ReP USP - Resultado da busca

Artigo de periodico
Jaboticaba (Plinia jaboticaba (Vell.) Berg) polyphenols alleviate skeletal muscle insulin resistance by modulating PI3K/Akt/GLUT‑4 and AMPK signaling pathways in diet‑induced obese mice (2024)
Pessoa, Érika Vicencia Monteiro ; Moura, Márcio Hercules Caldas; Rodrigues, Larissa; Silva, Rafaela Rossi e; Castro, Érique de; Festuccia, William Tadeu Lara ; Pestana, Carlos Mario Donado ; Genovese, Maria Inês
Source: Food Production, Processing and Nutrition. Unidades: ICB, FCF
Subjects: JABUTICABA, POLIFENÓIS, INSULINA, MÚSCULO ESQUELÉTICO, DIETA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PESSOA, Érika Vicencia Monteiro et al. Jaboticaba (Plinia jaboticaba (Vell.) Berg) polyphenols alleviate skeletal muscle insulin resistance by modulating PI3K/Akt/GLUT‑4 and AMPK signaling pathways in diet‑induced obese mice. Food Production, Processing and Nutrition, v. 6, p. 1-9 art. 47, 2024Tradução . . Disponível em: https://dx.doi.org/10.1186/s43014-024-00230-y. Acesso em: 15 nov. 2024.
APA
Pessoa, É. V. M., Moura, M. H. C., Rodrigues, L., Silva, R. R. e, Castro, É. de, Festuccia, W. T. L., et al. (2024). Jaboticaba (Plinia jaboticaba (Vell.) Berg) polyphenols alleviate skeletal muscle insulin resistance by modulating PI3K/Akt/GLUT‑4 and AMPK signaling pathways in diet‑induced obese mice. Food Production, Processing and Nutrition, 6, 1-9 art. 47. doi:10.1186/s43014-024-00230-y
NLM
Pessoa ÉVM, Moura MHC, Rodrigues L, Silva RR e, Castro É de, Festuccia WTL, Pestana CMD, Genovese MI. Jaboticaba (Plinia jaboticaba (Vell.) Berg) polyphenols alleviate skeletal muscle insulin resistance by modulating PI3K/Akt/GLUT‑4 and AMPK signaling pathways in diet‑induced obese mice [Internet]. Food Production, Processing and Nutrition. 2024 ; 6 1-9 art. 47.[citado 2024 nov. 15 ] Available from: https://dx.doi.org/10.1186/s43014-024-00230-y
Vancouver
Pessoa ÉVM, Moura MHC, Rodrigues L, Silva RR e, Castro É de, Festuccia WTL, Pestana CMD, Genovese MI. Jaboticaba (Plinia jaboticaba (Vell.) Berg) polyphenols alleviate skeletal muscle insulin resistance by modulating PI3K/Akt/GLUT‑4 and AMPK signaling pathways in diet‑induced obese mice [Internet]. Food Production, Processing and Nutrition. 2024 ; 6 1-9 art. 47.[citado 2024 nov. 15 ] Available from: https://dx.doi.org/10.1186/s43014-024-00230-y
Artigo de periodico
Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function (2024)
Cardinali, Camila Aparecida Errerias Fernandes ; Martins, Yandara Akamine ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Alencar, Mayke Bezerra ; Silber, Ariel Mariano ; Torrão, Andréa da Silva
Source: ACS Chemical Neuroscience ISSN:1948-7193. Unidades: ICB, FM
Subjects: FISIOLOGIA, PARASITOLOGIA, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, HIPOCAMPU DE ANIMAL, COGNIÇÃO, INSULINA, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARDINALI, Camila Aparecida Errerias Fernandes et al. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function. ACS Chemical Neuroscience ISSN:1948-7193, v. 15, n. 16, p. 2982-2994, 2024Tradução . . Disponível em: https://doi.org/10.1021/acschemneuro.4c00113. Acesso em: 15 nov. 2024.
