15 . doi:10.1186/s12951-021-00796-6
NLM
Bella DD, Ferreira JPS, Silva R de NO da, Pereira CE de S, Milan A, Akamine EH, Carvalho MHC de, Rodrigues SF de P. Gold nanoparticles reduce inflammation in cerebral microvessels of mice with sepsis [Internet]. Journal of Nanobiotechnology. 2021 ; 19 15 .[citado 2024 jun. 28 ] Available from: https://doi.org/10.1186/s12951-021-00796-6
Vancouver
Bella DD, Ferreira JPS, Silva R de NO da, Pereira CE de S, Milan A, Akamine EH, Carvalho MHC de, Rodrigues SF de P. Gold nanoparticles reduce inflammation in cerebral microvessels of mice with sepsis [Internet]. Journal of Nanobiotechnology. 2021 ; 19 15 .[citado 2024 jun. 28 ] Available from: https://doi.org/10.1186/s12951-021-00796-6
Artigo de periodico
Immune spleen cells attenuate the inflammatory profile of the mesenteric perivascular adipose tissue in obese mice (2021)
Silva, Renée de Nazaré Oliveira da ; Eichler, Rosangela Aparecida dos Santos ; Dias, Carolina; Rodrigues, Stephen Fernandes de Paula ; Skiba, Dominik S. ; Landgraf, Richardt Gama ; Carvalho, Maria Helena Catelli de ; Guzik, Tomasz ; Fock, Ricardo Ambrósio ; Akamine, Eliana Hiromi
Source: Scientific Reports. Unidades: ICB, FCF
Subjects: TECIDO ADIPOSO, BAÇO, INFLAMAÇÃO, FARMACOLOGIA, DIETA ANIMAL, CITOMETRIA DE FLUXO, CITOCINAS, QUIMIOTAXIA DE LEUCÓCITO, OBESIDADE
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SILVA, Renée de Nazaré Oliveira da et al. Immune spleen cells attenuate the inflammatory profile of the mesenteric perivascular adipose tissue in obese mice. Scientific Reports, v. 11, p. 1-11 art. 11153, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41598-021-90600-0. Acesso em: 28 jun. 2024.
APA
Silva, R. de N. O. da, Eichler, R. A. dos S., Dias, C., Rodrigues, S. F. de P., Skiba, D. S., Landgraf, R. G., et al. (2021). Immune spleen cells attenuate the inflammatory profile of the mesenteric perivascular adipose tissue in obese mice. Scientific Reports, 11, 1-11 art. 11153. doi:10.1038/s41598-021-90600-0
NLM
Silva R de NO da, Eichler RA dos S, Dias C, Rodrigues SF de P, Skiba DS, Landgraf RG, Carvalho MHC de, Guzik T, Fock RA, Akamine EH. Immune spleen cells attenuate the inflammatory profile of the mesenteric perivascular adipose tissue in obese mice [Internet]. Scientific Reports. 2021 ; 11 1-11 art. 11153.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1038/s41598-021-90600-0
Vancouver
Silva R de NO da, Eichler RA dos S, Dias C, Rodrigues SF de P, Skiba DS, Landgraf RG, Carvalho MHC de, Guzik T, Fock RA, Akamine EH. Immune spleen cells attenuate the inflammatory profile of the mesenteric perivascular adipose tissue in obese mice [Internet]. Scientific Reports. 2021 ; 11 1-11 art. 11153.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1038/s41598-021-90600-0
Artigo de periodico
Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry (2019)
Filgueira, Fernando Paranaiba; Lobato, Núbia de Souza; Nascimento, Denise L.; Ceravolo, Graziela Scalianti; Giachini, Fernanda R. C.; Lima, Victor V.; Dantas, Ana Paula; Fortes, Zuleica Bruno; Webb, R. Clinton; Tostes, Rita de Cássia Aleixo ; Carvalho, Maria Helena Catelli de
Source: Steroids. Unidades: ICB, FMRP
Subjects: ESTRADIOL, ARTÉRIAS, VASODILATAÇÃO
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FILGUEIRA, Fernando Paranaiba et al. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry. Steroids, v. 141, p. 46-54, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.steroids.2018.11.006. Acesso em: 28 jun. 2024.
