Fatness rather than leptin sensitivity determines the timing of puberty in female mice (2016)
- Authors:
- USP affiliated authors: FRAZÃO, RENATA - ICB ; DONATO JÚNIOR, JOSÉ - ICB
- Unidade: ICB
- DOI: 10.1016/j.mce.2015.12.022
- Subjects: ANATOMIA; FISIOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular and Cellular Endocrinology
- ISSN: 1872-8057
- Volume/Número/Paginação/Ano: v. 423, p. 11-21, 2016
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BOHLEN, Tabata Mariz et al. Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, v. 423, p. 11-21, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.12.022. Acesso em: 27 dez. 2025. -
APA
Bohlen, T. M., Silveira, M. A., Zampieri, T. T., Frazão, R., & Donato Junior, J. (2016). Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, 423, 11-21. doi:10.1016/j.mce.2015.12.022 -
NLM
Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022 -
Vancouver
Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022 - Interactions between prolactin and kisspeptin to control reproduction
- Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice
- Brain STAT5 signaling and behavioral control
- Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice
- Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala
- Central regulation of metabolism by growth hormone
- Understanding the role of growth hormone in situations of metabolic stress
- A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system
- Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice
- Effects of growth hormone in the central nervous system
Informações sobre o DOI: 10.1016/j.mce.2015.12.022 (Fonte: oaDOI API)
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