APA
Cardinali, C. A. E. F., Martins, Y. A., Moraes, R. C. M. de, Costa, A. P., Alencar, M. B., Silber, A. M., & Torrão, A. da S. (2024). Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function. ACS Chemical Neuroscience ISSN:1948-7193, 15( 16), 2982-2994. doi:10.1021/acschemneuro.4c00113
NLM
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Alencar MB, Silber AM, Torrão A da S. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function [Internet]. ACS Chemical Neuroscience ISSN:1948-7193. 2024 ; 15( 16): 2982-2994.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1021/acschemneuro.4c00113
Vancouver
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Alencar MB, Silber AM, Torrão A da S. Exploring the therapeutic potential of benfotiamine in a sporadic Alzheimer's-Like Disease rat model: insights into insulin signaling and cognitive function [Internet]. ACS Chemical Neuroscience ISSN:1948-7193. 2024 ; 15( 16): 2982-2994.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1021/acschemneuro.4c00113
Artigo de periodico
Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy (2023)
Ortiz-Silva, Milene ; Leonardi, Bianca Franco ; Castro, Érique de ; Peixoto, Albert Souza ; Gilio, Gustavo Renan ; Oliveira, Tiago E. ; Tomazelli, Caroline Aparecida ; Andrade, Maynara L.; Moreno, Mayara Franzoi ; Belchior, Thiago ; Magdalon, Juliana ; Vieira, Tayana S.; Donado-Pestana, Carlos M. ; Festuccia, William T.
Source: Molecular and Cellular Endocrinology. Unidades: ICB, FCF
Subjects: FISIOLOGIA, HOMEOSTASE, GLICOSE, LIPÍDEOS, ROEDORES, OBESIDADE, CAMUNDONGOS, DIETA ANIMAL, GORDURAS NA DIETA, ADIPOSIDADE, PESOS E MEDIDAS CORPORAIS, INSULINA, FIBROBLASTOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ORTIZ-SILVA, Milene et al. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, v. 578, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2023.112074. Acesso em: 15 nov. 2024.
APA
Ortiz-Silva, M., Leonardi, B. F., Castro, É. de, Peixoto, A. S., Gilio, G. R., Oliveira, T. E., et al. (2023). Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, 578, 11 . doi:10.1016/j.mce.2023.112074
NLM
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Vancouver
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Artigo de periodico
The adaptation of maternal energy metabolism to lactation and its underlying mechanisms (2022)
Anhê, Gabriel Forato ; Bordin, Silvana
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, METABOLISMO ENERGÉTICO, INSULINA, PROLACTINA, GRAVIDEZ, LACTAÇÃO, TECIDO ADIPOSO, MAMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANHÊ, Gabriel Forato e BORDIN, Silvana. The adaptation of maternal energy metabolism to lactation and its underlying mechanisms. Molecular and Cellular Endocrinology, v. 553, p. 1-22, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2022.111697. Acesso em: 15 nov. 2024.
APA
Anhê, G. F., & Bordin, S. (2022). The adaptation of maternal energy metabolism to lactation and its underlying mechanisms. Molecular and Cellular Endocrinology, 553, 1-22. doi:10.1016/j.mce.2022.111697
NLM
Anhê GF, Bordin S. The adaptation of maternal energy metabolism to lactation and its underlying mechanisms [Internet]. Molecular and Cellular Endocrinology. 2022 ; 553 1-22.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2022.111697
Vancouver
Anhê GF, Bordin S. The adaptation of maternal energy metabolism to lactation and its underlying mechanisms [Internet]. Molecular and Cellular Endocrinology. 2022 ; 553 1-22.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2022.111697
Artigo de periodico
Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion (2022)
Santos, Willian Oliveira dos ; Wasinski, Frederick ; Tavares, Mariana Rosolen ; Campos, Ana Maria Peracoli; Elias, Carol F. ; List, Edward O. ; Kopchick, John J. ; Szawka, Raphael E. ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIO DO CRESCIMENTO, HIPOTÁLAMO, LEPTINA, CAMUNDONGOS, RNA MENSAGEIRO, GLUTAMATOS, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Willian Oliveira dos et al. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion. Endocrinology, v. 163, n. 8, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac103. Acesso em: 15 nov. 2024.