APA
Filgueira, F. P., Lobato, N. de S., Nascimento, D. L., Ceravolo, G. S., Giachini, F. R. C., Lima, V. V., et al. (2019). Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry. Steroids, 141, 46-54. doi:10.1016/j.steroids.2018.11.006
NLM
Filgueira FP, Lobato N de S, Nascimento DL, Ceravolo GS, Giachini FRC, Lima VV, Dantas AP, Fortes ZB, Webb RC, Tostes R de CA, Carvalho MHC de. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry [Internet]. Steroids. 2019 ; 141 46-54.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.steroids.2018.11.006
Vancouver
Filgueira FP, Lobato N de S, Nascimento DL, Ceravolo GS, Giachini FRC, Lima VV, Dantas AP, Fortes ZB, Webb RC, Tostes R de CA, Carvalho MHC de. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry [Internet]. Steroids. 2019 ; 141 46-54.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.steroids.2018.11.006
Artigo de periodico
Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 (2019)
Costa, Tiago Januário ; Altayó, Francesc Jiménez ; Pereira, Cinthya Echem de Souza; Akamine, Eliana Hiromi ; Tostes, Rita de Cássia Aleixo ; Vila, Elisabet ; Dantas, Ana Paula ; Carvalho, Maria Helena Catelli de
Source: Cells. Unidades: ICB, FMRP
Subjects: FAMACOLOGIA, ESTRÓGENOS, ARTÉRIAS CARÓTIDAS, EICOSANOIDES, ENSAIO CLÍNICO, ESTRADIOL, OVARIECTOMIA, CAMUNDONGOS
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COSTA, Tiago Januário et al. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, v. 8, n. 10, p. 17 , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8101217. Acesso em: 28 jun. 2024.
APA
Costa, T. J., Altayó, F. J., Pereira, C. E. de S., Akamine, E. H., Tostes, R. de C. A., Vila, E., et al. (2019). Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, 8( 10), 17 . doi:10.3390/cells8101217
NLM
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 jun. 28 ] Available from: https://doi.org/10.3390/cells8101217
Vancouver
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 jun. 28 ] Available from: https://doi.org/10.3390/cells8101217
Artigo de periodico
Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension (2019)
Pereira, Cinthya Echem de Souza ; Costa, Tiago Januário da ; Oliveira, Vanessa; Colli, Lucas Giglio; Landgraf, Maristella de Almeida Vitta ; Franco, Maria do Carmo Pinho ; Landgraf, Richardt Gama ; Santos-Eichler, Rosângela Aparecida ; Bomfim, Gisele Facholi ; Rodrigues, Stephen Fernandes de Paula ; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de
Source: Pharmacological Research. Unidades: ICB, FMRP
Subjects: MITOCÔNDRIAS, DNA MITOCONDRIAL, DIFERENÇAS SEXUAIS ANIMAL, AORTA DE ANIMAL, RATOS WISTAR, ESTRESSE OXIDATIVO
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PEREIRA, Cinthya Echem de Souza et al. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension. Pharmacological Research, v. 144, p. 141-150, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2019.04.008. Acesso em: 28 jun. 2024.
APA
Pereira, C. E. de S., Costa, T. J. da, Oliveira, V., Colli, L. G., Landgraf, M. de A. V., Franco, M. do C. P., et al. (2019). Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension. Pharmacological Research, 144, 141-150. doi:10.1016/j.phrs.2019.04.008
NLM
Pereira CE de S, Costa TJ da, Oliveira V, Colli LG, Landgraf M de AV, Franco M do CP, Landgraf RG, Santos-Eichler RA, Bomfim GF, Rodrigues SF de P, Akamine EH, Carvalho MHC de. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension [Internet]. Pharmacological Research. 2019 ; 144 141-150.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.phrs.2019.04.008
Vancouver
Pereira CE de S, Costa TJ da, Oliveira V, Colli LG, Landgraf M de AV, Franco M do CP, Landgraf RG, Santos-Eichler RA, Bomfim GF, Rodrigues SF de P, Akamine EH, Carvalho MHC de. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension [Internet]. Pharmacological Research. 2019 ; 144 141-150.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.phrs.2019.04.008
Artigo de periodico
Intrauterine and lactational exposure to fluoxetine enhances endothelial modulation of aortic contractile response in adult female rats (2018)
Higashi, Carolina Matias ; Sartoretto, Simone Marcieli ; Pereira, Cinthya Echem de Souza ; Lucchetti, Bruno Fernando Cruz ; Carvalho, Maria Helena Catelli de ; Gomes, Gislaine Garcia Pelosi ; Pinge Filho, Phileno ; Gerardin, Daniela Cristina Ceccatto ; Moreira, Estefânia Gastaldello ; Akamine, Eliana Hiromi ; Ceravolo, Graziela Scalianti
Source: Vascular Pharmacology. Unidade: ICB
Subjects: FARMACOLOGIA, ANTIDEPRESSIVOS, ENDOTÉLIO VASCULAR, AORTA DE ANIMAL, RATOS WISTAR, ÓXIDO NÍTRICO, VASODILATAÇÃO, CATALASE, ATIVAÇÃO ENZIMÁTICA
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HIGASHI, Carolina Matias et al. Intrauterine and lactational exposure to fluoxetine enhances endothelial modulation of aortic contractile response in adult female rats. Vascular Pharmacology, v. 108, p. 67-73, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2018.04.004. Acesso em: 28 jun. 2024.
