ReP USP - Resultado da busca

Artigo de periodico
The severity of COVID-19 affects the plasma soluble levels of the immune checkpoint HLA-G molecule (2022)
Cordeiro, Jéssica Fernanda Corrêa ; Fernandes, Talita Morais ; Toro, Diana Mota; Silva-Neto, Pedro V. da; Pimentel, Vinícius Eduardo; Pérez, Malena Martínez ; Carvalho, Jonatan Constança Silva de; Fraga-Silva, Thais Fernanda de Campos ; Oliveira, Camilla Narjara Simão; Argolo, Jamille Guedes Malta; Degiovani, Augusto Marcussi; Ostini, Fátima Magro; Puggina, Enrico Fuini ; Silva, João Santana da ; Santos, Isabel Kinney Ferreira de Miranda ; Bonato, Vânia Luiza Deperon ; Cardoso, Cristina Ribeiro de Barros ; Dias-Baruffi, Marcelo ; Faccioli, Lucia Helena ; Donadi, Eduardo Antônio ; Sorgi, Carlos Arterio ; Fernandes, Ana Paula Morais
Source: International Journal of Molecular Sciences. Unidades: FMRP, FCFRP, FFCLRP, EERP, RUSP, EE/EERP
Subjects: COVID-19, CITOCINAS, COMORBIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORDEIRO, Jéssica Fernanda Corrêa et al. The severity of COVID-19 affects the plasma soluble levels of the immune checkpoint HLA-G molecule. International Journal of Molecular Sciences, v. 23, n. 17, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23179736. Acesso em: 05 nov. 2024.
APA
Cordeiro, J. F. C., Fernandes, T. M., Toro, D. M., Silva-Neto, P. V. da, Pimentel, V. E., Pérez, M. M., et al. (2022). The severity of COVID-19 affects the plasma soluble levels of the immune checkpoint HLA-G molecule. International Journal of Molecular Sciences, 23( 17), 1-16. doi:10.3390/ijms23179736
NLM
Cordeiro JFC, Fernandes TM, Toro DM, Silva-Neto PV da, Pimentel VE, Pérez MM, Carvalho JCS de, Fraga-Silva TF de C, Oliveira CNS, Argolo JGM, Degiovani AM, Ostini FM, Puggina EF, Silva JS da, Santos IKF de M, Bonato VLD, Cardoso CR de B, Dias-Baruffi M, Faccioli LH, Donadi EA, Sorgi CA, Fernandes APM. The severity of COVID-19 affects the plasma soluble levels of the immune checkpoint HLA-G molecule [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 17): 1-16.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3390/ijms23179736
Vancouver
Cordeiro JFC, Fernandes TM, Toro DM, Silva-Neto PV da, Pimentel VE, Pérez MM, Carvalho JCS de, Fraga-Silva TF de C, Oliveira CNS, Argolo JGM, Degiovani AM, Ostini FM, Puggina EF, Silva JS da, Santos IKF de M, Bonato VLD, Cardoso CR de B, Dias-Baruffi M, Faccioli LH, Donadi EA, Sorgi CA, Fernandes APM. The severity of COVID-19 affects the plasma soluble levels of the immune checkpoint HLA-G molecule [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 17): 1-16.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3390/ijms23179736
Artigo de periodico
T lymphocyte exhaustion during human and experimental visceral leishmaniasis (2022)
Costa-Madeira, Juliana Cristina; Trindade, Gabrielly Bindo; Almeida, Paulo Henrique do Prado; Silva, João Santana da ; Carregaro, Vanessa
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: INFLAMAÇÃO, LINFÓCITOS T, SISTEMA IMUNE, IMUNOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA-MADEIRA, Juliana Cristina et al. T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, v. 13, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.835711. Acesso em: 05 nov. 2024.
