Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer (2023)
- Authors:
- USP affiliated authors: SAITO, KELLY CRISTINA - ICB ; KIMURA, EDNA TERUKO - ICB ; MELLO, DIEGO CLARO DE - ICB ; FUZIWARA, CESAR SEIGI - ICB ; Cristovão, Marcella Maringolo - ICB
- Unidade: ICB
- DOI: 10.3390/ijms24097872
- Subjects: BIOLOGIA CELULAR; ANTINEOPLÁSICOS; CÉLULAS CULTIVADAS DE TUMOR; GLÂNDULA TIREOIDE; NEOPLASIAS DA TIREOIDE; PROLIFERAÇÃO CELULAR
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: International Journal of Molecular Sciences
- ISSN: 1422-0067
- Volume/Número/Paginação/Ano: v. 24, n. 9, art. 7872, p. 1-22, 2023
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
MELLO, Diego Claro de et al. Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer. International Journal of Molecular Sciences, v. 24, n. 9, p. 1-22, 2023Tradução . . Disponível em: https://doi.org/10.3390/ijms24097872. Acesso em: 06 out. 2024. -
APA
Mello, D. C. de, Saito, K. C., Cristovão, M. M., Kimura, E. T., & Fuziwara, C. S. (2023). Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer. International Journal of Molecular Sciences, 24( 9), 1-22. doi:10.3390/ijms24097872 -
NLM
Mello DC de, Saito KC, Cristovão MM, Kimura ET, Fuziwara CS. Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 9): 1-22.[citado 2024 out. 06 ] Available from: https://doi.org/10.3390/ijms24097872 -
Vancouver
Mello DC de, Saito KC, Cristovão MM, Kimura ET, Fuziwara CS. Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 9): 1-22.[citado 2024 out. 06 ] Available from: https://doi.org/10.3390/ijms24097872 - Thyroid follicular cell loss of differentiation induced by MicroRNA miR-17-92 cluster is attenuated by CRISPR/Cas9n gene silencing in anaplastic thyroid cancer
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- Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we?
- The highly expressed FAM83F Protein in papillary thyroid cancer exerts a pro-oncogenic role in thyroid follicular cells
- O cluster de microRNAs miR-17-92 e seus alvos na oncogênese tiroidiana: influência de BRAFT1799A e de iodo
- Genetic mutations and variants in the susceptibility of familial non-medullary thyroid cancer
- How does microRNA modulate Wnt/β-catenin signaling in thyroid oncogenesis?
- Expression of notch and nodal embryonic components in BRAF- and RAS-like thyroid cancer cell lines
- Targeting the highly expressed microRNA miR-146b with CRISPR/Cas9n gene editing system in thyroid cancer
- Influência do MicroRNA let-7 e miR-17-92 como oncomiRs no câncer.
Informações sobre o DOI: 10.3390/ijms24097872 (Fonte: oaDOI API)
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