Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we? (2022)
- Authors:
- USP affiliated authors: KIMURA, EDNA TERUKO - ICB ; FUZIWARA, CESAR SEIGI - ICB ; MELLO, DIEGO CLARO DE - ICB
- Unidade: ICB
- DOI: 10.3390/cancers14030844
- Subjects: BIOLOGIA CELULAR; NEOPLASIAS DA TIREOIDE; CÉLULAS CULTIVADAS DE TUMOR; GENES; GENES; GENOMAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
FUZIWARA, Cesar Seigi e MELLO, Diego Claro de e KIMURA, Edna Teruko. Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we?. Cancers, v. 14, n. 3, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.3390/cancers14030844. Acesso em: 28 dez. 2025. -
APA
Fuziwara, C. S., Mello, D. C. de, & Kimura, E. T. (2022). Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we? Cancers, 14( 3), 1-21. doi:10.3390/cancers14030844 -
NLM
Fuziwara CS, Mello DC de, Kimura ET. Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we? [Internet]. Cancers. 2022 ; 14( 3): 1-21.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3390/cancers14030844 -
Vancouver
Fuziwara CS, Mello DC de, Kimura ET. Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we? [Internet]. Cancers. 2022 ; 14( 3): 1-21.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3390/cancers14030844 - Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer
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- Genetic mutations and variants in the susceptibility of familial non-medullary thyroid cancer
- Modulating gene expression as a strategy to investigate thyroid cancer biology
- Thyroid follicular cell loss of differentiation induced by MicroRNA miR-17-92 cluster is attenuated by CRISPR/Cas9n gene silencing in anaplastic thyroid cancer
- Interplay of TGFβ signaling and microRNA in thyroid cell loss of differentiation and cancer progression
- O cluster de microRNAs miR-17-92 e seus alvos na oncogênese tiroidiana: influência de BRAFT1799A e de iodo
- The highly expressed FAM83F Protein in papillary thyroid cancer exerts a pro-oncogenic role in thyroid follicular cells
- Expression of notch and nodal embryonic components in BRAF- and RAS-like thyroid cancer cell lines
- Influência do MicroRNA let-7 e miR-17-92 como oncomiRs no câncer.
Informações sobre o DOI: 10.3390/cancers14030844 (Fonte: oaDOI API)
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