APA
Costa-Madeira, J. C., Trindade, G. B., Almeida, P. H. do P., Silva, J. S. da, & Carregaro, V. (2022). T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, 13, 1-15. doi:10.3389/fimmu.2022.835711
NLM
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2022.835711
Vancouver
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2022.835711
Artigo de periodico
Use of N-acetylcysteine as treatment adjuvant regulates immune response in visceral leishmaniasis: Pilot clinical trial and in vitro experiments (2022)
Magalhães, Lucas Sousa; Melo, Enaldo Vieira; Damascena, Nayra Prata; Albuquerque, Adriana Cardoso Batista; Santos, Camilla Natália Oliveira; Rebouças, Mônica Cardozo; Bezerra, Mariana de Oliveira; Silva, Ricardo Louzada da; Oliveira, Fabricia Alvisi de; Santos, Priscila Lima; Silva, João Santana da ; Lipscomb, Michael Wheeler; Silva, Ângela Maria da; Jesus, Amélia Ribeiro de; Almeida, Roque Pacheco de
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: LEISHMANIOSE VISCERAL, QUIMIOTERAPIA ADJUVANTE, FARMACOTERAPIA, INTERLEUCINA 10, LEUCÓCITOS MONONUCLEARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGALHÃES, Lucas Sousa et al. Use of N-acetylcysteine as treatment adjuvant regulates immune response in visceral leishmaniasis: Pilot clinical trial and in vitro experiments. Frontiers in Cellular and Infection Microbiology, v. 12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2022.1045668. Acesso em: 05 nov. 2024.
APA
Magalhães, L. S., Melo, E. V., Damascena, N. P., Albuquerque, A. C. B., Santos, C. N. O., Rebouças, M. C., et al. (2022). Use of N-acetylcysteine as treatment adjuvant regulates immune response in visceral leishmaniasis: Pilot clinical trial and in vitro experiments. Frontiers in Cellular and Infection Microbiology, 12. doi:10.3389/fcimb.2022.1045668
NLM
Magalhães LS, Melo EV, Damascena NP, Albuquerque ACB, Santos CNO, Rebouças MC, Bezerra M de O, Silva RL da, Oliveira FA de, Santos PL, Silva JS da, Lipscomb MW, Silva ÂM da, Jesus AR de, Almeida RP de. Use of N-acetylcysteine as treatment adjuvant regulates immune response in visceral leishmaniasis: Pilot clinical trial and in vitro experiments [Internet]. Frontiers in Cellular and Infection Microbiology. 2022 ; 12[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2022.1045668
Vancouver
Magalhães LS, Melo EV, Damascena NP, Albuquerque ACB, Santos CNO, Rebouças MC, Bezerra M de O, Silva RL da, Oliveira FA de, Santos PL, Silva JS da, Lipscomb MW, Silva ÂM da, Jesus AR de, Almeida RP de. Use of N-acetylcysteine as treatment adjuvant regulates immune response in visceral leishmaniasis: Pilot clinical trial and in vitro experiments [Internet]. Frontiers in Cellular and Infection Microbiology. 2022 ; 12[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2022.1045668
Artigo de periodico
Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection (2022)
Maruyama, Sandra Regina; Fuzo, Carlos Alessandro ; Oliveira, Antonio Edson Rocha; Rogerio, Luana Aparecida; Takamiya, Nayore Tamie; Pessenda, Gabriela; Melo, Enaldo Vieira de; Silva, Ângela Maria da; Jesus, Amelia Ribeiro de; Carregaro, Vanessa ; Nakaya, Helder Takashi Imoto ; Almeida, Roque Pacheco de; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, FCF, FCFRP
Subjects: LEISHMANIOSE VISCERAL, LEISHMANIA, RNA, SEQUENCIAMENTO GENÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARUYAMA, Sandra Regina et al. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, v. 13, p. 1-18, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.784463. Acesso em: 05 nov. 2024.
APA
Maruyama, S. R., Fuzo, C. A., Oliveira, A. E. R., Rogerio, L. A., Takamiya, N. T., Pessenda, G., et al. (2022). Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, 13, 1-18. doi:10.3389/fimmu.2022.784463
NLM
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Vancouver
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Artigo de periodico
Parasitic load correlates with left ventricular dysfunction in patients with chronic Chagas cardiomyopathy (2021)
Oliveira, Maykon Tavares de; Schmidt, Andre; Silva, Maria Cláudia da; Donadi, Eduardo Antônio; Silva, João Santana da ; Marin Neto, José Antonio
Source: Frontiers in Cardiovascular Medicine. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, DOENÇA DE CHAGAS, MIOCARDIOPATIA CONGESTIVA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Maykon Tavares de et al. Parasitic load correlates with left ventricular dysfunction in patients with chronic Chagas cardiomyopathy. Frontiers in Cardiovascular Medicine, v. 8, p. 1-6, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcvm.2021.741347. Acesso em: 05 nov. 2024.
