Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation (2013)
- Authors:
- USP affiliated authors: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- Subjects: NEOPLASIAS; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Publisher: AACR
- Publisher place: Washington
- Date published: 2013
- Source:
- Título: Proceedings
- Conference titles: Annual Meeting of American Association for Cancer Research
-
ABNT
ALMEIDA, Luciana et al. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. 2013, Anais.. Washington: AACR, 2013. . Acesso em: 04 ago. 2025. -
APA
Almeida, L., Tannous, M., Matsumoto, C., Sobral, L., Curti, C., & Leopoldino, A. M. (2013). Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. In Proceedings. Washington: AACR. -
NLM
Almeida L, Tannous M, Matsumoto C, Sobral L, Curti C, Leopoldino AM. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. Proceedings. 2013 ;[citado 2025 ago. 04 ] -
Vancouver
Almeida L, Tannous M, Matsumoto C, Sobral L, Curti C, Leopoldino AM. Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation. Proceedings. 2013 ;[citado 2025 ago. 04 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
- Potential modulation of PTEN phosphatase by PP2A and oxidative stress
- SET regulates levels of HnRNP K and its binding to nucleic acid in HNSCC
- The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
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