Tumor necrosis factor α and interleukin-1β acutely inhibit AgRP neurons in the arcuate nucleus of the hypothalamus (2020)
- Authors:
- USP affiliated authors: FRAZÃO, RENATA - ICB ; DONATO JÚNIOR, JOSÉ - ICB ; CHAVES, FERNANDA MACHADO - ICB ; MANSANO, NAIRA DA SILVA - ICB
- Unidade: ICB
- DOI: 10.3390/ijms21238928
- Subjects: ANATOMIA; FISIOLOGIA; INTERLEUCINA 6; CITOCINAS; INFLAMAÇÃO; OBESIDADE; SEPSE; HIPOTÁLAMO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: International Journal of Molecular Sciences
- ISSN: 1422-0067
- Volume/Número/Paginação/Ano: v. 21, n. 23, art. 8928, 12 p., 2020
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
CHAVES, Fernanda Machado et al. Tumor necrosis factor α and interleukin-1β acutely inhibit AgRP neurons in the arcuate nucleus of the hypothalamus. International Journal of Molecular Sciences, v. 21, n. 23, p. 12 , 2020Tradução . . Disponível em: https://doi.org/10.3390/ijms21238928. Acesso em: 26 dez. 2025. -
APA
Chaves, F. M., Mansano, N. da S., Frazão, R., & Donato Junior, J. (2020). Tumor necrosis factor α and interleukin-1β acutely inhibit AgRP neurons in the arcuate nucleus of the hypothalamus. International Journal of Molecular Sciences, 21( 23), 12 . doi:10.3390/ijms21238928 -
NLM
Chaves FM, Mansano N da S, Frazão R, Donato Junior J. Tumor necrosis factor α and interleukin-1β acutely inhibit AgRP neurons in the arcuate nucleus of the hypothalamus [Internet]. International Journal of Molecular Sciences. 2020 ; 21( 23): 12 .[citado 2025 dez. 26 ] Available from: https://doi.org/10.3390/ijms21238928 -
Vancouver
Chaves FM, Mansano N da S, Frazão R, Donato Junior J. Tumor necrosis factor α and interleukin-1β acutely inhibit AgRP neurons in the arcuate nucleus of the hypothalamus [Internet]. International Journal of Molecular Sciences. 2020 ; 21( 23): 12 .[citado 2025 dez. 26 ] Available from: https://doi.org/10.3390/ijms21238928 - Growth hormone receptor in dopaminergic neurones regulates stress‐induced prolactin release in male mice
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- Interactions between prolactin and kisspeptin to control reproduction
- Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice
- Brain STAT5 signaling and behavioral control
- Fatness rather than leptin sensitivity determines the timing of puberty in female mice
- Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice
- Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice
Informações sobre o DOI: 10.3390/ijms21238928 (Fonte: oaDOI API)
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