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Artigo de periodico
Nitric oxide favours tumour-promoting inflammation through mitochondria-dependent and -independent actions on macrophages (2022)
Drehmer, Daiana; Luiz, João Paulo Mesquita ; Hernandez, Cesar Augusto Speck; Alves Filho, José Carlos Farias ; Hussell, Tracy; Townsend, Paul Andrew; Moncada, Salvador
Source: Redox Biology. Unidade: FMRP
Subjects: INFLAMAÇÃO, MACRÓFAGOS, MITOCÔNDRIAS, NEOPLASIAS, ÓXIDO NÍTRICO
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DREHMER, Daiana et al. Nitric oxide favours tumour-promoting inflammation through mitochondria-dependent and -independent actions on macrophages. Redox Biology, v. 54, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2022.102350. Acesso em: 02 jul. 2024.
APA
Drehmer, D., Luiz, J. P. M., Hernandez, C. A. S., Alves Filho, J. C. F., Hussell, T., Townsend, P. A., & Moncada, S. (2022). Nitric oxide favours tumour-promoting inflammation through mitochondria-dependent and -independent actions on macrophages. Redox Biology, 54, 1-13. doi:10.1016/j.redox.2022.102350
NLM
Drehmer D, Luiz JPM, Hernandez CAS, Alves Filho JCF, Hussell T, Townsend PA, Moncada S. Nitric oxide favours tumour-promoting inflammation through mitochondria-dependent and -independent actions on macrophages [Internet]. Redox Biology. 2022 ; 54 1-13.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.redox.2022.102350
Vancouver
Drehmer D, Luiz JPM, Hernandez CAS, Alves Filho JCF, Hussell T, Townsend PA, Moncada S. Nitric oxide favours tumour-promoting inflammation through mitochondria-dependent and -independent actions on macrophages [Internet]. Redox Biology. 2022 ; 54 1-13.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.redox.2022.102350
Artigo de periodico
Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy (2022)
Cardoso, Fabrízio dos Santos; Gonzalez-Lima, Francisco; Coimbra, Norberto Cysne
Source: Frontiers in Neurology. Unidade: FMRP
Subjects: EPILEPSIA, TERAPIA A LASER, MITOCÔNDRIAS, ESTRESSE OXIDATIVO, CONVULSÕES
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CARDOSO, Fabrízio dos Santos e GONZALEZ-LIMA, Francisco e COIMBRA, Norberto Cysne. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy. Frontiers in Neurology, v. 13, p. 1-5, 2022Tradução . . Disponível em: https://doi.org/10.3389/fneur.2022.873496. Acesso em: 02 jul. 2024.
APA
Cardoso, F. dos S., Gonzalez-Lima, F., & Coimbra, N. C. (2022). Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy. Frontiers in Neurology, 13, 1-5. doi:10.3389/fneur.2022.873496
NLM
Cardoso F dos S, Gonzalez-Lima F, Coimbra NC. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy [Internet]. Frontiers in Neurology. 2022 ; 13 1-5.[citado 2024 jul. 02 ] Available from: https://doi.org/10.3389/fneur.2022.873496
Vancouver
Cardoso F dos S, Gonzalez-Lima F, Coimbra NC. Mitochondrial photobiomodulation as a neurotherapeutic strategy for epilepsy [Internet]. Frontiers in Neurology. 2022 ; 13 1-5.[citado 2024 jul. 02 ] Available from: https://doi.org/10.3389/fneur.2022.873496
Artigo de periodico
Expanding the limits of photodynamic therapy: the design of organelles and hypoxia-targeting nanomaterials for enhanced photokilling of cancer (2021)
Calori, Italo Rodrigo ; Bi, Hong; Tedesco, Antônio Cláudio
Source: ACS Applied Bio Materials. Unidade: FFCLRP
Subjects: TERAPIA FOTODINÂMICA, ANÓXIA, MITOCÔNDRIAS, LISOSSOMOS, OXIGÊNIO
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CALORI, Italo Rodrigo e BI, Hong e TEDESCO, Antônio Cláudio. Expanding the limits of photodynamic therapy: the design of organelles and hypoxia-targeting nanomaterials for enhanced photokilling of cancer. ACS Applied Bio Materials, v. 4, n. 1, p. 195-228, 2021Tradução . . Disponível em: https://doi.org/10.1021/acsabm.0c00945. Acesso em: 02 jul. 2024.
