Chronic nuclear factor-kappa B inhibition prevents experimental aging nephropathy (2025)
- Authors:
- USP affiliated authors: CÂMARA, NIELS OLSEN SARAIVA - ICB ; ZATZ, ROBERTO - FM ; FUJIHARA, CLARICE KAZUE - FM ; MALHEIROS, DENISE MARIA AVANCINI COSTA - FM ; TESSARO, HELENA MENDONÇA - FM ; ZAMBOM, FERNANDA FLORENCIA FREGNAN - FM ; FORESTO NETO, ORESTES - ICB
- Unidades: ICB; FM
- DOI: 10.1159/000540761
- Subjects: IMUNOLOGIA; NEFROPATIAS; MACRÓFAGOS; FÁRMACOS (SISTEMA CARDIOVASCULAR); CÉLULAS-TRONCO; ENVELHECIMENTO CELULAR; ANGIOTENSINA II; RATOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Gerontology
- ISSN: 1423-0003
- Volume/Número/Paginação/Ano: v. 71, p. 112-124, 2025
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
TESSARO, Helena Mendonça et al. Chronic nuclear factor-kappa B inhibition prevents experimental aging nephropathy. Gerontology, v. 71, p. 112-124, 2025Tradução . . Disponível em: https://doi.org/10.1159/000540761. Acesso em: 10 fev. 2026. -
APA
Tessaro, H. M., Zambom, F. F. F., Foresto Neto, O., Ribeiro, S. C. F., Sena, C. R. de, Malheiros, D. M. A. C., et al. (2025). Chronic nuclear factor-kappa B inhibition prevents experimental aging nephropathy. Gerontology, 71, 112-124. doi:10.1159/000540761 -
NLM
Tessaro HM, Zambom FFF, Foresto Neto O, Ribeiro SCF, Sena CR de, Malheiros DMAC, Fujihara CK, Câmara NOS, Zatz R. Chronic nuclear factor-kappa B inhibition prevents experimental aging nephropathy [Internet]. Gerontology. 2025 ; 71 112-124.[citado 2026 fev. 10 ] Available from: https://doi.org/10.1159/000540761 -
Vancouver
Tessaro HM, Zambom FFF, Foresto Neto O, Ribeiro SCF, Sena CR de, Malheiros DMAC, Fujihara CK, Câmara NOS, Zatz R. Chronic nuclear factor-kappa B inhibition prevents experimental aging nephropathy [Internet]. Gerontology. 2025 ; 71 112-124.[citado 2026 fev. 10 ] Available from: https://doi.org/10.1159/000540761 - Pathogenic role of innate immunity in a model of chronic NO inhibition associated with salt overload
- TLR2 and TLR4 play opposite role in autophagy associated with cisplatin-induced acute kidney injury
- Simultaneous activation of innate and adaptive immunity participates in the development of renal injury in a model of heavy proteinuria
- NLRP3 inflammasome inhibition ameliorates tubulointerstitial injury in the remnant kidney model
- NF-kappa B system is chronically activated and promotes glomerular injury in experimental type 1 diabetic kidney disease
- NF-κB blockade during short-term l-NAME and salt overload strongly attenuates the late development of chronic kidney disease
- Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis
- Hypoxia enhances ILC3 responses through HIF-1α-dependent mechanism
- The role of uric acid in inflammasome-mediated kidney injury
- An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney
Informações sobre o DOI: 10.1159/000540761 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