APA
Higashi, C. M., Sartoretto, S. M., Pereira, C. E. de S., Lucchetti, B. F. C., Carvalho, M. H. C. de, Gomes, G. G. P., et al. (2018). Intrauterine and lactational exposure to fluoxetine enhances endothelial modulation of aortic contractile response in adult female rats. Vascular Pharmacology, 108, 67-73. doi:10.1016/j.vph.2018.04.004
NLM
Higashi CM, Sartoretto SM, Pereira CE de S, Lucchetti BFC, Carvalho MHC de, Gomes GGP, Pinge Filho P, Gerardin DCC, Moreira EG, Akamine EH, Ceravolo GS. Intrauterine and lactational exposure to fluoxetine enhances endothelial modulation of aortic contractile response in adult female rats [Internet]. Vascular Pharmacology. 2018 ; 108 67-73.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.vph.2018.04.004
Vancouver
Higashi CM, Sartoretto SM, Pereira CE de S, Lucchetti BFC, Carvalho MHC de, Gomes GGP, Pinge Filho P, Gerardin DCC, Moreira EG, Akamine EH, Ceravolo GS. Intrauterine and lactational exposure to fluoxetine enhances endothelial modulation of aortic contractile response in adult female rats [Internet]. Vascular Pharmacology. 2018 ; 108 67-73.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.vph.2018.04.004
Artigo de periodico
Protective effects of exogenous and endogenous hydrogensulfide in mast cell-mediated pruritus and cutaneous acute inflammation in mice (2017)
Rodrigues, L.; Ekundi-Valentim, E.; Florenzano, J.; Cerqueira, A. R. A.; Soares, A. G.; Schmidt, T. P.; Santos K. T.; Teixeira, S. A.; Ribela, Maria Teresa de Carvalho Pinto; Rodrigues, Stephen Fernandes de Paula ; de Carvalho, M. H.; De Nucci, Gilberto ; Wood, M.; Whiteman, M.; Muscará, Marcelo Nicolás ; Costa, Soraia Katia Pereira
Source: Pharmacological Research. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
RODRIGUES, L. et al. Protective effects of exogenous and endogenous hydrogensulfide in mast cell-mediated pruritus and cutaneous acute inflammation in mice. Pharmacological Research, v. 115, p. 255-266, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.11.006. Acesso em: 28 jun. 2024.
APA
Rodrigues, L., Ekundi-Valentim, E., Florenzano, J., Cerqueira, A. R. A., Soares, A. G., Schmidt, T. P., et al. (2017). Protective effects of exogenous and endogenous hydrogensulfide in mast cell-mediated pruritus and cutaneous acute inflammation in mice. Pharmacological Research, 115, 255-266. doi:10.1016/j.phrs.2016.11.006
NLM
Rodrigues L, Ekundi-Valentim E, Florenzano J, Cerqueira ARA, Soares AG, Schmidt TP, Santos K. T., Teixeira SA, Ribela MT de CP, Rodrigues SF de P, de Carvalho MH, De Nucci G, Wood M, Whiteman M, Muscará MN, Costa SKP. Protective effects of exogenous and endogenous hydrogensulfide in mast cell-mediated pruritus and cutaneous acute inflammation in mice [Internet]. Pharmacological Research. 2017 ; 115 255-266.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.phrs.2016.11.006
Vancouver
Rodrigues L, Ekundi-Valentim E, Florenzano J, Cerqueira ARA, Soares AG, Schmidt TP, Santos K. T., Teixeira SA, Ribela MT de CP, Rodrigues SF de P, de Carvalho MH, De Nucci G, Wood M, Whiteman M, Muscará MN, Costa SKP. Protective effects of exogenous and endogenous hydrogensulfide in mast cell-mediated pruritus and cutaneous acute inflammation in mice [Internet]. Pharmacological Research. 2017 ; 115 255-266.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.phrs.2016.11.006
Artigo de periodico
Laser photobiomodulation of pro-inflammatory mediators on Walker Tumor 256 induced rats (2017)
Petrellis, Maria Carla; Frigo, Lúcio; Marcos, Rodrigo Labat; Pallotta, Rodney Capp; Carvalho, Maria Helena Catelli de; Muscará, Marcelo Nicolás ; Maria, Durvanei Augusto; Lopes-Martins, Rodrigo Álvaro Brandão
Source: Journal of Photochemistry and Photobiology. B, Biology. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
PETRELLIS, Maria Carla et al. Laser photobiomodulation of pro-inflammatory mediators on Walker Tumor 256 induced rats. Journal of Photochemistry and Photobiology. B, Biology, v. 177, p. 69-75, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jphotobiol.2017.09.011. Acesso em: 28 jun. 2024.