APA
Calori, I. R., Bi, H., & Tedesco, A. C. (2021). Expanding the limits of photodynamic therapy: the design of organelles and hypoxia-targeting nanomaterials for enhanced photokilling of cancer. ACS Applied Bio Materials, 4( 1), 195-228. doi:10.1021/acsabm.0c00945
NLM
Calori IR, Bi H, Tedesco AC. Expanding the limits of photodynamic therapy: the design of organelles and hypoxia-targeting nanomaterials for enhanced photokilling of cancer [Internet]. ACS Applied Bio Materials. 2021 ; 4( 1): 195-228.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1021/acsabm.0c00945
Vancouver
Calori IR, Bi H, Tedesco AC. Expanding the limits of photodynamic therapy: the design of organelles and hypoxia-targeting nanomaterials for enhanced photokilling of cancer [Internet]. ACS Applied Bio Materials. 2021 ; 4( 1): 195-228.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1021/acsabm.0c00945
Artigo de periodico
The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition (2021)
Queiroz, André L.; Lessard, Sarah J.; Ouchida, Amanda T.; Araujo, Hygor N.; Gonçalves, Dawit A.; Guimarães, Dimitrius Santiago Passos Simões Fróes; Teodoro, Bruno G.; So, Kawai; Espreáfico, Enilza Maria ; Hirshman, Michael F.; Alberici, Luciane Carla ; Kettelhut, Isis do Carmo ; Goodyear, Laurie J.; Silveira, Leonardo R.
Source: Molecular Metabolism. Unidades: FMRP, FCFRP
Subjects: MICRORNAS, MÚSCULO ESQUELÉTICO, MITOCÔNDRIAS, REGULAÇÃO GÊNICA, PROLIFERAÇÃO CELULAR
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QUEIROZ, André L. et al. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, v. 51, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2021.101226. Acesso em: 02 jul. 2024.
APA
Queiroz, A. L., Lessard, S. J., Ouchida, A. T., Araujo, H. N., Gonçalves, D. A., Guimarães, D. S. P. S. F., et al. (2021). The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, 51. doi:10.1016/j.molmet.2021.101226
NLM
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Vancouver
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Artigo de periodico
Energy metabolism and redox state in brains of wistar audiogenic rats, a genetic model of epilepsy (2019)
Dechandt, Carlos Roberto Porto ; Ferrari, Gustavo Duarte ; Santos, Jonathas Rodrigo dos ; Oliveira, José Antonio Cortes de; Silva Júnior, Rui Milton Patrício da; Cunha, Alexandra Olimpio Siqueira ; Garcia-Cairasco, Norberto ; Alberici, Luciane Carla
Source: Frontiers in Neurology. Unidades: FMRP, FCFRP
Subjects: MITOCÔNDRIAS, EPILEPSIA, GENÉTICA ANIMAL
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DECHANDT, Carlos Roberto Porto et al. Energy metabolism and redox state in brains of wistar audiogenic rats, a genetic model of epilepsy. Frontiers in Neurology, v. 10, p. art. 1007 [15] , 2019Tradução . . Disponível em: https://doi.org/10.3389/fneur.2019.01007. Acesso em: 02 jul. 2024.
