ReP USP - Resultado da busca

Artigo de periodico
Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes (2024)
Michalani, Maria Luiza Estimo ; Passarelli, Marisa ; Machado, Ubiratan Fabres
Fonte: International Journal of Molecular Sciences. Unidades: FM, ICB
Assuntos: FISIOLOGIA, ADIPOSIDADE, TECIDO ADIPOSO, EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, TRANSCRIÇÃO GÊNICA, DIABETES MELLITUS, GLICOSE, WESTERN BLOTTING, ATIVAÇÃO ENZIMÁTICA, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MICHALANI, Maria Luiza Estimo e PASSARELLI, Marisa e MACHADO, Ubiratan Fabres. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes. International Journal of Molecular Sciences, v. 25, n. 15, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25158242. Acesso em: 13 nov. 2024.
APA
Michalani, M. L. E., Passarelli, M., & Machado, U. F. (2024). Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes. International Journal of Molecular Sciences, 25( 15), 14 . doi:10.3390/ijms25158242
NLM
Michalani MLE, Passarelli M, Machado UF. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 15): 14 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms25158242
Vancouver
Michalani MLE, Passarelli M, Machado UF. Nuclear Factor-Kappa-B mediates the advanced glycation end product-induced repression of Slc2a4 gene expression in 3T3-L1 adipocytes [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 15): 14 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms25158242
Artigo de periodico
Programming of metabolism by adipokines during development (2023)
Donato Junior, José
Fonte: Nature Reviews Endocrinology. Unidade: ICB
Assuntos: FISIOLOGIA, TECIDO ADIPOSO, METABOLISMO, DOENÇAS METABÓLICAS, HOMEOSTASE, GLICOSE, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DONATO JUNIOR, José. Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41574-023-00828-1. Acesso em: 13 nov. 2024.
APA
Donato Junior, J. (2023). Programming of metabolism by adipokines during development. Nature Reviews Endocrinology, 1-13. doi:10.1038/s41574-023-00828-1
NLM
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Vancouver
Donato Junior J. Programming of metabolism by adipokines during development [Internet]. Nature Reviews Endocrinology. 2023 ; 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41574-023-00828-1
Artigo de periodico-carta/editorial
Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism (2023)
Caballero-Sánchez, Noemi ; Winn, Nathan ; Laszlo Nagy ; Rosa Neto, José Cesar
Fonte: Frontiers Endocrinology. Unidade: ICB
Assuntos: BIOLOGIA CELULAR, MÚSCULO ESQUELÉTICO, GLICOSE, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CABALLERO-SÁNCHEZ, Noemi et al. Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism. Frontiers Endocrinology. Lausanne: Frontiers Research Foundation. Disponível em: https://doi.org/10.3389/fendo.2023.1219036. Acesso em: 13 nov. 2024. , 2023
APA
Caballero-Sánchez, N., Winn, N., Laszlo Nagy,, & Rosa Neto, J. C. (2023). Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism. Frontiers Endocrinology. Lausanne: Frontiers Research Foundation. doi:10.3389/fendo.2023.1219036
NLM
Caballero-Sánchez N, Winn N, Laszlo Nagy, Rosa Neto JC. Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism [Internet]. Frontiers Endocrinology. 2023 ; 14[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fendo.2023.1219036
Vancouver
Caballero-Sánchez N, Winn N, Laszlo Nagy, Rosa Neto JC. Corrigendum: Editorial: Cross-talk between heterogeneous cell types in skeletal muscle: implications for glucose metabolism [Internet]. Frontiers Endocrinology. 2023 ; 14[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fendo.2023.1219036
Artigo de periodico
The role of captopril in leukotriene deficient type 1 diabetic mice (2023)
Guimarâes, João Pedro Tôrres ; Queiroz, Luiz Adriano Damasceno de ; Menikdiwela, Kalhara R. ; Pereira, Nayara ; Ramalho, Theresa Raquel de Oliveira ; Moussa, Naima Moustaid ; Martins, Joilson de Oliveira ; Jancar, Sonia
