Dual effect of EZH2 gene editing with CRISPR/Cas9 in lung cancer (2026)
- Authors:
- USP affiliated authors: SAITO, KELLY CRISTINA - ICB ; KIMURA, EDNA TERUKO - ICB ; MENEZES, JOICE MORAES - ICB ; MELLO, DIEGO CLARO DE - ICB ; FUZIWARA, CESAR SEIGI - ICB
- Unidade: ICB
- DOI: 10.3390/biology15030251
- Subjects: BIOLOGIA CELULAR; NEOPLASIAS EPITELIAIS E GLANDULARES; ADENOCARCINOMA; METÁSTASE NEOPLÁSICA; EXPRESSÃO GÊNICA; CAMUNDONGOS; MODELOS ANIMAIS DE DOENÇAS; CÉLULAS CULTIVADAS DE TUMOR; EPIGÊNESE GENÉTICA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
MENEZES, Joice Moraes et al. Dual effect of EZH2 gene editing with CRISPR/Cas9 in lung cancer. Biology, v. 15, 2026Tradução . . Disponível em: https://doi.org/10.3390/biology15030251. Acesso em: 02 abr. 2026. -
APA
Menezes, J. M., Mello, D. C. de, Saito, K. C., Kimura, E. T., & Fuziwara, C. S. (2026). Dual effect of EZH2 gene editing with CRISPR/Cas9 in lung cancer. Biology, 15. doi:10.3390/biology15030251 -
NLM
Menezes JM, Mello DC de, Saito KC, Kimura ET, Fuziwara CS. Dual effect of EZH2 gene editing with CRISPR/Cas9 in lung cancer [Internet]. Biology. 2026 ; 15[citado 2026 abr. 02 ] Available from: https://doi.org/10.3390/biology15030251 -
Vancouver
Menezes JM, Mello DC de, Saito KC, Kimura ET, Fuziwara CS. Dual effect of EZH2 gene editing with CRISPR/Cas9 in lung cancer [Internet]. Biology. 2026 ; 15[citado 2026 abr. 02 ] Available from: https://doi.org/10.3390/biology15030251 - Modulation of EZH2 activity induces an antitumoral effect and cell redifferentiation in anaplastic Thyroid Cancer
- Gene editing with CRISPR/Cas methodology and thyroid cancer: where are we?
- Thyroid follicular cell loss of differentiation induced by MicroRNA miR-17-92 cluster is attenuated by CRISPR/Cas9n gene silencing in anaplastic thyroid cancer
- Interplay of TGFβ signaling and microRNA in thyroid cell loss of differentiation and cancer progression
- Modulating gene expression as a strategy to investigate thyroid cancer biology
- Expression of notch and nodal embryonic components in BRAF- and RAS-like thyroid cancer cell lines
- Targeting the highly expressed microRNA miR-146b with CRISPR/Cas9n gene editing system in thyroid cancer
- The highly expressed FAM83F Protein in papillary thyroid cancer exerts a pro-oncogenic role in thyroid follicular cells
- Nucleic acid recovery from thyroid fine-needle cytology slides
- Oncogenic changes in a new transgenic model of BRAFV600E in thyroid
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