APA
Dechandt, C. R. P., Ferrari, G. D., Santos, J. R. dos, Oliveira, J. A. C. de, Silva Júnior, R. M. P. da, Cunha, A. O. S., et al. (2019). Energy metabolism and redox state in brains of wistar audiogenic rats, a genetic model of epilepsy. Frontiers in Neurology, 10, art. 1007 [15] . doi:10.3389/fneur.2019.01007
NLM
Dechandt CRP, Ferrari GD, Santos JR dos, Oliveira JAC de, Silva Júnior RMP da, Cunha AOS, Garcia-Cairasco N, Alberici LC. Energy metabolism and redox state in brains of wistar audiogenic rats, a genetic model of epilepsy [Internet]. Frontiers in Neurology. 2019 ; 10 art. 1007 [15] .[citado 2024 jul. 02 ] Available from: https://doi.org/10.3389/fneur.2019.01007
Vancouver
Dechandt CRP, Ferrari GD, Santos JR dos, Oliveira JAC de, Silva Júnior RMP da, Cunha AOS, Garcia-Cairasco N, Alberici LC. Energy metabolism and redox state in brains of wistar audiogenic rats, a genetic model of epilepsy [Internet]. Frontiers in Neurology. 2019 ; 10 art. 1007 [15] .[citado 2024 jul. 02 ] Available from: https://doi.org/10.3389/fneur.2019.01007
Artigo de periodico
Photobiomodulation effect on the proliferation of adipose tissue mesenchymal stem cells (2019)
Andrade, Ana Laura Martins de; Luna, Genoveva Flores; Brassolatti, Patrícia; Leite, Marcel Nani ; Parisi, Julia Risso; Leal, Ângela Merice de Oliveira; Frade, Marco Andrey Cipriani ; Anibal, Fernanda de Freitas; Parizotto, Nivaldo Antonio
Source: Lasers in Medical Science. Unidade: FMRP
Subjects: CÉLULAS-TRONCO, TECIDO ADIPOSO, PROLIFERAÇÃO CELULAR, MITOCÔNDRIAS, SOBREVIVÊNCIA CELULAR
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ANDRADE, Ana Laura Martins de et al. Photobiomodulation effect on the proliferation of adipose tissue mesenchymal stem cells. Lasers in Medical Science, v. 34, n. 4, p. 677-683, 2019Tradução . . Disponível em: https://doi.org/10.1007/s10103-018-2642-2. Acesso em: 02 jul. 2024.
APA
Andrade, A. L. M. de, Luna, G. F., Brassolatti, P., Leite, M. N., Parisi, J. R., Leal, Â. M. de O., et al. (2019). Photobiomodulation effect on the proliferation of adipose tissue mesenchymal stem cells. Lasers in Medical Science, 34( 4), 677-683. doi:10.1007/s10103-018-2642-2
NLM
Andrade ALM de, Luna GF, Brassolatti P, Leite MN, Parisi JR, Leal ÂM de O, Frade MAC, Anibal F de F, Parizotto NA. Photobiomodulation effect on the proliferation of adipose tissue mesenchymal stem cells [Internet]. Lasers in Medical Science. 2019 ; 34( 4): 677-683.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10103-018-2642-2
Vancouver
Andrade ALM de, Luna GF, Brassolatti P, Leite MN, Parisi JR, Leal ÂM de O, Frade MAC, Anibal F de F, Parizotto NA. Photobiomodulation effect on the proliferation of adipose tissue mesenchymal stem cells [Internet]. Lasers in Medical Science. 2019 ; 34( 4): 677-683.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10103-018-2642-2
Artigo de periodico
Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma (2018)
Goto, Renata Nishida; Sobral, Lays Martin ; Sousa, Lucas Oliveira ; Garcia, Cristiana Bernadelli; Lopes, Norberto Peporine ; Marín-Prida, Javier; Ochoa-Rodríguez, Estael; Verdecia-Reyes, Yamila; Pardo-Andreu, Gilberto Lázaro; Curti, Carlos ; Leopoldino, Andréia Machado
Source: European Journal of Pharmacology. Unidade: FCFRP
Subjects: ANTINEOPLÁSICOS, APOPTOSE, NEOPLASIAS DE CABEÇA E PESCOÇO, MITOCÔNDRIAS, NEOVASCULARIZAÇÃO PATOLÓGICA
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GOTO, Renata Nishida et al. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, v. 819, p. 198-206, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.12.009. Acesso em: 02 jul. 2024.