Fonte: Scientific Reports. Unidades: FCF, ICB
Assuntos: IMUNOLOGIA, RESISTÊNCIA À INSULINA, SISTEMA RENINA-ANGIOTENSINA, CAMUNDONGOS, ANGIOTENSINAS, GLICOSE, LEUCOTRIENOS, MODELOS ANIMAIS DE DOENÇAS, FÍGADO, FÁRMACOS (SISTEMA CARDIOVASCULAR), DIABETES MELLITUS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUIMARÂES, João Pedro Tôrres et al. The role of captopril in leukotriene deficient type 1 diabetic mice. Scientific Reports, v. 13, n. 1, p. 15 , 2023Tradução . . Disponível em: https://doi.org/10.1038/s41598-023-49449-8. Acesso em: 13 nov. 2024.
APA
Guimarâes, J. P. T., Queiroz, L. A. D. de, Menikdiwela, K. R., Pereira, N., Ramalho, T. R. de O., Moussa, N. M., et al. (2023). The role of captopril in leukotriene deficient type 1 diabetic mice. Scientific Reports, 13( 1), 15 . doi:10.1038/s41598-023-49449-8
NLM
Guimarâes JPT, Queiroz LAD de, Menikdiwela KR, Pereira N, Ramalho TR de O, Moussa NM, Martins J de O, Jancar S. The role of captopril in leukotriene deficient type 1 diabetic mice [Internet]. Scientific Reports. 2023 ; 13( 1): 15 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-023-49449-8.
Vancouver
Guimarâes JPT, Queiroz LAD de, Menikdiwela KR, Pereira N, Ramalho TR de O, Moussa NM, Martins J de O, Jancar S. The role of captopril in leukotriene deficient type 1 diabetic mice [Internet]. Scientific Reports. 2023 ; 13( 1): 15 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-023-49449-8.
Artigo de periodico
Metformin acts in the gut and induces gut-liver crosstalk (2023)
Tobar, Natália ; Rocha, Guilherme Z. ; Santos, Andrey dos ; Guadagnini, Dioze ; Assalin, Heloísa B. ; Camargo, Juliana Alves de ; Gonçalves, Any E. S. S. ; Pallis, Flavia R.; Oliveira, Alexandre G. ; Rocco, Silvana A. ; Neto, Raphael M. ; Sousa, Irene Layane de ; Alborguetti, Marcos R. ; Sforça, Mauricio L. ; Rodrigues, Patricia B.; Ludwig, Raissa G. ; Vanzela, Emerielle C. ; Brunetto, Sergio Q. ; Boer, Patricia A. ; Gontijo, José A. R. ; Geloneze, Bruno; Prada, Patricia O. ; Frolli, Franco; Curi, Rui ; Mori, Marcelo A. ; Vinolo, Marco A. R. ; Ramos, Celso D. ; Franchini, Kleber G. ; Tormena, Cláudio F. ; Saad, Mario J. A. ; Carvalho, Carla Roberta de Oliveira
Fonte: PNAS Nexus. Unidade: ICB
Assuntos: FISIOLOGIA, CAMUNDONGOS, ANTIDIABÉTICOS (HIPOGLICEMIANTES), DIABETES MELLITUS, HIPERGLICEMIA, METFORMINA, GLICOSE, FÍGADO, INTESTINO DE ANIMAL, TOMOGRAFIA COMPUTADORIZADA DE EMISSÃO, SISTEMA GASTROINTESTINAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TOBAR, Natália et al. Metformin acts in the gut and induces gut-liver crosstalk. PNAS Nexus, v. 120, n. 4, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2211933120. Acesso em: 13 nov. 2024.
APA
Tobar, N., Rocha, G. Z., Santos, A. dos, Guadagnini, D., Assalin, H. B., Camargo, J. A. de, et al. (2023). Metformin acts in the gut and induces gut-liver crosstalk. PNAS Nexus, 120( 4), 1-11. doi:10.1073/pnas.2211933120
NLM
Tobar N, Rocha GZ, Santos A dos, Guadagnini D, Assalin HB, Camargo JA de, Gonçalves AESS, Pallis FR, Oliveira AG, Rocco SA, Neto RM, Sousa IL de, Alborguetti MR, Sforça ML, Rodrigues PB, Ludwig RG, Vanzela EC, Brunetto SQ, Boer PA, Gontijo JAR, Geloneze B, Prada PO, Frolli F, Curi R, Mori MA, Vinolo MAR, Ramos CD, Franchini KG, Tormena CF, Saad MJA, Carvalho CR de O. Metformin acts in the gut and induces gut-liver crosstalk [Internet]. PNAS Nexus. 2023 ; 120( 4): 1-11.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1073/pnas.2211933120
Vancouver
Tobar N, Rocha GZ, Santos A dos, Guadagnini D, Assalin HB, Camargo JA de, Gonçalves AESS, Pallis FR, Oliveira AG, Rocco SA, Neto RM, Sousa IL de, Alborguetti MR, Sforça ML, Rodrigues PB, Ludwig RG, Vanzela EC, Brunetto SQ, Boer PA, Gontijo JAR, Geloneze B, Prada PO, Frolli F, Curi R, Mori MA, Vinolo MAR, Ramos CD, Franchini KG, Tormena CF, Saad MJA, Carvalho CR de O. Metformin acts in the gut and induces gut-liver crosstalk [Internet]. PNAS Nexus. 2023 ; 120( 4): 1-11.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1073/pnas.2211933120