APA
Oliveira, M. T. de, Schmidt, A., Silva, M. C. da, Donadi, E. A., Silva, J. S. da, & Marin Neto, J. A. (2021). Parasitic load correlates with left ventricular dysfunction in patients with chronic Chagas cardiomyopathy. Frontiers in Cardiovascular Medicine, 8, 1-6. doi:10.3389/fcvm.2021.741347
NLM
Oliveira MT de, Schmidt A, Silva MC da, Donadi EA, Silva JS da, Marin Neto JA. Parasitic load correlates with left ventricular dysfunction in patients with chronic Chagas cardiomyopathy [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-6.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.741347
Vancouver
Oliveira MT de, Schmidt A, Silva MC da, Donadi EA, Silva JS da, Marin Neto JA. Parasitic load correlates with left ventricular dysfunction in patients with chronic Chagas cardiomyopathy [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-6.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.741347
Artigo de periodico
Intra-discrete typing unit TcV genetic variability of Trypanosoma cruzi in Chronic Chagas' disease bolivian immigrant patients in Barcelona, Spain (2021)
Oliveira, Maykon Tavares de; Sulleiro, Elena; Silva, Maria Cláudia da; Silgado, Aroa; Lana, Marta de; Silva, João Santana da ; Molina, Israel; Marin Neto, José Antonio
Source: Frontiers in Cardiovascular Medicine. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI, VARIAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Maykon Tavares de et al. Intra-discrete typing unit TcV genetic variability of Trypanosoma cruzi in Chronic Chagas' disease bolivian immigrant patients in Barcelona, Spain. Frontiers in Cardiovascular Medicine, v. 8, p. 1-9, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcvm.2021.665624. Acesso em: 05 nov. 2024.
APA
Oliveira, M. T. de, Sulleiro, E., Silva, M. C. da, Silgado, A., Lana, M. de, Silva, J. S. da, et al. (2021). Intra-discrete typing unit TcV genetic variability of Trypanosoma cruzi in Chronic Chagas' disease bolivian immigrant patients in Barcelona, Spain. Frontiers in Cardiovascular Medicine, 8, 1-9. doi:10.3389/fcvm.2021.665624
NLM
Oliveira MT de, Sulleiro E, Silva MC da, Silgado A, Lana M de, Silva JS da, Molina I, Marin Neto JA. Intra-discrete typing unit TcV genetic variability of Trypanosoma cruzi in Chronic Chagas' disease bolivian immigrant patients in Barcelona, Spain [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-9.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.665624
Vancouver
Oliveira MT de, Sulleiro E, Silva MC da, Silgado A, Lana M de, Silva JS da, Molina I, Marin Neto JA. Intra-discrete typing unit TcV genetic variability of Trypanosoma cruzi in Chronic Chagas' disease bolivian immigrant patients in Barcelona, Spain [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-9.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.665624
Artigo de periodico
Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth (2021)
Souza, Débora Maria Soares de; Silva, Maria Cláudia da; Farias, Silvia Elvira Barros; Menezes, Ana Paula de J.; Milanezi, Cristiane Maria; Lúcio, Karine de P.; Paiva, Nívia Carolina N.; Abreu, Paula Melo de; Costa, Daniela Caldeira; Pinto, Kelerson Mauro de Castro; Costa, Guilherme de Paula; Silva, João Santana da ; Talvani, André
Source: Frontiers in Cardiovascular Medicine. Unidades: EERP, FMRP
Subjects: INFLAMAÇÃO, TRYPANOSOMA CRUZI, ÁCIDOS GRAXOS, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Débora Maria Soares de et al. Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth. Frontiers in Cardiovascular Medicine, v. 8, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcvm.2021.667580. Acesso em: 05 nov. 2024.