APA
Santos, W. O. dos, Wasinski, F., Tavares, M. R., Campos, A. M. P., Elias, C. F., List, E. O., et al. (2022). Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion. Endocrinology, 163( 8), 1-17. doi:10.1210/endocr/bqac103
NLM
Santos WO dos, Wasinski F, Tavares MR, Campos AMP, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion [Internet]. Endocrinology. 2022 ; 163( 8): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac103
Vancouver
Santos WO dos, Wasinski F, Tavares MR, Campos AMP, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion [Internet]. Endocrinology. 2022 ; 163( 8): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac103
Artigo de periodico
Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells (2022)
Gusmão, Daniela de Oliveira ; Sousa, Maria Eletícia de ; Tavares, Mariana Rosolen ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIO DO CRESCIMENTO, HORMÔNIO LIBERADOR DE HORMÔNIO DE CRESCIMENTO, GLÂNDULA PITUITÁRIA, RNA MENSAGEIRO, FATOR DE CRESCIMENTO INSULIN-LIKE I, CAMUNDONGOS, HIPOTÁLAMO, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUSMÃO, Daniela de Oliveira et al. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells. Endocrinology, v. 163, n. 11, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac151. Acesso em: 15 nov. 2024.
APA
Gusmão, D. de O., Sousa, M. E. de, Tavares, M. R., & Donato Junior, J. (2022). Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells. Endocrinology, 163( 11), 1-13. doi:10.1210/endocr/bqac151
NLM
Gusmão D de O, Sousa ME de, Tavares MR, Donato Junior J. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells [Internet]. Endocrinology. 2022 ; 163( 11): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac151
Vancouver
Gusmão D de O, Sousa ME de, Tavares MR, Donato Junior J. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells [Internet]. Endocrinology. 2022 ; 163( 11): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac151
Artigo de periodico
BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 (2022)
Jesus, Daniel Simões de; Souza, Paula Bargi ; Cruzat, Vinicius ; Yechoor, Vijay; Carpinelli, Angelo Rafael ; Garcia, Rodrigo Antonio Peliciari
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, PÂNCREAS, INSULINA, RITMOS BIOLÓGICOS, SUPERÓXIDO DISMUTASE, FATORES DE TRANSCRIÇÃO, GLICOSE, CAMUNDONGOS, PERÓXIDO DE HIDROGÊNIO, GLUTATIONA TRANSFERASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JESUS, Daniel Simões de et al. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, v. 555, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2022.111725. Acesso em: 15 nov. 2024.
APA
Jesus, D. S. de, Souza, P. B., Cruzat, V., Yechoor, V., Carpinelli, A. R., & Garcia, R. A. P. (2022). BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2. Molecular and Cellular Endocrinology, 555, 1-8. doi:10.1016/j.mce.2022.111725
NLM
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Vancouver
Jesus DS de, Souza PB, Cruzat V, Yechoor V, Carpinelli AR, Garcia RAP. BMAL1 modulates ROS generation and insulin secretion in pancreatic β-cells: an effect possibly mediated via NOX2 [Internet]. Molecular and Cellular Endocrinology. 2022 ; 555 1-8.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2022.111725
Artigo de periodico
Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro (2022)
Valle, Maíra Mello Rezende ; Vilas-Boas, Eloisa Aparecida ; Lucena, Camila Ferraz ; Teixeira, Simone Aparecida ; Muscará, Marcelo Nicolás (Orientador) ; Carpinelli, Angelo Rafael
Source: Journal of Biochemical and Molecular Toxicology. Unidade: ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, METFORMINA, ANTIOXIDANTES, DIABETES MELLITUS, INSULINA, ESTRESSE OXIDATIVO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALLE, Maíra Mello Rezende et al. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro. Journal of Biochemical and Molecular Toxicology, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1002/jbt.23007. Acesso em: 15 nov. 2024.