Artigo de periodico
Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy (2023)
Ortiz-Silva, Milene ; Leonardi, Bianca Franco ; Castro, Érique de ; Peixoto, Albert Souza ; Gilio, Gustavo Renan ; Oliveira, Tiago E. ; Tomazelli, Caroline Aparecida ; Andrade, Maynara L.; Moreno, Mayara Franzoi ; Belchior, Thiago ; Magdalon, Juliana ; Vieira, Tayana S.; Donado-Pestana, Carlos M. ; Festuccia, William T.
Fonte: Molecular and Cellular Endocrinology. Unidades: ICB, FCF
Assuntos: FISIOLOGIA, HOMEOSTASE, GLICOSE, LIPÍDEOS, ROEDORES, OBESIDADE, CAMUNDONGOS, DIETA ANIMAL, GORDURAS NA DIETA, ADIPOSIDADE, PESOS E MEDIDAS CORPORAIS, INSULINA, FIBROBLASTOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ORTIZ-SILVA, Milene et al. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, v. 578, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2023.112074. Acesso em: 13 nov. 2024.
APA
Ortiz-Silva, M., Leonardi, B. F., Castro, É. de, Peixoto, A. S., Gilio, G. R., Oliveira, T. E., et al. (2023). Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy. Molecular and Cellular Endocrinology, 578, 11 . doi:10.1016/j.mce.2023.112074
NLM
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Vancouver
Ortiz-Silva M, Leonardi BF, Castro É de, Peixoto AS, Gilio GR, Oliveira TE, Tomazelli CA, Andrade ML, Moreno MF, Belchior T, Magdalon J, Vieira TS, Donado-Pestana CM, Festuccia WT. Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy [Internet]. Molecular and Cellular Endocrinology. 2023 ; 578 11 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.mce.2023.112074
Artigo de periodico
mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction (2023)
Souza, Gabriel Orefice de ; Teixeira, Pryscila Dryelle Sousa ; Camara, Niels Olsen Saraiva ; Donato Junior, José
Fonte: Cells. Unidade: ICB
Assuntos: IMUNOLOGIA, FISIOLOGIA, HIPOTÁLAMO, PROTEÍNAS, CAMUNDONGOS, GLICOSE, ALIMENTAÇÃO ANIMAL, NEURÔNIOS, DIETA ANIMAL, PESOS E MEDIDAS CORPORAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Gabriel Orefice de et al. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, v. 12, n. 20, p. 17 , 2023Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf. Acesso em: 13 nov. 2024.
APA
Souza, G. O. de, Teixeira, P. D. S., Camara, N. O. S., & Donato Junior, J. (2023). mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, 12( 20), 17 . doi:10.3390/cells12202442
NLM
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 nov. 13 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Vancouver
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 nov. 13 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Artigo de periodico
Neurolysin knockout mice in a diet-induced obesity model (2023)
Caprioli, Bruna; Eichler, Rosangela A. S. ; Silva, Renée N. O. ; Martucci, Luiz Felipe ; Reckziegel, Patricia ; Ferro, Emer Suavinho
Fonte: International Journal of Molecular Sciences. Unidades: ICB, FCF, FM
Assuntos: OLIGOPEPTÍDEOS, OBESIDADE, DIETA ANIMAL, ZINCO, CAMUNDONGOS, LEPTINA, RATOS, GLICOSE, EXPRESSÃO GÊNICA, GLUCAGON, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAPRIOLI, Bruna et al. Neurolysin knockout mice in a diet-induced obesity model. International Journal of Molecular Sciences, v. 24, n. 20, p. 22 , 2023Tradução . . Disponível em: https://doi.org/10.3390/ijms242015190. Acesso em: 13 nov. 2024.
APA
Caprioli, B., Eichler, R. A. S., Silva, R. N. O., Martucci, L. F., Reckziegel, P., & Ferro, E. S. (2023). Neurolysin knockout mice in a diet-induced obesity model. International Journal of Molecular Sciences, 24( 20), 22 . doi:10.3390/ijms242015190
NLM
Caprioli B, Eichler RAS, Silva RNO, Martucci LF, Reckziegel P, Ferro ES. Neurolysin knockout mice in a diet-induced obesity model [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 20): 22 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms242015190
Vancouver