APA
Petrellis, M. C., Frigo, L., Marcos, R. L., Pallotta, R. C., Carvalho, M. H. C. de, Muscará, M. N., et al. (2017). Laser photobiomodulation of pro-inflammatory mediators on Walker Tumor 256 induced rats. Journal of Photochemistry and Photobiology. B, Biology, 177, 69-75. doi:10.1016/j.jphotobiol.2017.09.011
NLM
Petrellis MC, Frigo L, Marcos RL, Pallotta RC, Carvalho MHC de, Muscará MN, Maria DA, Lopes-Martins RÁB. Laser photobiomodulation of pro-inflammatory mediators on Walker Tumor 256 induced rats [Internet]. Journal of Photochemistry and Photobiology. B, Biology. 2017 ; 177 69-75.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.jphotobiol.2017.09.011
Vancouver
Petrellis MC, Frigo L, Marcos RL, Pallotta RC, Carvalho MHC de, Muscará MN, Maria DA, Lopes-Martins RÁB. Laser photobiomodulation of pro-inflammatory mediators on Walker Tumor 256 induced rats [Internet]. Journal of Photochemistry and Photobiology. B, Biology. 2017 ; 177 69-75.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.jphotobiol.2017.09.011
Artigo de periodico
Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers (2017)
Soares, Antonio Garcia; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi
Source: BioMed Research International. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
SOARES, Antonio Garcia e CARVALHO, Maria Helena Catelli de e AKAMINE, Eliana Hiromi. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, v. 2017, p. 1-10, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/5038602. Acesso em: 28 jun. 2024.
APA
Soares, A. G., Carvalho, M. H. C. de, & Akamine, E. H. (2017). Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers. BioMed Research International, 2017, 1-10. doi:10.1155/2017/5038602
NLM
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1155/2017/5038602
Vancouver
Soares AG, Carvalho MHC de, Akamine EH. Obesity induces artery-specific alterations: evaluation of vascular function and inflammatory and smooth muscle phenotypic markers [Internet]. BioMed Research International. 2017 ; 2017 1-10.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1155/2017/5038602
Artigo de periodico
Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? (2017)
Oliveira, V.; Silva Junior, S. D.; Carvalho, M. H. C. de; Akamine, Eliana Hiromi ; Michelini, Lisete Compagno ; Franco, M. C.
Source: Journal of Developmental Origins of Health and Disease. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
OLIVEIRA, V. et al. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations?. Journal of Developmental Origins of Health and Disease, v. 8, n. 2, p. 236-243, 2017Tradução . . Disponível em: https://doi.org/10.1017/S2040174416000714. Acesso em: 28 jun. 2024.