APA
Valle, M. M. R., Vilas-Boas, E. A., Lucena, C. F., Teixeira, S. A., & Carpinelli, A. R. (2022). Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro. Journal of Biochemical and Molecular Toxicology, 1-13. doi:10.1002/jbt.23007
NLM
Valle MMR, Vilas-Boas EA, Lucena CF, Teixeira SA, Carpinelli AR. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro [Internet]. Journal of Biochemical and Molecular Toxicology. 2022 ; 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1002/jbt.23007
Vancouver
Valle MMR, Vilas-Boas EA, Lucena CF, Teixeira SA, Carpinelli AR. Metformin disrupts insulin secretion, causes proapoptotic and oxidative effects in rat pancreatic beta-cells in vitro [Internet]. Journal of Biochemical and Molecular Toxicology. 2022 ; 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1002/jbt.23007
Artigo de periodico
Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats (2022)
Moraes, Ruan Carlos Macêdo de ; Lima, Gabriely Cristina Alves; Cardinali, Camila Aparecida Errerias Fernandes ; Gonçalves, Alisson Carvalho de ; Portari, Guilherme Vannucchi ; Guerra-Shinohara, Elvira Maria ; Leboucher, Antoine ; Donato Junior, José ; Kleinridders, André ; Torrão, Andréa da Silva
Source: Life Sciences. Unidades: FCF, ICB, FM
Subjects: DOENÇA DE ALZHEIMER, HIPOTÁLAMO, FISIOLOGIA, VITAMINAS, DEMÊNCIA, INSULINA, DOENÇAS METABÓLICAS, METABOLISMO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇAS NEURODEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, Ruan Carlos Macêdo de et al. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, v. 306, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120841. Acesso em: 15 nov. 2024.
APA
Moraes, R. C. M. de, Lima, G. C. A., Cardinali, C. A. E. F., Gonçalves, A. C. de, Portari, G. V., Guerra-Shinohara, E. M., et al. (2022). Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, 306, 1-13. doi:10.1016/j.lfs.2022.120841
NLM
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Vancouver
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Artigo de periodico
INitial steps of Insulin action in parotid glands of male Wistar rats (2021)
Moraes, André Guaraci De Vito ; Marques, Victor Di Donato; Caperuto, Luciana Chagas ; Ibuki, Flávia Kazue ; Nogueira, Fernando Neves ; Francci, Carlos Eduardo ; Carvalho, Carla Roberta de Oliveira
Source: Cell Biochemistry and Biophysics. Unidades: FO, ICB
Subjects: FISIOLOGIA, INSULINA, GLÂNDULA PARÓTIDA, RATOS WISTAR, SALIVA, WESTERN BLOTTING
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, André Guaraci De Vito et al. INitial steps of Insulin action in parotid glands of male Wistar rats. Cell Biochemistry and Biophysics, p. 1-7, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12013-021-01025-5. Acesso em: 15 nov. 2024.
APA
Moraes, A. G. D. V., Marques, V. D. D., Caperuto, L. C., Ibuki, F. K., Nogueira, F. N., Francci, C. E., & Carvalho, C. R. de O. (2021). INitial steps of Insulin action in parotid glands of male Wistar rats. Cell Biochemistry and Biophysics, 1-7. doi:10.1007/s12013-021-01025-5
NLM
Moraes AGDV, Marques VDD, Caperuto LC, Ibuki FK, Nogueira FN, Francci CE, Carvalho CR de O. INitial steps of Insulin action in parotid glands of male Wistar rats [Internet]. Cell Biochemistry and Biophysics. 2021 ; 1-7.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12013-021-01025-5
Vancouver
Moraes AGDV, Marques VDD, Caperuto LC, Ibuki FK, Nogueira FN, Francci CE, Carvalho CR de O. INitial steps of Insulin action in parotid glands of male Wistar rats [Internet]. Cell Biochemistry and Biophysics. 2021 ; 1-7.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12013-021-01025-5
Artigo de periodico
New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia (2021)
Silva, Renée de Nazaré Oliveira da ; Llanos, Ricardo Pariona ; Eichler, Rosangela Aparecida dos Santos ; Oliveira, Thiago Belchior de ; Gozzo, Fábio Cesar ; Festuccia, William Tadeu Lara ; Ferro, Emer Suavinho
Source: Pharmaceutics. Unidade: ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, PEPTÍDEOS, INSULINA, DIABETES MELLITUS, GLICOSE, GLICEMIA, HEMOGLOBINAS, CAMUNDONGOS, CÉLULAS MUSCULARES, EXPRESSÃO GÊNICA, MEMBRANA PLASMÁTICA, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Renée de Nazaré Oliveira da et al. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia. Pharmaceutics, v. 13, n. 12, p. 1-18, 2021Tradução . . Disponível em: https://doi.org/10.3390/pharmaceutics13122175. Acesso em: 15 nov. 2024.
