Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection (2017)
- Authors:
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; SILVA, JOÃO SANTANA DA - FMRP ; PEREIRA, VANESSA CARREGARO - FMRP
- Unidade: FMRP
- DOI: 10.3389/fimmu.2017.00815
- Subjects: LEISHMANIOSE VISCERAL; INFLAMAÇÃO; ADENOSINA
- Keywords: Visceral leishmaniasis,; Adenosine; Immunoregulation; A2AR signaling; Inflammation
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 8, art. 815, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
LIMA, Mikhael Haruo Fernandes de et al. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00815. Acesso em: 28 dez. 2025. -
APA
Lima, M. H. F. de, Sacramento, L. A., Quirino, G. F. da S., Ferreira, M. D., Benevides, L., Santana, A. K. M., et al. (2017). Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00815 -
NLM
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2025 dez. 28 ] Available from: https://doi.org/10.3389/fimmu.2017.00815 -
Vancouver
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2025 dez. 28 ] Available from: https://doi.org/10.3389/fimmu.2017.00815 - Dual effect of Lutzomyia longipalpis saliva on Leishmania braziliensis infection is mediated by distinct saliva-induced
- Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection
- Toll-like receptor 9 signaling in dendritic cells regulates neutrophil recruitment to inflammatory foci following Leishmania infantum infection
- Protective role of 5-lipoxigenase during Leishmania infantum infection is associated with TH17 subset
- Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection
- T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses
- CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection
- Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response
- T1/ST2 deficient mice display protection against Leishmania infantum experimental infection
- Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment
Informações sobre o DOI: 10.3389/fimmu.2017.00815 (Fonte: oaDOI API)
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