Caprioli B, Eichler RAS, Silva RNO, Martucci LF, Reckziegel P, Ferro ES. Neurolysin knockout mice in a diet-induced obesity model [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 20): 22 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms242015190
Artigo de periodico
α-cell electrophysiology and the regulation of glucagon secretion (2023)
Gao, Rui ; Acreman, Samuel ; Ma, Jinfang; Wendt, Anne ; Quan Zhang ; Abdulkader, Fernando
Fonte: Journal of Endocrinology. Unidade: ICB
Assuntos: FISIOLOGIA, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, GLUCAGON, ELETROFISIOLOGIA, GLICOSE, ILHOTAS DE LANGERHANS, HIPOGLICEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GAO, Rui et al. α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, v. 26, n. 2, p. 1-20, 2023Tradução . . Disponível em: https://doi.org/10.1530/JOE-22-0295. Acesso em: 13 nov. 2024.
APA
Gao, R., Acreman, S., Ma, J., Wendt, A., Quan Zhang,, & Abdulkader, F. (2023). α-cell electrophysiology and the regulation of glucagon secretion. Journal of Endocrinology, 26( 2), 1-20. doi:10.1530/JOE-22-0295
NLM
Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1530/JOE-22-0295
Vancouver
Gao R, Acreman S, Ma J, Wendt A, Quan Zhang, Abdulkader F. α-cell electrophysiology and the regulation of glucagon secretion [Internet]. Journal of Endocrinology. 2023 ; 26( 2): 1-20.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1530/JOE-22-0295
Artigo de periodico
Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 (2023)
Ferreira, Clílton Kraüss de Oliveira ; Campolim, Clara Machado ; Zordão, Olívia Pizetta ; Simabuco, Fernando Moreira ; Anaruma, Chadi Pellegrini ; Pereira, Rodrigo Martins; Boico, Vitor Ferreira ; Salvino, Luiz Guilherme ; Costa, Maira Maftoum ; Ruiz, Nathalia Quintero ; Moura, Leandro Pereira de ; Saad, Mario Jose Abdalla ; Kim, Young-Bum ; Patricia de Oliveira Prada; Costa, Soraia Katia Pereira
Fonte: Toxicology Reports. Unidade: ICB
Assuntos: FARMACOLOGIA, POLUIÇÃO ATMOSFÉRICA, MACRÓFAGOS, TECIDO ADIPOSO, INFLAMAÇÃO, HOMEOSTASE, CAMUNDONGOS, METABOLISMO ENERGÉTICO, GLICEMIA, GLICOSE, RESISTÊNCIA À INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Clílton Kraüss de Oliveira et al. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4. Toxicology Reports, v. 11, p. 10-22, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.toxrep.2023.06.002. Acesso em: 13 nov. 2024.
APA
Ferreira, C. K. de O., Campolim, C. M., Zordão, O. P., Simabuco, F. M., Anaruma, C. P., Pereira, R. M., et al. (2023). Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4. Toxicology Reports, 11, 10-22. doi:10.1016/j.toxrep.2023.06.002
NLM
Ferreira CK de O, Campolim CM, Zordão OP, Simabuco FM, Anaruma CP, Pereira RM, Boico VF, Salvino LG, Costa MM, Ruiz NQ, Moura LP de, Saad MJA, Kim Y-B, Patricia de Oliveira Prada, Costa SKP. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 [Internet]. Toxicology Reports. 2023 ; 11 10-22.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.toxrep.2023.06.002
Vancouver
Ferreira CK de O, Campolim CM, Zordão OP, Simabuco FM, Anaruma CP, Pereira RM, Boico VF, Salvino LG, Costa MM, Ruiz NQ, Moura LP de, Saad MJA, Kim Y-B, Patricia de Oliveira Prada, Costa SKP. Subchronic exposure to 1,2-naphthoquinone induces adipose tissue inflammation and changes the energy homeostasis of mice, partially due to TNFR1 and TLR4 [Internet]. Toxicology Reports. 2023 ; 11 10-22.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.toxrep.2023.06.002
Artigo de periodico
Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia (2023)
Yonamine, Caio Yogi ; Michalani, Maria Luiza Estimo ; Moreira, Rafael Junges ; Machado, Ubiratan Fabres
Fonte: International Journal of Molecular Sciences. Unidade: ICB
Assuntos: FISIOLOGIA, GLICOSE, OBESIDADE, DIABETES MELLITUS, RESISTÊNCIA À INSULINA, DOENÇA DE PARKINSON, DEMÊNCIA, ESTRESSE OXIDATIVO, HIPOCAMPO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YONAMINE, Caio Yogi et al. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia. International Journal of Molecular Sciences, v. 24, n. 22, p. 24 , 2023Tradução . . Disponível em: https://doi.org/10.3390/ijms242216480. Acesso em: 13 nov. 2024.