APA
Silva, R. de N. O. da, Llanos, R. P., Eichler, R. A. dos S., Oliveira, T. B. de, Gozzo, F. C., Festuccia, W. T. L., & Ferro, E. S. (2021). New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia. Pharmaceutics, 13( 12), 1-18. doi:10.3390/pharmaceutics13122175
NLM
Silva R de NO da, Llanos RP, Eichler RA dos S, Oliveira TB de, Gozzo FC, Festuccia WTL, Ferro ES. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia [Internet]. Pharmaceutics. 2021 ; 13( 12): 1-18.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/pharmaceutics13122175
Vancouver
Silva R de NO da, Llanos RP, Eichler RA dos S, Oliveira TB de, Gozzo FC, Festuccia WTL, Ferro ES. New intracellular peptide derived from Hemoglobin Alpha chain induces glucose uptake and reduces blood Glycemia [Internet]. Pharmaceutics. 2021 ; 13( 12): 1-18.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/pharmaceutics13122175
Artigo de periodico
Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets (2021)
Vilas-Boas, Eloisa Aparecida ; Nalbach, Lisa; Ampofo, Emmanuel; Lucena, Camila Ferraz ; Naudet, Léa ; Morgan, Bruce ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, OBESIDADE, DIABETES MELLITUS, HÁBITOS ALIMENTARES, ÁCIDOS GRAXOS, ILHOTAS DE LANGERHANS, ESTRESSE OXIDATIVO, CAMUNDONGOS, INSULINA, HOMEOSTASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VILAS-BOAS, Eloisa Aparecida et al. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, v. 162, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.11.023. Acesso em: 15 nov. 2024.
APA
Vilas-Boas, E. A., Nalbach, L., Ampofo, E., Lucena, C. F., Naudet, L., Morgan, B., et al. (2021). Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, 162, 13 . doi:10.1016/j.freeradbiomed.2020.11.023
NLM
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Vancouver
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Artigo de periodico
Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice (2021)
Wasinski, Frederick ; Chaves, Fernanda Machado; Pedroso, João Alfredo Bolivar ; Mansano, Naira da Silva ; Camporez, João Paulo Gabriel; Gusmão, Daniela O. ; List, Edward O. ; Kopchick, John J. ; Frazão, Renata ; Szawka, Raphael E. ; Donato Junior, José
Source: Journal of Neuroendocrinology. Unidades: FMRP, ICB
Subjects: ANATOMIA, FISIOLOGIA, RECEPTORES DE DOPAMINA, GLÂNDULA PITUITÁRIA, HIDROXILASE, HORMÔNIO DO CRESCIMENTO, PROLACTINA, ESTRESSE, CAMUNDONGOS, BALANÇO DE ENERGIA, HOMEOSTASE, INSULINA, HIPOGLICEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WASINSKI, Frederick et al. Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice. Journal of Neuroendocrinology, v. 33, n. 3, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.1111/jne.12957. Acesso em: 15 nov. 2024.
APA
Wasinski, F., Chaves, F. M., Pedroso, J. A. B., Mansano, N. da S., Camporez, J. P. G., Gusmão, D. O., et al. (2021). Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice. Journal of Neuroendocrinology, 33( 3), 1-13. doi:10.1111/jne.12957
NLM
Wasinski F, Chaves FM, Pedroso JAB, Mansano N da S, Camporez JPG, Gusmão DO, List EO, Kopchick JJ, Frazão R, Szawka RE, Donato Junior J. Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice [Internet]. Journal of Neuroendocrinology. 2021 ; 33( 3): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1111/jne.12957
Vancouver
Wasinski F, Chaves FM, Pedroso JAB, Mansano N da S, Camporez JPG, Gusmão DO, List EO, Kopchick JJ, Frazão R, Szawka RE, Donato Junior J. Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice [Internet]. Journal of Neuroendocrinology. 2021 ; 33( 3): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1111/jne.12957
Artigo de periodico
Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress (2021)
Vilas-Boas, Eloisa Aparecida ; Almeida, Davidson Correa ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Cells. Unidades: ICB, IQ
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, LIPÍDEOS, MEMBRANA PLASMÁTICA, OBESIDADE, ÁCIDOS GRAXOS, INSULINA, GLICOSE, CÉLULAS MORTAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VILAS-BOAS, Eloisa Aparecida et al. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, v. 10, n. 12, p. 1-23, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10123328. Acesso em: 15 nov. 2024.