APA
Oliveira, V., Silva Junior, S. D., Carvalho, M. H. C. de, Akamine, E. H., Michelini, L. C., & Franco, M. C. (2017). Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? Journal of Developmental Origins of Health and Disease, 8( 2), 236-243. doi:10.1017/S2040174416000714
NLM
Oliveira V, Silva Junior SD, Carvalho MHC de, Akamine EH, Michelini LC, Franco MC. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? [Internet]. Journal of Developmental Origins of Health and Disease. 2017 ; 8( 2): 236-243.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1017/S2040174416000714
Vancouver
Oliveira V, Silva Junior SD, Carvalho MHC de, Akamine EH, Michelini LC, Franco MC. Intrauterine growth restriction increases circulating mitochondrial DNA and Toll-like receptor 9 expression in adult offspring: could aerobic training counteract these adaptations? [Internet]. Journal of Developmental Origins of Health and Disease. 2017 ; 8( 2): 236-243.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1017/S2040174416000714
Artigo de periodico
Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats (2017)
Araujo, Priscila Xavier; Costa, Tiago Januário; Echem, Cinthya; Oliveira, Maria Aparecida de; Santos-Eichler, Rosangela Aparecida; Colli, Lucas Giglio; Jiménez-Altayó, Francesc; Vila, Elisabet; Akamine, Eliana Hiromi ; Dantas, Ana Paula; Ceravolo, Graziela Scalianti; Carvalho, Maria Helena Catelli de
Source: Journal of Pharmacology and Experimental Therapeutics. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
ARAUJO, Priscila Xavier et al. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, v. 362, n. 1, p. 98-107, 2017Tradução . . Disponível em: https://doi.org/10.1124/jpet.117.240465. Acesso em: 28 jun. 2024.
APA
Araujo, P. X., Costa, T. J., Echem, C., Oliveira, M. A. de, Santos-Eichler, R. A., Colli, L. G., et al. (2017). Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, 362( 1), 98-107. doi:10.1124/jpet.117.240465
NLM
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1124/jpet.117.240465
Vancouver
Araujo PX, Costa TJ, Echem C, Oliveira MA de, Santos-Eichler RA, Colli LG, Jiménez-Altayó F, Vila E, Akamine EH, Dantas AP, Ceravolo GS, Carvalho MHC de. Treatment with standard and low dose of conjugated equine estrogen differentially modulates estrogen receptor expression and response to angiotensin II in mesenteric venular bed of surgically postmenopausal hypertensive rats [Internet]. Journal of Pharmacology and Experimental Therapeutics. 2017 ; 362( 1): 98-107.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1124/jpet.117.240465
Artigo de periodico
Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products (2016)
Almeida, Paula Campi Locatelli de; Herrera, Bruno Schneider; Jesus, Flavia Neto de; Napolitano, Mauro; Teixeira, Simone Aparecida; Dantas, Aline Maia; Spolidorio, Luis Carlos; Akamine, Eliana Hiromi ; Mayer, Marcia Pinto Alves ; Carvalho, Maria Helena Catelli de; Costa, Soraia Katia Pereira ; Muscará, Marcelo Nicolás
Source: Archives of Oral Biology. Unidade: ICB
Subjects: PERIODONTITE, ENDOTÉLIO, AORTA, DOENÇAS CARDIOVASCULARES, ÓXIDO NÍTRICO, INFLAÇÃO
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ABNT
ALMEIDA, Paula Campi Locatelli de et al. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, v. 63, p. 66-74, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.archoralbio.2015.11.022. Acesso em: 28 jun. 2024.
APA
Almeida, P. C. L. de, Herrera, B. S., Jesus, F. N. de, Napolitano, M., Teixeira, S. A., Dantas, A. M., et al. (2016). Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products. Archives of Oral Biology, 63, 66-74. doi:10.1016/j.archoralbio.2015.11.022
NLM
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Vancouver
Almeida PCL de, Herrera BS, Jesus FN de, Napolitano M, Teixeira SA, Dantas AM, Spolidorio LC, Akamine EH, Mayer MPA, Carvalho MHC de, Costa SKP, Muscará MN. Endothelial dysfunction in rats with ligature-induced periodontitis: participation of nitric oxide and cycloxygenase-2-derived products [Internet]. Archives of Oral Biology. 2016 ; 63 66-74.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.archoralbio.2015.11.022
Artigo de periodico
H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
Costa, Rafael M.; Filgueira, Fernando P.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena C.; Akamine, Eliana Hiromi ; Lobato, Nubia S.
Source: Vascular Pharmacology. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 28 jun. 2024.
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Artigo de periodico
Reactive oxygen species: players in the cardiovascular effects oftestosterone (2016)
Tostes, Rita C.; Carvalho, Maria Helena Catelli; Carneiro, Fernando S.; Reckelhoff, Jane F.
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Subjects: FARMACOLOGIA, DOENÇAS CARDIOVASCULARES, ANTIOXIDANTES
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ABNT
TOSTES, Rita C. et al. Reactive oxygen species: players in the cardiovascular effects oftestosterone. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 28 jun. 2024.