APA
Yonamine, C. Y., Michalani, M. L. E., Moreira, R. J., & Machado, U. F. (2023). Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia. International Journal of Molecular Sciences, 24( 22), 24 . doi:10.3390/ijms242216480
NLM
Yonamine CY, Michalani MLE, Moreira RJ, Machado UF. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 22): 24 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms242216480
Vancouver
Yonamine CY, Michalani MLE, Moreira RJ, Machado UF. Glucose transport and utilization in the hippocampus: from neurophysiology to diabetes-related development of dementia [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 22): 24 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms242216480
Artigo de periodico
Constant fed-batch cultivation with glucose and propionate as co-substrate: a strategy to fine-tune polyhydroxyalkanoates monomeric composition in Pseudomonas spp. (2023)
Santos-Oliveira, Pedro Henrique ; Silva, Jefferson Gonçalves Pinheiro ; Blank, Lars Mathias ; Silva, Luiziana Ferreira da ; Gomez, José Gregório Cabrera
Fonte: International Journal of Biological Macromolecules. Unidades: ICB, BIOTECNOLOGIA
Assuntos: MICROBIOLOGIA, PSEUDOMONAS, BACTÉRIAS AERÓBICAS GRAM-NEGATIVAS, OXIDAÇÃO, GLICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS-OLIVEIRA, Pedro Henrique et al. Constant fed-batch cultivation with glucose and propionate as co-substrate: a strategy to fine-tune polyhydroxyalkanoates monomeric composition in Pseudomonas spp. International Journal of Biological Macromolecules, v. 256, p. 9 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.ijbiomac.2023.128287. Acesso em: 13 nov. 2024.
APA
Santos-Oliveira, P. H., Silva, J. G. P., Blank, L. M., Silva, L. F. da, & Gomez, J. G. C. (2023). Constant fed-batch cultivation with glucose and propionate as co-substrate: a strategy to fine-tune polyhydroxyalkanoates monomeric composition in Pseudomonas spp. International Journal of Biological Macromolecules, 256, 9 . doi:10.1016/j.ijbiomac.2023.128287
NLM
Santos-Oliveira PH, Silva JGP, Blank LM, Silva LF da, Gomez JGC. Constant fed-batch cultivation with glucose and propionate as co-substrate: a strategy to fine-tune polyhydroxyalkanoates monomeric composition in Pseudomonas spp. [Internet]. International Journal of Biological Macromolecules. 2023 ; 256 9 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ijbiomac.2023.128287
Vancouver
Santos-Oliveira PH, Silva JGP, Blank LM, Silva LF da, Gomez JGC. Constant fed-batch cultivation with glucose and propionate as co-substrate: a strategy to fine-tune polyhydroxyalkanoates monomeric composition in Pseudomonas spp. [Internet]. International Journal of Biological Macromolecules. 2023 ; 256 9 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ijbiomac.2023.128287
Artigo de periodico
Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice (2023)
Valdivia, Luís Felipe Galvão ; Castro, Érique ; Eichler, Rosangela Aparecida dos Santos ; Moreno, Mayara Franzoi ; Sousa, Érica de; Jardim, Giovanna Freitas Rodrigues; Peixoto, Álbert Souza ; Moraes, Maria Nathália ; Castrucci, Ana Maria de Lauro ; Nedergaard, Jan ; Petrovic, Natasa ; Reckziegel, Patrícia ; Festuccia, William Tadeu Lara
Fonte: American Journal of Physiology-Endocrinology and Metabolism. Unidades: ICB, IB, FCF
Assuntos: FISIOLOGIA, TERMOGÊNESE, TECIDO ADIPOSO, ACLIMATAÇÃO, GLICOSE, CAMUNDONGOS, PEROXISSOMOS, TEMPERATURA ANIMAL, PROTEÍNAS DA MEMBRANA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALDIVIA, Luís Felipe Galvão et al. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice. American Journal of Physiology-Endocrinology and Metabolism, v. 324, n. 4, p. E289-E373, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00217.202. Acesso em: 13 nov. 2024.
APA