APA
Souza, D. M. S. de, Silva, M. C. da, Farias, S. E. B., Menezes, A. P. de J., Milanezi, C. M., Lúcio, K. de P., et al. (2021). Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth. Frontiers in Cardiovascular Medicine, 8, 1-12. doi:10.3389/fcvm.2021.667580
NLM
Souza DMS de, Silva MC da, Farias SEB, Menezes AP de J, Milanezi CM, Lúcio K de P, Paiva NCN, Abreu PM de, Costa DC, Pinto KM de C, Costa G de P, Silva JS da, Talvani A. Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-12.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.667580
Vancouver
Souza DMS de, Silva MC da, Farias SEB, Menezes AP de J, Milanezi CM, Lúcio K de P, Paiva NCN, Abreu PM de, Costa DC, Pinto KM de C, Costa G de P, Silva JS da, Talvani A. Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth [Internet]. Frontiers in Cardiovascular Medicine. 2021 ; 8 1-12.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcvm.2021.667580
Artigo de periodico
The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway (2020)
Leite, Jefferson Antonio; Pessenda, Gabriela ; Gomes, Isabel Cristina Guerra ; Santana, Alynne Karen Mendonça de; Pereira, Camila André; Costa, Frederico Ribeiro Campos; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Faria, Ana Maria Caetano ; Almeida, Danilo Candido de ; Câmara, Niels Olsen Saraiva ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Cells. Unidades: FMRP, ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Jefferson Antonio et al. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, v. 9, n. 4, p. 21 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9040959. Acesso em: 05 nov. 2024.
APA
Leite, J. A., Pessenda, G., Gomes, I. C. G., Santana, A. K. M. de, Pereira, C. A., Costa, F. R. C., et al. (2020). The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, 9( 4), 21 . doi:10.3390/cells9040959
NLM
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3390/cells9040959
Vancouver
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3390/cells9040959
Artigo de periodico
Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication (2020)
Mota, Caroline M.; Lima Júnior, Djalma de Souza; França, Flávia Batista Ferreira; Torres, Jhoan David Aguillón; Barros, Patrício da Silva Cardoso; Santiago, Fernanda Maria; Silva, João Santana da ; Mineo, José Roberto; Zamboni, Dario Simões; Mineo, Tiago Wilson Patriarca
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: INTERLEUCINA 1, NEOSPORA CANINUM, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOTA, Caroline M. et al. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2020.00243. Acesso em: 05 nov. 2024.
APA
Mota, C. M., Lima Júnior, D. de S., França, F. B. F., Torres, J. D. A., Barros, P. da S. C., Santiago, F. M., et al. (2020). Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, 10. doi:10.3389/fcimb.2020.00243
NLM
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Vancouver
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Artigo de periodico
Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes (2020)
Pereira, Camila André; Carlos, Daniela; Ferreira, Nathanne dos Santos; Silva, Josiane F. da; Zanotto, Camila Ziliotto; Zamboni, Dario Simões ; Garcia, Valéria Duarte; Ventura, Dora Selma Fix ; Silva, João Santana da ; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidades: FMRP, IP
Subjects: INFLAMAÇÃO, DNA MITOCONDRIAL, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Camila André et al. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01557. Acesso em: 05 nov. 2024.
APA
Pereira, C. A., Carlos, D., Ferreira, N. dos S., Silva, J. F. da, Zanotto, C. Z., Zamboni, D. S., et al. (2020). Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01557
NLM
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Vancouver
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Artigo de periodico
NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? (2020)
Oliveira, Jefferson Elias ; Leite, Jefferson Antônio; Pereira, Ítalo Sousa; Guimarães, Jhefferson Barbosa; Manso, Gabriel Martins da Costa; Silva, João Santana da ; Passaglia, Rita de Cassia Aleixo Tostes ; Sartori, Daniela Carlos
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, DIABETES MELLITUS, HOMEOSTASE, INTESTINOS, BACTÉRIAS, RECEPTORES IMUNOLÓGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Jefferson Elias et al. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers?. Frontiers in Immunology, v. 11, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01810. Acesso em: 05 nov. 2024.