APA
Vilas-Boas, E. A., Almeida, D. C., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2021). Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, 10( 12), 1-23. doi:10.3390/cells10123328
NLM
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/cells10123328
Vancouver
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/cells10123328
Artigo de periodico
miRNA array and oxi-proteome analyses reveal posttranscriptional actions of T3 on diabetic rats’ liver that could improve glycemia control (2020)
Costa, Ana Carolina Panveloski ; Braga, Camila Pereira ; Geraldo, Murilo Vieira ; Adamec, Jiri ; Nunes, Maria Tereza
Source: Metabolism. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIOS TIREOIDIANOS, DIABETES MELLITUS, HIPOTIREOIDISMO ANIMAL, RATOS, GLICEMIA, CITOCINAS, INSULINA, GLICOSE, REAÇÃO EM CADEIA POR POLIMERASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Ana Carolina Panveloski et al. miRNA array and oxi-proteome analyses reveal posttranscriptional actions of T3 on diabetic rats’ liver that could improve glycemia control. Metabolism, v. 104, p. 2 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2019.12.067. Acesso em: 15 nov. 2024.
APA
Costa, A. C. P., Braga, C. P., Geraldo, M. V., Adamec, J., & Nunes, M. T. (2020). miRNA array and oxi-proteome analyses reveal posttranscriptional actions of T3 on diabetic rats’ liver that could improve glycemia control. Metabolism, 104, 2 . doi:10.1016/j.metabol.2019.12.067
NLM
Costa ACP, Braga CP, Geraldo MV, Adamec J, Nunes MT. miRNA array and oxi-proteome analyses reveal posttranscriptional actions of T3 on diabetic rats’ liver that could improve glycemia control [Internet]. Metabolism. 2020 ; 104 2 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.metabol.2019.12.067
Vancouver
Costa ACP, Braga CP, Geraldo MV, Adamec J, Nunes MT. miRNA array and oxi-proteome analyses reveal posttranscriptional actions of T3 on diabetic rats’ liver that could improve glycemia control [Internet]. Metabolism. 2020 ; 104 2 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.metabol.2019.12.067
Artigo de periodico
Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells (2020)
Librais, Gabriela Nunes Marsiglio ; Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher; Hoffmann, Markus Daniel Alexander; Roma, Leticia Prates ; Carpinelli, Angelo Rafael
Source: Redox Report. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, ILHOTAS DE LANGERHANS, ÁCIDOS GRAXOS, GLICOSE, MEMBRANA PLASMÁTICA, PÂNCREAS, ÁCIDO LINOLEICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIBRAIS, Gabriela Nunes Marsiglio et al. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, v. 25, n. 1, p. 41–50, 2020Tradução . . Disponível em: https://doi.org/10.1080/13510002.2020.1757877. Acesso em: 15 nov. 2024.
APA
Librais, G. N. M., Vilas-Boas, E. A., Carlein, C., Hoffmann, M. D. A., Roma, L. P., & Carpinelli, A. R. (2020). Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, 25( 1), 41–50. doi:10.1080/13510002.2020.1757877
NLM
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Vancouver
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Artigo de periodico
MicroRNA miR-222 mediates pioglitazone beneficial effects on skeletal muscle of diet-induced obese mice (2020)
Mendonça, Mariana de ; Sousa, Érica de; Paixão, Ailma Oliveira da; Santos, Bruna de Araújo Cardoso dos ; Spagnol, Alexandre Roveratti ; Murata, Gilson Masahiro ; Araújo, Hygor Nunes ; Lima, Tanes Imamura de ; Guimarães, Dimitrius Santiago Passos Simões Fróes ; Silveira, Leonardo Reis ; Rodrigues, Alice Cristina
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FARMACOLOGIA, CAMUNDONGOS, INSULINA, RESISTÊNCIA À INSULINA, DIABETES MELLITUS, TECIDO ADIPOSO, ÁCIDOS GRAXOS, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MENDONÇA, Mariana de et al. MicroRNA miR-222 mediates pioglitazone beneficial effects on skeletal muscle of diet-induced obese mice. Molecular and Cellular Endocrinology, v. 501, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110661. Acesso em: 15 nov. 2024.
