CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection (2016)
- Authors:
- USP affiliated authors: PEREIRA, VANESSA CARREGARO - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.1189/jlb.4a0715-288r
- Subjects: LEISHMANIOSE CUTÂNEA; LEISHMANIA BRASILIENSIS; QUIMIOCINAS; SISTEMA IMUNE; TERAPÊUTICA MÉDICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Leukocyte Biology
- ISSN: 0741-5400
- Volume/Número/Paginação/Ano: v. 100, n. 2, p. 423-432, 2016
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
COSTA, Diego Luís et al. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection. Journal of Leukocyte Biology, v. 100, n. 2, p. 423-432, 2016Tradução . . Disponível em: https://doi.org/10.1189/jlb.4a0715-288r. Acesso em: 27 dez. 2025. -
APA
Costa, D. L., Lima Júnior, D. de S., Nascimento, M. S. L., Sacramento, L. A., Almeida, R. P. de, Carregaro, V., & Silva, J. S. da. (2016). CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection. Journal of Leukocyte Biology, 100( 2), 423-432. doi:10.1189/jlb.4a0715-288r -
NLM
Costa DL, Lima Júnior D de S, Nascimento MSL, Sacramento LA, Almeida RP de, Carregaro V, Silva JS da. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection [Internet]. Journal of Leukocyte Biology. 2016 ; 100( 2): 423-432.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1189/jlb.4a0715-288r -
Vancouver
Costa DL, Lima Júnior D de S, Nascimento MSL, Sacramento LA, Almeida RP de, Carregaro V, Silva JS da. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection [Internet]. Journal of Leukocyte Biology. 2016 ; 100( 2): 423-432.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1189/jlb.4a0715-288r - Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection
- T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses
- Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response
- T1/ST2 deficient mice display protection against Leishmania infantum experimental infection
- Inflammasome-derived IL-1'beta' production induces nitric oxide-mediated resistance to Leishmania
- Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection
- Blimp-1 signaling pathways in T lymphocytes is essential to control the Trypanosoma cruzi infection-induced inflammation
- Dual effect of Lutzomyia longipalpis saliva on Leishmania braziliensis infection is mediated by distinct saliva-induced
- Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection
- Association of IL-9, IL-10, and IL-17 cytokines with hepatic fibrosis in human Schistosoma mansoni infection
Informações sobre o DOI: 10.1189/jlb.4a0715-288r (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