APA
Oliveira, J. E., Leite, J. A., Pereira, Í. S., Guimarães, J. B., Manso, G. M. da C., Silva, J. S. da, et al. (2020). NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? Frontiers in Immunology, 11, 1-9. doi:10.3389/fimmu.2020.01810
NLM
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Vancouver
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 05 nov. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis (2019)
Gupta, Gaurav; Santana, Alynne Karen Mendonça de; Gomes, Ciro Martins ; Turatti, Aline; Milanezi, Cristiane Maria; Bueno Filho, Roberto ; Fuzo, Carlos Alessandro ; Almeida, Roque Pacheco de ; Carregaro, Vanessa ; Roselino, Ana Maria Ferreira ; Silva, João Santana da
Source: Cellular Immunology. Unidades: FMRP, FCFRP
Subjects: LEISHMANIOSE CUTÂNEA, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUPTA, Gaurav et al. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, v. 341, p. [8] , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cellimm.2019.04.008. Acesso em: 05 nov. 2024.
APA
Gupta, G., Santana, A. K. M. de, Gomes, C. M., Turatti, A., Milanezi, C. M., Bueno Filho, R., et al. (2019). Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, 341, [8] . doi:10.1016/j.cellimm.2019.04.008
NLM
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Vancouver
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Artigo de periodico
NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection (2019)
Benevides, Luciana ; Saltarelli, Verônica Marcela; Pioto, Franciele ; Sacramento, Laís Amorim ; Dias, Murilo Solano; Rodríguez, Gretel Rodríguez ; Viola, João Paulo de Biaso ; Carregaro, Vanessa ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ESALQ
Subjects: INFLAMAÇÃO, LINFÓCITOS T, MONÓCITOS, TOXOPLASMA GONDII, TOXOPLASMOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BENEVIDES, Luciana et al. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, v. 10, p. [10] , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.02105. Acesso em: 05 nov. 2024.
APA
Benevides, L., Saltarelli, V. M., Pioto, F., Sacramento, L. A., Dias, M. S., Rodríguez, G. R., et al. (2019). NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, 10, [10] . doi:10.3389/fimmu.2019.02105
NLM
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2019.02105
Vancouver
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2019.02105
Artigo de periodico
CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development (2018)
Braga, Tárcio Teodoro; Costa, Matheus Correa; Silva, Reinaldo Correia; Cruz, Mário Costa; Hiyane, Meire Ioshie; Silva, João Santana da; Perez, Katia Regina; Cuccovia, Iolanda Midea ; Câmara, Niels Olsen Saraiva
Source: Inflammopharmacology. Unidades: ICB, FMRP, IQ
Subjects: MONÓCITOS, QUIMIOCINAS, INFLAMAÇÃO, TÉCNICAS DE DIAGNÓSTICO UROLÓGICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAGA, Tárcio Teodoro et al. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, v. 26, p. 403-411, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10787-017-0317-4. Acesso em: 05 nov. 2024.
APA
Braga, T. T., Costa, M. C., Silva, R. C., Cruz, M. C., Hiyane, M. I., Silva, J. S. da, et al. (2018). CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, 26, 403-411. doi:10.1007/s10787-017-0317-4
NLM
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Vancouver
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Artigo de periodico
Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection (2017)
Lima, Mikhael Haruo Fernandes de; Sacramento, Laís Amorim; Quirino, Gustavo Fernando da Silva; Ferreira, Marcela Davoli; Benevides, Luciana; Santana, Alynne Karen M.; Cunha, Fernando de Queiroz; Almeida, Roque P.; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: LEISHMANIOSE VISCERAL, INFLAMAÇÃO, ADENOSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Mikhael Haruo Fernandes de et al. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00815. Acesso em: 05 nov. 2024.
APA
Lima, M. H. F. de, Sacramento, L. A., Quirino, G. F. da S., Ferreira, M. D., Benevides, L., Santana, A. K. M., et al. (2017). Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00815
NLM
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00815
Vancouver
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00815
Artigo de periodico
Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis (2017)
Tristão, Fabrine Sales Massafera; Rocha, Fernanda Agostini; Carlos, Daniela; Carneiro, Natália Ketelut; Souza, Camila Oliveira Silva; Milanezi, Cristiane Maria; Silva, João Santana da
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: PARACOCCIDIOIDOMICOSE, QUIMIOCINAS, CITOCINAS, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine Sales Massafera et al. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00949. Acesso em: 05 nov. 2024.