APA
Goto, R. N., Sobral, L. M., Sousa, L. O., Garcia, C. B., Lopes, N. P., Marín-Prida, J., et al. (2018). Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, 819, 198-206. doi:10.1016/j.ejphar.2017.12.009
NLM
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Vancouver
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Artigo de periodico
Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma (2018)
Araujo, Luiza Ferreira de ; Siena, Ádamo Davi Diogenes; Plaça, Jessica Rodrigues ; Brotto, Danielle Barbosa ; Barros, Isabela Ichihara de ; Muys, Bruna Rodrigues; Biagi Jr, Carlos Alberto Oliveira de ; Peronni, Kamila Chagas; Sousa, Josane de Freitas; Molfetta, Greice Andreotti de ; West, L. C.; West, A. P.; Leopoldino, Andréia Machado ; Espreáfico, Enilza Maria ; Silva Júnior, Wilson Araújo da
Source: Scientific Reports. Unidades: FCFRP, FMRP
Subjects: MELANOMA, MITOCÔNDRIAS
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ARAUJO, Luiza Ferreira de et al. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-31170-6. Acesso em: 02 jul. 2024.
APA
Araujo, L. F. de, Siena, Á. D. D., Plaça, J. R., Brotto, D. B., Barros, I. I. de, Muys, B. R., et al. (2018). Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma. Scientific Reports, 8. doi:10.1038/s41598-018-31170-6
NLM
Araujo LF de, Siena ÁDD, Plaça JR, Brotto DB, Barros II de, Muys BR, Biagi Jr CAO de, Peronni KC, Sousa J de F, Molfetta GA de, West LC, West AP, Leopoldino AM, Espreáfico EM, Silva Júnior WA da. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma [Internet]. Scientific Reports. 2018 ; 8[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/s41598-018-31170-6
Vancouver
Araujo LF de, Siena ÁDD, Plaça JR, Brotto DB, Barros II de, Muys BR, Biagi Jr CAO de, Peronni KC, Sousa J de F, Molfetta GA de, West LC, West AP, Leopoldino AM, Espreáfico EM, Silva Júnior WA da. Mitochondrial transcription factor A (TFAM) shapes metabolic and invasion gene signatures in melanoma [Internet]. Scientific Reports. 2018 ; 8[citado 2024 jul. 02 ] Available from: https://doi.org/10.1038/s41598-018-31170-6
Artigo de periodico
Mitochondrial damage and apoptosis: key features in BDE-153-induced hepatotoxicity (2018)
Pereira, Lílian Cristina ; Miranda, Luiz Felipe Cabral; Bernardes, Mariana Furio Franco ; Tasso, Maria Júlia ; Duarte, Filipe Valente; Varela, Ana Teresa Inácio; Rolo, Anabela Pinto; Palmeira, Carlos Manuel Marques; Dorta, Daniel Junqueira
Source: Chemico-Biological Interactions. Unidades: FFCLRP, FCFRP
Subjects: APOPTOSE, MITOCÔNDRIAS, TOXICOLOGIA AMBIENTAL, CÉLULAS MORTAS
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PEREIRA, Lílian Cristina et al. Mitochondrial damage and apoptosis: key features in BDE-153-induced hepatotoxicity. Chemico-Biological Interactions, v. 291, p. 192-201, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2018.06.021. Acesso em: 02 jul. 2024.
