Potential modulation of PTEN phosphatase by PP2A and oxidative stress (2012)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: ESTRESSE OXIDATIVO; PROTEÍNAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher place: Foz do Iguaçu
- Date published: 2012
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
MATSUMOTO, C. S. et al. Potential modulation of PTEN phosphatase by PP2A and oxidative stress. 2012, Anais.. Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 25 abr. 2024. -
APA
Matsumoto, C. S., Curti, C., Squarize, C. H., & Leopoldino, A. M. (2012). Potential modulation of PTEN phosphatase by PP2A and oxidative stress. In . Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Matsumoto CS, Curti C, Squarize CH, Leopoldino AM. Potential modulation of PTEN phosphatase by PP2A and oxidative stress. 2012 ;[citado 2024 abr. 25 ] -
Vancouver
Matsumoto CS, Curti C, Squarize CH, Leopoldino AM. Potential modulation of PTEN phosphatase by PP2A and oxidative stress. 2012 ;[citado 2024 abr. 25 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
- SET regulates levels of HnRNP K and its binding to nucleic acid in HNSCC
- The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction
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