Valdivia, L. F. G., Castro, É., Eichler, R. A. dos S., Moreno, M. F., Sousa, É. de, Jardim, G. F. R., et al. (2023). Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice. American Journal of Physiology-Endocrinology and Metabolism, 324( 4), E289-E373. doi:10.1152/ajpendo.00217.202
NLM
Valdivia LFG, Castro É, Eichler RA dos S, Moreno MF, Sousa É de, Jardim GFR, Peixoto ÁS, Moraes MN, Castrucci AM de L, Nedergaard J, Petrovic N, Reckziegel P, Festuccia WTL. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2023 ; 324( 4): E289-E373.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpendo.00217.202
Vancouver
Valdivia LFG, Castro É, Eichler RA dos S, Moreno MF, Sousa É de, Jardim GFR, Peixoto ÁS, Moraes MN, Castrucci AM de L, Nedergaard J, Petrovic N, Reckziegel P, Festuccia WTL. Cold acclimation and pioglitazone combined increase thermogenic capacity of brown and white adipose tissues but this does not translate into higher energy expenditure in mice [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2023 ; 324( 4): E289-E373.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpendo.00217.202
Artigo de periodico
Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation (2023)
Yonamine, Caio Yogi ; Passarelli, Marisa ; Suemoto, Claudia Kimie ; Pasqualucci, Carlos Augusto ; Jacob-Filho, Wilson ; Alves, Venâncio Avancini Ferreira ; Marie, Suely Kazue Nagahashi ; Giannella, Maria Lucia Correa ; Britto, Luiz Roberto ; Machado, Ubiratan Fabres
Fonte: Cells. Unidades: FM, ICB
Assuntos: FISIOLOGIA, DOENÇA DE ALZHEIMER, DIABETES MELLITUS, HIPOCAMPO, NEUROBLASTOMA, OBESIDADE, DOENÇAS NEURODEGENERATIVAS, ESTRESSE OXIDATIVO, GLICOSE, DNA POLIMERASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YONAMINE, Caio Yogi et al. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, v. 12, n. 9, p. 1-21, 2023Tradução . . Disponível em: https://doi.org/10.3390/cells12091250. Acesso em: 13 nov. 2024.
APA
Yonamine, C. Y., Passarelli, M., Suemoto, C. K., Pasqualucci, C. A., Jacob-Filho, W., Alves, V. A. F., et al. (2023). Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, 12( 9), 1-21. doi:10.3390/cells12091250
NLM
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells12091250
Vancouver
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells12091250
Artigo de periodico
How much (ATP) does it cost to build a trypanosome? A theoretical study on the quantity of ATP needed to maintain and duplicate a bloodstream-form Trypanosoma brucei cell (2023)
Nascimento, Janaina F. ; Souza, Rodolpho Ornitz Oliveira ; Alencar, Mayke B. ; Marsiccobetre, S. ; Murillo, Ana M. ; Damasceno, Flávia ; Girard, Richard M. B. M. ; Marchese, Letícia ; Luévano-Martinez, Luis A. ; Achjian, Renan W. ; Haanstra, Jurgen R. ; Michels, Paul A. M. ; Silber, Ariel Mariano
Fonte: PLoS Pathogens. Unidade: ICB
Assuntos: PARASITOLOGIA, TRYPANOSOMA, ANIMAIS PARASITOS, BIOMASSA, CICLO CELULAR, POLIMERIZAÇÃO, GLICOSE, GLICOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Janaina F. et al. How much (ATP) does it cost to build a trypanosome? A theoretical study on the quantity of ATP needed to maintain and duplicate a bloodstream-form Trypanosoma brucei cell. PLoS Pathogens, v. 19, n. 7, p. 35, 2023Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1011522. Acesso em: 13 nov. 2024.
APA
Nascimento, J. F., Souza, R. O. O., Alencar, M. B., Marsiccobetre, S., Murillo, A. M., Damasceno, F., et al. (2023). How much (ATP) does it cost to build a trypanosome? A theoretical study on the quantity of ATP needed to maintain and duplicate a bloodstream-form Trypanosoma brucei cell. PLoS Pathogens, 19( 7), 35. doi:10.1371/journal.ppat.1011522
NLM
Nascimento JF, Souza ROO, Alencar MB, Marsiccobetre S, Murillo AM, Damasceno F, Girard RMBM, Marchese L, Luévano-Martinez LA, Achjian RW, Haanstra JR, Michels PAM, Silber AM. How much (ATP) does it cost to build a trypanosome? A theoretical study on the quantity of ATP needed to maintain and duplicate a bloodstream-form Trypanosoma brucei cell [Internet]. PLoS Pathogens. 2023 ; 19( 7): 35.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1011522