APA
Mendonça, M. de, Sousa, É. de, Paixão, A. O. da, Santos, B. de A. C. dos, Spagnol, A. R., Murata, G. M., et al. (2020). MicroRNA miR-222 mediates pioglitazone beneficial effects on skeletal muscle of diet-induced obese mice. Molecular and Cellular Endocrinology, 501, 10 . doi:10.1016/j.mce.2019.110661
NLM
Mendonça M de, Sousa É de, Paixão AO da, Santos B de AC dos, Spagnol AR, Murata GM, Araújo HN, Lima TI de, Guimarães DSPSF, Silveira LR, Rodrigues AC. MicroRNA miR-222 mediates pioglitazone beneficial effects on skeletal muscle of diet-induced obese mice [Internet]. Molecular and Cellular Endocrinology. 2020 ; 501 10 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2019.110661
Vancouver
Mendonça M de, Sousa É de, Paixão AO da, Santos B de AC dos, Spagnol AR, Murata GM, Araújo HN, Lima TI de, Guimarães DSPSF, Silveira LR, Rodrigues AC. MicroRNA miR-222 mediates pioglitazone beneficial effects on skeletal muscle of diet-induced obese mice [Internet]. Molecular and Cellular Endocrinology. 2020 ; 501 10 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.mce.2019.110661
Artigo de periodico
Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats (2020)
Silva, Flaviane de Fátima; Morais, Hely de; Silva, Milene Ortiz; Silva, Francemilson Goulart da ; Croffi, Rafael Vianna; Nascimento, Caroline Serrano ; Graciano, Maria Fernanda Rodrigues ; Carpinelli, Angelo Rafael ; Bazotte, Roberto Barbosa ; Souza, Helenir Medri de
Source: Journal of Cellular Biochemistry. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, TUMOR DE WALKER ANIMAL, CAQUEXIA, GLUTAMINA, DOENÇAS METABÓLICAS, CITOCINAS, RESISTÊNCIA À INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Flaviane de Fátima et al. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats. Journal of Cellular Biochemistry, p. 1-11, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcb.29682. Acesso em: 15 nov. 2024.
APA
Silva, F. de F., Morais, H. de, Silva, M. O., Silva, F. G. da, Croffi, R. V., Nascimento, C. S., et al. (2020). Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats. Journal of Cellular Biochemistry, 1-11. doi:10.1002/jcb.29682
NLM
Silva F de F, Morais H de, Silva MO, Silva FG da, Croffi RV, Nascimento CS, Graciano MFR, Carpinelli AR, Bazotte RB, Souza HM de. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats [Internet]. Journal of Cellular Biochemistry. 2020 ; 1-11.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1002/jcb.29682
Vancouver
Silva F de F, Morais H de, Silva MO, Silva FG da, Croffi RV, Nascimento CS, Graciano MFR, Carpinelli AR, Bazotte RB, Souza HM de. Akt activation by insulin treatment attenuates cachexia in Walker‐256 tumor‐bearing rats [Internet]. Journal of Cellular Biochemistry. 2020 ; 1-11.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1002/jcb.29682
Artigo de periodico
Mice born to females with oocytespecific deletion of mitofusin 2 have increased weight gain and impaired glucose homeostasis (2020)
Garcia, Bruna M. ; Machado, Thiago Simões; Carvalho, Karen F.; Nolasco, Patrícia ; Nociti, Ricardo Perecin ; Del Collado, Maite Barrondo ; Bianco, Maria J. D. Capo ; Grejo, Mateus P.; Augustro Neto, José Djaci Augusto ; Sugiyama, Fabrícia H. C. ; Tostes, Katiane ; Pandey, Anand K. ; Gonçalves, Luciana M.; Perecin, Felipe ; Meirelles, Flávio Vieira ; Ferraz, José Bento Sterman ; Vanzela, Emerielle C.; Boschero, Antônio C. ; Guimarães, Francisco Eduardo Gontijo ; Abdulkader, Fernando Rodrigues de Moraes ; Laurindo, Francisco R. M; Kowaltowski, Alicia Juliana ; Chiaratti, Marcos Roberto
Source: Molecular Human Reproduction. Unidades: FZEA, IFSC, ICB, IQ, IB
Subjects: MITOCÔNDRIAS, OBESIDADE, DIABETES MELLITUS, FISIOLOGIA, INSULINA, OÓCITOS, DOENÇAS METABÓLICAS, RETÍCULO ENDOPLASMÁTICO, CAMUNDONGOS, GLICOSE, HOMEOSTASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GARCIA, Bruna M. et al. Mice born to females with oocytespecific deletion of mitofusin 2 have increased weight gain and impaired glucose homeostasis. Molecular Human Reproduction, v. 26, n. 12, p. 938-952 + supplementary data, 2020Tradução . . Disponível em: https://doi.org/10.1093/molehr/gaaa071. Acesso em: 15 nov. 2024.