APA
Pereira, L. C., Miranda, L. F. C., Bernardes, M. F. F., Tasso, M. J., Duarte, F. V., Varela, A. T. I., et al. (2018). Mitochondrial damage and apoptosis: key features in BDE-153-induced hepatotoxicity. Chemico-Biological Interactions, 291, 192-201. doi:10.1016/j.cbi.2018.06.021
NLM
Pereira LC, Miranda LFC, Bernardes MFF, Tasso MJ, Duarte FV, Varela ATI, Rolo AP, Palmeira CMM, Dorta DJ. Mitochondrial damage and apoptosis: key features in BDE-153-induced hepatotoxicity [Internet]. Chemico-Biological Interactions. 2018 ; 291 192-201.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cbi.2018.06.021
Vancouver
Pereira LC, Miranda LFC, Bernardes MFF, Tasso MJ, Duarte FV, Varela ATI, Rolo AP, Palmeira CMM, Dorta DJ. Mitochondrial damage and apoptosis: key features in BDE-153-induced hepatotoxicity [Internet]. Chemico-Biological Interactions. 2018 ; 291 192-201.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cbi.2018.06.021
Artigo de periodico
Novel insights into mitochondrial molecular targets of iron-induced neurodegeneration: reversal by cannabidiol (2018)
Silva, Vanessa Kappel da; Freitas, Betânia Souza de; Dornelles, Victória Campos; Kist, Luiza Wilges; Bogo, Maurício Reis; Silva, Milena Carvalho; Streck, Emílio Luiz; Hallak, Jaime Eduardo Cecílio; Zuardi, Antônio Waldo; Crippa, José Alexandre de Souza; Schröder, Nadja
Source: Brain Research Bulletin. Unidade: FMRP
Subjects: CANABINOIDES, MITOCÔNDRIAS, DNA
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SILVA, Vanessa Kappel da et al. Novel insights into mitochondrial molecular targets of iron-induced neurodegeneration: reversal by cannabidiol. Brain Research Bulletin, v. 139, p. 1-8, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2018.01.014. Acesso em: 02 jul. 2024.
APA
Silva, V. K. da, Freitas, B. S. de, Dornelles, V. C., Kist, L. W., Bogo, M. R., Silva, M. C., et al. (2018). Novel insights into mitochondrial molecular targets of iron-induced neurodegeneration: reversal by cannabidiol. Brain Research Bulletin, 139, 1-8. doi:10.1016/j.brainresbull.2018.01.014
NLM
Silva VK da, Freitas BS de, Dornelles VC, Kist LW, Bogo MR, Silva MC, Streck EL, Hallak JEC, Zuardi AW, Crippa JA de S, Schröder N. Novel insights into mitochondrial molecular targets of iron-induced neurodegeneration: reversal by cannabidiol [Internet]. Brain Research Bulletin. 2018 ; 139 1-8.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.brainresbull.2018.01.014
Vancouver
Silva VK da, Freitas BS de, Dornelles VC, Kist LW, Bogo MR, Silva MC, Streck EL, Hallak JEC, Zuardi AW, Crippa JA de S, Schröder N. Novel insights into mitochondrial molecular targets of iron-induced neurodegeneration: reversal by cannabidiol [Internet]. Brain Research Bulletin. 2018 ; 139 1-8.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.brainresbull.2018.01.014
Artigo de periodico
Heterologous expression of mitochondrial nicotinamide adenine dinucleotide transporter (Ndt1) from Aspergillus fumigatus rescues impaired growth in Δndt1Δndt2 Saccharomyces cerevisiae strain (2017)
Balico, Laís de Lourdes de Lima; Santos, Emerson de Souza; Suzuki-Hatano, Silveli; Sousa, Lucas Oliveira; Azzolini, Ana Elisa Caleiro Seixas; Lucisano-Valim, Yara Maria; Dinamarco, Taisa Magnani; Kannen, Vinicius; Uyemura, Sérgio Akira
Source: Journal of Bioenergetics and Biomembranes. Unidades: FCFRP, FFCLRP
Subjects: MITOCÔNDRIAS, EXPRESSÃO GÊNICA, CÉLULAS, ASPERGILLUS
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ABNT
BALICO, Laís de Lourdes de Lima et al. Heterologous expression of mitochondrial nicotinamide adenine dinucleotide transporter (Ndt1) from Aspergillus fumigatus rescues impaired growth in Δndt1Δndt2 Saccharomyces cerevisiae strain. Journal of Bioenergetics and Biomembranes, v. 49, n. 6, p. 423-435, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10863-017-9732-x. Acesso em: 02 jul. 2024.
