T lymphocyte exhaustion during human and experimental visceral leishmaniasis (2022)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; PEREIRA, VANESSA CARREGARO - FMRP ; MADEIRA, JULIANA CRISTINA COSTA - FMRP ; TRINDADE, GABRIÉLLY BINDO - FMRP ; ALMEIDA, PAULO HENRIQUE DO PRADO - FMRP
- Unidade: FMRP
- DOI: 10.3389/fimmu.2022.835711
- Subjects: INFLAMAÇÃO; LINFÓCITOS T; SISTEMA IMUNE; IMUNOTERAPIA
- Keywords: Visceral leishmanaisis; T cell exhaustion; Th subsets; Inflammation; Inhibitory receptor
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 13, art. 835711, p. 1-15, 2022
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
COSTA-MADEIRA, Juliana Cristina et al. T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, v. 13, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.835711. Acesso em: 23 jul. 2024. -
APA
Costa-Madeira, J. C., Trindade, G. B., Almeida, P. H. do P., Silva, J. S. da, & Carregaro, V. (2022). T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, 13, 1-15. doi:10.3389/fimmu.2022.835711 -
NLM
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 jul. 23 ] Available from: https://doi.org/10.3389/fimmu.2022.835711 -
Vancouver
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 jul. 23 ] Available from: https://doi.org/10.3389/fimmu.2022.835711 - Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response
- T1/ST2 deficient mice display protection against Leishmania infantum experimental infection
- CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection
- Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection
- T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses
- Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection
- Inflammasome-derived IL-1'beta' production induces nitric oxide-mediated resistance to Leishmania
- Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection
- Dual effect of Lutzomyia longipalpis saliva on Leishmania braziliensis infection is mediated by distinct saliva-induced
- Toll-like receptor 9 signaling in dendritic cells regulates neutrophil recruitment to inflammatory foci following Leishmania infantum infection
Informações sobre o DOI: 10.3389/fimmu.2022.835711 (Fonte: oaDOI API)
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