APA
Garcia, B. M., Machado, T. S., Carvalho, K. F., Nolasco, P., Nociti, R. P., Del Collado, M. B., et al. (2020). Mice born to females with oocytespecific deletion of mitofusin 2 have increased weight gain and impaired glucose homeostasis. Molecular Human Reproduction, 26( 12), 938-952 + supplementary data. doi:10.1093/molehr/gaaa071
NLM
Garcia BM, Machado TS, Carvalho KF, Nolasco P, Nociti RP, Del Collado MB, Bianco MJDC, Grejo MP, Augustro Neto JDA, Sugiyama FHC, Tostes K, Pandey AK, Gonçalves LM, Perecin F, Meirelles FV, Ferraz JBS, Vanzela EC, Boschero AC, Guimarães FEG, Abdulkader FR de M, Laurindo FRM, Kowaltowski AJ, Chiaratti MR. Mice born to females with oocytespecific deletion of mitofusin 2 have increased weight gain and impaired glucose homeostasis [Internet]. Molecular Human Reproduction. 2020 ; 26( 12): 938-952 + supplementary data.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/molehr/gaaa071
Vancouver
Garcia BM, Machado TS, Carvalho KF, Nolasco P, Nociti RP, Del Collado MB, Bianco MJDC, Grejo MP, Augustro Neto JDA, Sugiyama FHC, Tostes K, Pandey AK, Gonçalves LM, Perecin F, Meirelles FV, Ferraz JBS, Vanzela EC, Boschero AC, Guimarães FEG, Abdulkader FR de M, Laurindo FRM, Kowaltowski AJ, Chiaratti MR. Mice born to females with oocytespecific deletion of mitofusin 2 have increased weight gain and impaired glucose homeostasis [Internet]. Molecular Human Reproduction. 2020 ; 26( 12): 938-952 + supplementary data.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/molehr/gaaa071
Artigo de periodico
Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats (2020)
Bonassa, Ana Cláudia Munhoz ; Vilas-Boas, Eloisa Aparecida ; Costa, Ana Carolina Panveloski ; Leite, Jaqueline Santos Moreira ; Monaco, Camila Ferraz Lucena ; Patrício, Patrícia Riva; Emilio, Henriette; Carpinelli, Angelo Rafael
Source: Nutrients. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, ILHOTAS DE LANGERHANS, JEJUNO DE ANIMAL, RATOS WISTAR, GLICOSE, PLASMA DE ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BONASSA, Ana Cláudia Munhoz et al. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats. Nutrients, v. 12, n. 4, p. 24 , 2020Tradução . . Disponível em: https://doi.org/10.3390/nu12041029. Acesso em: 15 nov. 2024.
APA
Bonassa, A. C. M., Vilas-Boas, E. A., Costa, A. C. P., Leite, J. S. M., Monaco, C. F. L., Patrício, P. R., et al. (2020). Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats. Nutrients, 12( 4), 24 . doi:10.3390/nu12041029
NLM
Bonassa ACM, Vilas-Boas EA, Costa ACP, Leite JSM, Monaco CFL, Patrício PR, Emilio H, Carpinelli AR. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats [Internet]. Nutrients. 2020 ; 12( 4): 24 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/nu12041029
Vancouver
Bonassa ACM, Vilas-Boas EA, Costa ACP, Leite JSM, Monaco CFL, Patrício PR, Emilio H, Carpinelli AR. Intermittent fasting for twelve weeks leads to increases in fat mass and hyperinsulinemia in young female Wistar rats [Internet]. Nutrients. 2020 ; 12( 4): 24 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/nu12041029

USP Schools

ReP

Filtros

Document Types

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Publisher

Conference titles

Countries/Territories

Grupo de pesquisa

Funding Agencies

Indexado em

Non normalized affiliation

Countries of institutions of collaboration