APA
Balico, L. de L. de L., Santos, E. de S., Suzuki-Hatano, S., Sousa, L. O., Azzolini, A. E. C. S., Lucisano-Valim, Y. M., et al. (2017). Heterologous expression of mitochondrial nicotinamide adenine dinucleotide transporter (Ndt1) from Aspergillus fumigatus rescues impaired growth in Δndt1Δndt2 Saccharomyces cerevisiae strain. Journal of Bioenergetics and Biomembranes, 49( 6), 423-435. doi:10.1007/s10863-017-9732-x
NLM
Balico L de L de L, Santos E de S, Suzuki-Hatano S, Sousa LO, Azzolini AECS, Lucisano-Valim YM, Dinamarco TM, Kannen V, Uyemura SA. Heterologous expression of mitochondrial nicotinamide adenine dinucleotide transporter (Ndt1) from Aspergillus fumigatus rescues impaired growth in Δndt1Δndt2 Saccharomyces cerevisiae strain [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 6): 423-435.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10863-017-9732-x
Vancouver
Balico L de L de L, Santos E de S, Suzuki-Hatano S, Sousa LO, Azzolini AECS, Lucisano-Valim YM, Dinamarco TM, Kannen V, Uyemura SA. Heterologous expression of mitochondrial nicotinamide adenine dinucleotide transporter (Ndt1) from Aspergillus fumigatus rescues impaired growth in Δndt1Δndt2 Saccharomyces cerevisiae strain [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 6): 423-435.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1007/s10863-017-9732-x
Artigo de periodico
The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms (2017)
Prida, Javier Marín; Andreu, Gilberto L. Pardo; Rossignoli, Camila Pederiva; Durruthy, Michael González; Rodríguez, Estael Ochoa; Reyes, Yamila Verdecia; Acosta, Roberto Fernández; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Source: Toxicology in Vitro. Unidade: FCFRP
Subjects: NEOPLASIAS, CITOTOXICIDADE IMUNOLÓGICA, MITOCÔNDRIAS, FÁRMACOS
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ABNT
PRIDA, Javier Marín et al. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, v. 42, p. 21-30, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2017.03.011. Acesso em: 02 jul. 2024.
APA
Prida, J. M., Andreu, G. L. P., Rossignoli, C. P., Durruthy, M. G., Rodríguez, E. O., Reyes, Y. V., et al. (2017). The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms. Toxicology in Vitro, 42, 21-30. doi:10.1016/j.tiv.2017.03.011
NLM
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Vancouver
Prida JM, Andreu GLP, Rossignoli CP, Durruthy MG, Rodríguez EO, Reyes YV, Acosta RF, Uyemura SA, Alberici LC. The cytotoxic effects of VE-3N, a novel 1,4-dihydropyridine derivative, involve the mitochondrial bioenergetic disruption via uncoupling mechanisms [Internet]. Toxicology in Vitro. 2017 ; 42 21-30.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.tiv.2017.03.011
Artigo de periodico
Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle (2017)
Teodoro, Bruno G.; Sampaio, Igor H.; Bomfim, Lucas H. M.; Queiroz, André L.; Silveira, Leonardo R.; Souza, Anderson O.; Fernandes, Anna M. A. P.; Eberlin, Marcos N.; Huang, Tai-Yu; Zheng, Donghai; Neufer, P. Darrell; Cortright, Ronald N.; Alberici, Luciane Carla
Source: The Journal of Physiology. Unidade: FCFRP
Subjects: MÚSCULO ESQUELÉTICO, METABOLISMO DE GORDURA, MITOCÔNDRIAS, ÁCIDOS GRAXOS
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ABNT
TEODORO, Bruno G. et al. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle. The Journal of Physiology, v. 595, n. 3, p. 677-693, 2017Tradução . . Disponível em: https://doi.org/10.1113/jp272962. Acesso em: 02 jul. 2024.