Vancouver
Nascimento JF, Souza ROO, Alencar MB, Marsiccobetre S, Murillo AM, Damasceno F, Girard RMBM, Marchese L, Luévano-Martinez LA, Achjian RW, Haanstra JR, Michels PAM, Silber AM. How much (ATP) does it cost to build a trypanosome? A theoretical study on the quantity of ATP needed to maintain and duplicate a bloodstream-form Trypanosoma brucei cell [Internet]. PLoS Pathogens. 2023 ; 19( 7): 35.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1011522
Artigo de periodico
Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice (2022)
Veras, Katherine Maria de Araujo ; Monaco, Camila Ferraz Lucena ; Goedcke, Julia; Evangelista, Fabiana de Sant'Anna ; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Fonte: Frontiers in Endocrinology. Unidades: EACH, ICB
Assuntos: FISIOLOGIA, PROTEÍNAS DO LEITE, DIETA, EXERCÍCIO FÍSICO, TREINAMENTO AERÓBIO, SACAROSE, ILHOTAS DE LANGERHANS, RESISTÊNCIA À INSULINA, GLICOSE, OBESIDADE, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VERAS, Katherine Maria de Araujo et al. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, v. 13, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fendo.2022.881236. Acesso em: 13 nov. 2024.
APA
Veras, K. M. de A., Monaco, C. F. L., Goedcke, J., Evangelista, F. de S. 'A., Carpinelli, A. R., & Carvalho, C. R. de O. (2022). Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice. Frontiers in Endocrinology, 13, 1-12. doi:10.3389/fendo.2022.881236
NLM
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Vancouver
Veras KM de A, Monaco CFL, Goedcke J, Evangelista F de S'A, Carpinelli AR, Carvalho CR de O. Moderate exercise training combined with a high-fat and sucrose diet protects pancreatic islet function in male C57BL/6J mice [Internet]. Frontiers in Endocrinology. 2022 ; 13 1-12.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fendo.2022.881236
Artigo de periodico
Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice (2022)
Miranda, Juliane Braga ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Jensen, Leonardo ; Irigoyen, Maria Cláudia Costa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Fonte: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Assuntos: FISIOLOGIA, ANATOMIA, DIETA ANIMAL, GLICOSE, ISQUEMIA, OBESIDADE, CAMUNDONGOS, DOENÇAS CARDIOVASCULARES EM ANIMAL, DOENÇAS METABÓLICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MIRANDA, Juliane Braga et al. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, v. 56, n. 3, p. 293-309, 2022Tradução . . Disponível em: https://doi.org/10.33594/000000535. Acesso em: 13 nov. 2024.
APA
Miranda, J. B., Lunardon, G., Lima, V. M., Silva, T. de O., Lino, C. A., Jensen, L., et al. (2022). Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, 56( 3), 293-309. doi:10.33594/000000535
NLM
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 13 ] Available from: https://doi.org/10.33594/000000535
Vancouver
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 13 ] Available from: https://doi.org/10.33594/000000535
Artigo de periodico
Effects of captopril on glucose metabolism and autophagy in liver and muscle from mice with type 1 diabetes and diet-induced obesity (2022)
Guimarães, João Pedro Tôrres ; Menikdiwela, Kalhara R. ; Ramalho, Theresa Raquel de Oliveira ; Queiroz, Luiz Adriano Damasceno de ; Kalupahana, Nishan S. ; Jancar, Sonia ; Ramalingam, Latha ; Martins, Joilson de Oliveira ; Moussa, Naima Moustaid
Fonte: Biochimica et Biophysica Acta. Molecular Basis of Disease. Unidades: ICB, FCF
Assuntos: IMUNOLOGIA, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, FÁRMACOS (SISTEMA CARDIOVASCULAR), GLICOSE, SISTEMA RENINA-ANGIOTENSINA, DIABETES MELLITUS, OBESIDADE, FÍGADO, MÚSCULOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUIMARÃES, João Pedro Tôrres et al. Effects of captopril on glucose metabolism and autophagy in liver and muscle from mice with type 1 diabetes and diet-induced obesity. Biochimica et Biophysica Acta. Molecular Basis of Disease, v. 1868, n. 10, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2022.166477. Acesso em: 13 nov. 2024.