APA
Tristão, F. S. M., Rocha, F. A., Carlos, D., Carneiro, N. K., Souza, C. O. S., Milanezi, C. M., & Silva, J. S. da. (2017). Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00949
NLM
Tristão FSM, Rocha FA, Carlos D, Carneiro NK, Souza COS, Milanezi CM, Silva JS da. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00949
Vancouver
Tristão FSM, Rocha FA, Carlos D, Carneiro NK, Souza COS, Milanezi CM, Silva JS da. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00949
Artigo de periodico
Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis (2017)
Souza, Maria C.; Fonseca, Denise Morais da ; Kanashiro, Alexandre; Benevides, Luciana; Medina, Tiago S.; Dias, Murilo S.; Andrade, Warrison A.; Bonfá, Giuliano; Silva, Marcondes A. B.; Gozzi, Aline; Borges, Marcos de Carvalho; Gazzinelli, Ricardo T.; Alves Filho, José Carlos Farias; Cunha, Fernando Queiróz; Silva, João Santana da
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, INTERFERONS, ÓXIDO NÍTRICO, SEPSE, TOXOPLASMOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Maria C. et al. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2017.00116. Acesso em: 05 nov. 2024.
APA
Souza, M. C., Fonseca, D. M. da, Kanashiro, A., Benevides, L., Medina, T. S., Dias, M. S., et al. (2017). Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis. Frontiers in Cellular and Infection Microbiology, 7. doi:10.3389/fcimb.2017.00116
NLM
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Vancouver
Souza MC, Fonseca DM da, Kanashiro A, Benevides L, Medina TS, Dias MS, Andrade WA, Bonfá G, Silva MAB, Gozzi A, Borges M de C, Gazzinelli RT, Alves Filho JCF, Cunha FQ, Silva JS da. Chronic toxoplasma gondii infection exacerbates secondary polymicrobial sepsis [Internet]. Frontiers in Cellular and Infection Microbiology. 2017 ;7[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2017.00116
Artigo de periodico
Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
Sacramento, Laís A.; Costa, Jéssica L. da; Lima, Mikhael H. F. de; Sampaio, Pedro A.; Almeida, Roque P.; Cunha, Fernando Queiróz; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Microbiology. Unidade: FMRP
Subjects: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 05 nov. 2024.
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Artigo de periodico
Immune checkpoints in leprosy: immunotherapy as a feasible approach to control disease progression (2017)
Lima, Hayana Ramos; Gasparoto, Thaís Helena; Malaspina, Tatiana Salles de Souza; Marques, Vinícius Rizzo; Vicente, Marina Jurado; Marcos, Elaine Camarinha; Souza, Fabiana Corvolo; Nogueira, Maria Renata Sales; Barreto, Jaison Antônio; Garlet, Gustavo Pompermaier; Silva, João Santana da; Brito-de-Souza, Vânia Nieto; Campanelli, Ana Paula
Source: Frontiers in Immunology. Unidades: FOB, FMRP
Subjects: IMUNOTERAPIA, HANSENÍASE, PROTEÍNAS DA MEMBRANA, LINFÓCITOS T
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Hayana Ramos et al. Immune checkpoints in leprosy: immunotherapy as a feasible approach to control disease progression. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01724. Acesso em: 05 nov. 2024.
APA
Lima, H. R., Gasparoto, T. H., Malaspina, T. S. de S., Marques, V. R., Vicente, M. J., Marcos, E. C., et al. (2017). Immune checkpoints in leprosy: immunotherapy as a feasible approach to control disease progression. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.01724
NLM
Lima HR, Gasparoto TH, Malaspina TS de S, Marques VR, Vicente MJ, Marcos EC, Souza FC, Nogueira MRS, Barreto JA, Garlet GP, Silva JS da, Brito-de-Souza VN, Campanelli AP. Immune checkpoints in leprosy: immunotherapy as a feasible approach to control disease progression [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.01724
Vancouver
Lima HR, Gasparoto TH, Malaspina TS de S, Marques VR, Vicente MJ, Marcos EC, Souza FC, Nogueira MRS, Barreto JA, Garlet GP, Silva JS da, Brito-de-Souza VN, Campanelli AP. Immune checkpoints in leprosy: immunotherapy as a feasible approach to control disease progression [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.01724

USP Schools

ReP

Filtros

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation

Countries of institutions of collaboration