H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
- Authors:
- USP affiliated authors: CARVALHO, MARIA HELENA CATELLI DE - ICB ; AKAMINE, ELIANA HIROMI - ICB ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP
- Unidades: ICB; FMRP
- DOI: 10.1016/j.vph.2016.05.008
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Vascular Pharmacology
- ISSN: 1879-3649
- Volume/Número/Paginação/Ano: v. 84, p. 28-37, 2016
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 12 fev. 2026. -
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008 -
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008 -
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008 - Intrauterine growth restriction leads to a high-corticosterone producing offspring: an implication for pulmonary infection susceptibility
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Informações sobre o DOI: 10.1016/j.vph.2016.05.008 (Fonte: oaDOI API)
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