APA
Guimarães, J. P. T., Menikdiwela, K. R., Ramalho, T. R. de O., Queiroz, L. A. D. de, Kalupahana, N. S., Jancar, S., et al. (2022). Effects of captopril on glucose metabolism and autophagy in liver and muscle from mice with type 1 diabetes and diet-induced obesity. Biochimica et Biophysica Acta. Molecular Basis of Disease, 1868( 10), 1-13. doi:10.1016/j.bbadis.2022.166477
NLM
Guimarães JPT, Menikdiwela KR, Ramalho TR de O, Queiroz LAD de, Kalupahana NS, Jancar S, Ramalingam L, Martins J de O, Moussa NM. Effects of captopril on glucose metabolism and autophagy in liver and muscle from mice with type 1 diabetes and diet-induced obesity [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2022 ; 1868( 10): 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.bbadis.2022.166477
Vancouver
Guimarães JPT, Menikdiwela KR, Ramalho TR de O, Queiroz LAD de, Kalupahana NS, Jancar S, Ramalingam L, Martins J de O, Moussa NM. Effects of captopril on glucose metabolism and autophagy in liver and muscle from mice with type 1 diabetes and diet-induced obesity [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2022 ; 1868( 10): 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.bbadis.2022.166477
Artigo de periodico
SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism (2022)
Pedroso, João Alfredo Bolivar ; Silva, Ivson Bruno da ; Zampieri, Thais Tessari ; Totola, Leonardo Tedesco; Moreira, Thiago dos Santos ; Taniguti, Ana Paula Tiemi; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José
Fonte: International Journal of Molecular Sciences. Unidade: ICB
Assuntos: FISIOLOGIA, ANATOMIA, CARDIOPATIAS, HIPOTÁLAMO, LEPTINA, OBESIDADE, ENVELHECIMENTO, CAMUNDONGOS, METABOLISMO DE PROTEÍNA, METABOLISMO ENERGÉTICO, NEURÔNIOS, CITOCININAS, GLICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEDROSO, João Alfredo Bolivar et al. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, v. 23, n. 12, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23126484. Acesso em: 13 nov. 2024.
APA
Pedroso, J. A. B., Silva, I. B. da, Zampieri, T. T., Totola, L. T., Moreira, T. dos S., Taniguti, A. P. T., et al. (2022). SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, 23( 12), 1-19. doi:10.3390/ijms23126484
NLM
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms23126484
Vancouver
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms23126484
Artigo de periodico
Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study (2022)
Faria, Daniele de Paula ; Souza, Larissa Estessi de ; Duran, Fabio Luis de Souza ; Buchpiguel, Carlos Alberto ; Britto, Luiz Roberto Giorgetti de ; Crippa, José Alexandre de Souza ; Busatto Filho, Geraldo ; Gregório, Caroline Cristiano Real
Fonte: International Journal of Molecular Sciences. Unidades: FM, ICB, FMRP
Assuntos: FISIOLOGIA, CANABINOIDES, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, ANTINEOPLÁSICOS, MEMÓRIA ANIMAL, GLICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FARIA, Daniele de Paula et al. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, v. 23. n. 3, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23031076. Acesso em: 13 nov. 2024.
APA
Faria, D. de P., Souza, L. E. de, Duran, F. L. de S., Buchpiguel, C. A., Britto, L. R. G. de, Crippa, J. A. de S., et al. (2022). Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, 23. n. 3, 1-10. doi:10.3390/ijms23031076
NLM
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms23031076
Vancouver
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms23031076

Unidades USP

ReP

Filtros

Unidades USP

Departamento

Autores

Autores USP

Ano de publicação

Assuntos

Título da fonte

Editora

País de publicação

Agência de fomento

Indexado em

Afiliação dos autores externos normalizada

Afiliação dos autores externos não normalizada

País das instituições de afiliação dos autores externos