APA
Teodoro, B. G., Sampaio, I. H., Bomfim, L. H. M., Queiroz, A. L., Silveira, L. R., Souza, A. O., et al. (2017). Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle. The Journal of Physiology, 595( 3), 677-693. doi:10.1113/jp272962
NLM
Teodoro BG, Sampaio IH, Bomfim LHM, Queiroz AL, Silveira LR, Souza AO, Fernandes AMAP, Eberlin MN, Huang T-Y, Zheng D, Neufer PD, Cortright RN, Alberici LC. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle [Internet]. The Journal of Physiology. 2017 ; 595( 3): 677-693.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/jp272962
Vancouver
Teodoro BG, Sampaio IH, Bomfim LHM, Queiroz AL, Silveira LR, Souza AO, Fernandes AMAP, Eberlin MN, Huang T-Y, Zheng D, Neufer PD, Cortright RN, Alberici LC. Long-chain acyl-CoA synthetase 6 regulates lipid synthesis and mitochondrial oxidative capacity in human and rat skeletal muscle [Internet]. The Journal of Physiology. 2017 ; 595( 3): 677-693.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1113/jp272962
Artigo de periodico
QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload (2017)
Durruthy, Michael González; Castro, Micheli; Nunes, Silvana Manske; Lima, Juliane Ventura; Alberici, Luciane Carla; Naal, Zeki; Sawazaki, David T. Atique; Curti, Carlos; Ruas, Caroline Pires; Gelesky, Marcos A.; Roy, Kunal; González-Díaz, Humberto; Monserrat, José M.
Source: Carbon. Unidade: FCFRP
Subjects: NANOTUBOS DE CARBONO, MITOCÔNDRIAS, CURVAS DE DOSE-RESPOSTA, TEORIA DE PERTURBAÇÃO PARA OPERADORES LINEARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DURRUTHY, Michael González et al. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, v. 115, p. 312-330, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.carbon.2017.01.002. Acesso em: 02 jul. 2024.
APA
Durruthy, M. G., Castro, M., Nunes, S. M., Lima, J. V., Alberici, L. C., Naal, Z., et al. (2017). QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, 115, 312-330. doi:10.1016/j.carbon.2017.01.002
NLM
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Vancouver
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Artigo de periodico
SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line (2017)
Sobral, Lays M.; Coletta, Ricardo D.; Alberici, Luciane Carla; Curti, Carlos; Leopoldino, Andréia Machado
Source: The FEBS Journal. Unidade: FCFRP
Subjects: TOXICOLOGIA, CÉLULAS, METABOLISMO, MITOCÔNDRIAS, RESPIRAÇÃO
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ABNT
SOBRAL, Lays M. et al. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, v. 284, n. 17, p. 2774-2785, 2017Tradução . . Disponível em: https://doi.org/10.1111/febs.14148. Acesso em: 02 jul. 2024.
APA
Sobral, L. M., Coletta, R. D., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2017). SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, 284( 17), 2774-2785. doi:10.1111/febs.14148
NLM
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/febs.14148
Vancouver
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1111/febs.14148
Artigo de periodico
Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants (2017)
González-Durruthy, Michael; Alberici, Luciane C.; Curti, Carlos; Naal, Zeki; Sawazaki, David Tatsuo A; Vázquez-Naya, José M.; González-Díaz, Humberto; Munteanu, Cristian R.
Source: Journal of Chemical Information and Modeling. Unidade: FCFRP
Subjects: ENTROPIA, REGRESSÃO LINEAR, MITOCÔNDRIAS, NANOTUBOS DE CARBONO, BIOMEDICINA, OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZÁLEZ-DURRUTHY, Michael et al. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, v. 57, n. 5, p. 1029-1044, 2017Tradução . . Disponível em: https://doi.org/10.1021/acs.jcim.6b00458. Acesso em: 02 jul. 2024.
APA
González-Durruthy, M., Alberici, L. C., Curti, C., Naal, Z., Sawazaki, D. T. A., Vázquez-Naya, J. M., et al. (2017). Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, 57( 5), 1029-1044. doi:10.1021/acs.jcim.6b00458
NLM
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Vancouver
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Artigo de periodico
The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice (2016)
Baraldi, Flávia G.; Vicentini, Tatiane M.; Teodoro, Bruno G.; Dalalio, Felipe M.; Dechandt, Carlos R. P.; Prado, Ieda M. R.; Curti, Carlos; Cardoso, Fernanda C.; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Source: The Journal of Nutritional Biochemistry. Unidade: FCFRP
Subjects: METABOLISMO, OBESIDADE, AZEITE, ÁCIDO LINOLEICO, ÁCIDOS GRAXOS OMEGA 6, MITOCÔNDRIAS
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ABNT
BARALDI, Flávia G. et al. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, v. 28, p. 147-154, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2015.10.008. Acesso em: 02 jul. 2024.
