SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario (2015)
- Authors:
- USP affiliated authors: SOUSA, LUCAS OLIVEIRA - FCFRP ; CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.bbrc.2015.01.104
- Subjects: MITOCÔNDRIAS; PROTEÍNAS; NEOPLASIAS; DOENÇA DE ALZHEIMER
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2015
- Source:
- Título do periódico: Biochemical and Biophysical Research Communications
- ISSN: 0006-291X
- Volume/Número/Paginação/Ano: v. 458, n. 2, p. 300-306, 2015
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
ALMEIDA, Luciana O et al. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, v. 458, n. 2, p. 300-306, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.01.104. Acesso em: 30 set. 2024. -
APA
Almeida, L. O., Goto, R. N., Cavalcanti Neto, M. P., Sousa, L. O., Curti, C., & Leopoldino, A. M. (2015). SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, 458( 2), 300-306. doi:10.1016/j.bbrc.2015.01.104 -
NLM
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104 -
Vancouver
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104 - SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
Informações sobre o DOI: 10.1016/j.bbrc.2015.01.104 (Fonte: oaDOI API)
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