APA
Tostes, R. C., Carvalho, M. H. C., Carneiro, F. S., & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects oftestosterone. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
NLM
Tostes RC, Carvalho MHC, Carneiro FS, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects oftestosterone [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Vancouver
Tostes RC, Carvalho MHC, Carneiro FS, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects oftestosterone [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Artigo de periodico
Reactive oxygen species: players in the cardiovascular effects of testosterone (2016)
Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Carvalho, Maria Helena Catelli de; Reckelhoff, Jane F.
Source: American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. Unidades: FMRP, ICB
Subjects: DOENÇAS CARDIOVASCULARES, SISTEMA CARDIOVASCULAR, TERAPIA DA REPOSIÇÃO HORMONAL, ESTRESSE OXIDATIVO, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TOSTES, Rita de Cássia Aleixo et al. Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 28 jun. 2024.
APA
Tostes, R. de C. A., Carneiro, F. S., Carvalho, M. H. C. de, & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
NLM
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Vancouver
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Artigo de periodico
Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a (2015)
Queiroz, Eveline A. I. F. de; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de; Sampaio, Sandra Concuzzo; Fortes, Zuleica Bruno
Source: Life Sciences. Unidade: ICB
Subjects: FARMACOLOGIA, TUMOR DE WALKER ANIMAL, RATOS, OBESIDADE
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ABNT
QUEIROZ, Eveline A. I. F. de et al. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a. Life Sciences, v. 121, p. 78-87, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.11.028. Acesso em: 28 jun. 2024.
APA
Queiroz, E. A. I. F. de, Akamine, E. H., Carvalho, M. H. C. de, Sampaio, S. C., & Fortes, Z. B. (2015). Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a. Life Sciences, 121, 78-87. doi:10.1016/j.lfs.2014.11.028
NLM
Queiroz EAIF de, Akamine EH, Carvalho MHC de, Sampaio SC, Fortes ZB. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a [Internet]. Life Sciences. 2015 ; 121 78-87.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.lfs.2014.11.028
Vancouver
Queiroz EAIF de, Akamine EH, Carvalho MHC de, Sampaio SC, Fortes ZB. Metformin reduces the Walker-256 tumor development in obese-MSG rats via AMPK and FOXO3a [Internet]. Life Sciences. 2015 ; 121 78-87.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1016/j.lfs.2014.11.028
Artigo de periodico
Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats (2015)
Bomfim, G. F.; Echem, Cinthya; Martins, C. B.; Costa, T. J.; Sartoretto, Simone Marcieli; Santos, R. A. dos; Oliveira, M. A.; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo; Webb, R. C.; Carvalho, Maria Helena Catelli de
Source: Life Sciences. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, INFLAMAÇÃO, HIPERTENSÃO, RATOS
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ABNT
BOMFIM, G. F. et al. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, v. 122, p. 1-7, 2015Tradução . . Disponível em: http://lattes.cnpq.br/5055165961832291. Acesso em: 28 jun. 2024.
APA
Bomfim, G. F., Echem, C., Martins, C. B., Costa, T. J., Sartoretto, S. M., Santos, R. A. dos, et al. (2015). Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, 122, 1-7. doi:10.1016/j.lfs.2014.12.001
NLM
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 jun. 28 ] Available from: http://lattes.cnpq.br/5055165961832291
Vancouver
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 jun. 28 ] Available from: http://lattes.cnpq.br/5055165961832291
Artigo de periodico
Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells (2015)
Costa, Tiago Januário da; Ceravolo, Graziela Scalianti; Santos, Rosangela Aparecida dos; Oliveira, Maria Aparecida de; Araújo, Priscila Xavier de; Giaquinto, Luciana Rigueiral; Tostes, Rita de Cássia Aleixo; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli de
Source: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, TESTOSTERONA, ESTROGENOS, AORTA DE ANIMAL, CÉLULAS ENDOTELIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, v. 308, n. 7, p. H723-H732, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00681.2014. Acesso em: 28 jun. 2024.
APA
Costa, T. J. da, Ceravolo, G. S., Santos, R. A. dos, Oliveira, M. A. de, Araújo, P. X. de, Giaquinto, L. R., et al. (2015). Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, 308( 7), H723-H732. doi:10.1152/ajpheart.00681.2014
NLM
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Vancouver
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 28 jun. 2024.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 jun. 28 ] Available from: https://doi.org/10.1042/CS20140548

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