H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
- Authors:
- USP affiliated authors: CARVALHO, MARIA HELENA CATELLI DE - ICB ; AKAMINE, ELIANA HIROMI - ICB ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP
- Unidades: ICB; FMRP
- DOI: 10.1016/j.vph.2016.05.008
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Vascular Pharmacology
- ISSN: 1879-3649
- Volume/Número/Paginação/Ano: v. 84, p. 28-37, 2016
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: unspecified-oa
-
ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 27 dez. 2025. -
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008 -
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008 -
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008 - Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility
- Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats
- Mechanisms of endothelial dysfunction in obesity-associated hypertension
- Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells
- Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats
- Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats
- Increased acetylcholine-induced vasodilation in pregnant rats: a role for gap junctional communication
- Increased endothelium dependent relaxation in arteries from pregnant rats: a role for gap junctional communicaion
- Hipersensibilidade a angiotensina II(angII) em artérias epigástricas superficiais de gestantes com pré-eclâmpsia está associada ao aumento da expressão de receptores At1
- Increased endothelium-dependent relaxation in arteries from pregnant rats: a role for Gap junctional communication
Informações sobre o DOI: 10.1016/j.vph.2016.05.008 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