APA
Baraldi, F. G., Vicentini, T. M., Teodoro, B. G., Dalalio, F. M., Dechandt, C. R. P., Prado, I. M. R., et al. (2016). The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, 28, 147-154. doi:10.1016/j.jnutbio.2015.10.008
NLM
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Vancouver
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Artigo de periodico
Toxicity of brominated flame retardants, BDE-47 and BDE-99 stems from impaired mitochondrial bioenergetics (2015)
Pazin, Murilo; Pereira, Lilian Cristina; Dorta, Daniel Junqueira
Source: Toxicology Mechanisms and Methods. Unidade: FFCLRP
Subjects: BIOENERGÉTICA, MITOCÔNDRIAS, TOXICOLOGIA AMBIENTAL
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ABNT
PAZIN, Murilo e PEREIRA, Lilian Cristina e DORTA, Daniel Junqueira. Toxicity of brominated flame retardants, BDE-47 and BDE-99 stems from impaired mitochondrial bioenergetics. Toxicology Mechanisms and Methods, v. 25, n. 1, p. 34-41, 2015Tradução . . Disponível em: https://doi.org/10.3109/15376516.2014.974233. Acesso em: 02 jul. 2024.
APA
Pazin, M., Pereira, L. C., & Dorta, D. J. (2015). Toxicity of brominated flame retardants, BDE-47 and BDE-99 stems from impaired mitochondrial bioenergetics. Toxicology Mechanisms and Methods, 25( 1), 34-41. doi:10.3109/15376516.2014.974233
NLM
Pazin M, Pereira LC, Dorta DJ. Toxicity of brominated flame retardants, BDE-47 and BDE-99 stems from impaired mitochondrial bioenergetics [Internet]. Toxicology Mechanisms and Methods. 2015 ; 25( 1): 34-41.[citado 2024 jul. 02 ] Available from: https://doi.org/10.3109/15376516.2014.974233
Vancouver
Pazin M, Pereira LC, Dorta DJ. Toxicity of brominated flame retardants, BDE-47 and BDE-99 stems from impaired mitochondrial bioenergetics [Internet]. Toxicology Mechanisms and Methods. 2015 ; 25( 1): 34-41.[citado 2024 jul. 02 ] Available from: https://doi.org/10.3109/15376516.2014.974233
Artigo de periodico
SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario (2015)
Almeida, Luciana O; Goto, Renata N; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Curti, Carlos; Leopoldino, Andréia Machado
Source: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Subjects: MITOCÔNDRIAS, PROTEÍNAS, NEOPLASIAS, DOENÇA DE ALZHEIMER
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O et al. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, v. 458, n. 2, p. 300-306, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.01.104. Acesso em: 02 jul. 2024.
APA
Almeida, L. O., Goto, R. N., Cavalcanti Neto, M. P., Sousa, L. O., Curti, C., & Leopoldino, A. M. (2015). SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, 458( 2), 300-306. doi:10.1016/j.bbrc.2015.01.104
NLM
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Vancouver
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Artigo de periodico
The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling (2015)
Andreu, Gilberto L. Pardo; Reis, Felippe H. Z; Dalalio, Felipe M; Figueredo, Yanier Nuñez; Rubio, Osmany Cuesta; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Source: Chemico-Biological Interactions. Unidade: FCFRP
Subjects: PRÓPOLIS, MITOCÔNDRIAS, TESTES DE TOXICIDADE, TOXICOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREU, Gilberto L. Pardo et al. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, v. 228, p. 28-34, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.01.010. Acesso em: 02 jul. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z., Dalalio, F. M., Figueredo, Y. N., Rubio, O. C., Uyemura, S. A., et al. (2015). The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, 228, 28-34. doi:10.1016/j.cbi.2015.01.010
NLM
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Vancouver
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010

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