Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice (2015)
- Authors:
- USP affiliated authors: FRAZÃO, RENATA - ICB ; DONATO JÚNIOR, JOSÉ - ICB
- Unidade: ICB
- DOI: 10.1016/j.yhbeh.2015.04.004
- Subjects: ANATOMIA; FISIOLOGIA; CAMUNDONGO; LACTAÇÃO ANIMAL; COMPORTAMENTO MATERNO ANIMAL
- Language: Inglês
- Imprenta:
- Source:
- Título: Hormones and Behavior
- ISSN: 1095-6867
- Volume/Número/Paginação/Ano: v. 71, p. 60-68, 2015
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
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ABNT
BUONFIGLIO, Daniella do Carmo et al. Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice. Hormones and Behavior, v. 71, p. 60-68, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2015.04.004. Acesso em: 03 out. 2024. -
APA
Buonfiglio, D. do C., Lobo, A. M. R., Silveira, M. A., Furigo, I. C., Hennighausen, L., Frazão, R., & Donato Junior, J. (2015). Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice. Hormones and Behavior, 71, 60-68. doi:10.1016/j.yhbeh.2015.04.004 -
NLM
Buonfiglio D do C, Lobo AMR, Silveira MA, Furigo IC, Hennighausen L, Frazão R, Donato Junior J. Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice [Internet]. Hormones and Behavior. 2015 ; 71 60-68.[citado 2024 out. 03 ] Available from: https://doi.org/10.1016/j.yhbeh.2015.04.004 -
Vancouver
Buonfiglio D do C, Lobo AMR, Silveira MA, Furigo IC, Hennighausen L, Frazão R, Donato Junior J. Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice [Internet]. Hormones and Behavior. 2015 ; 71 60-68.[citado 2024 out. 03 ] Available from: https://doi.org/10.1016/j.yhbeh.2015.04.004 - Interactions between prolactin and kisspeptin to control reproduction
- Brain STAT5 signaling and behavioral control
- Fatness rather than leptin sensitivity determines the timing of puberty in female mice
- Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice
- Understanding the role of growth hormone in situations of metabolic stress
- Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala
- Central regulation of metabolism by growth hormone
- A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system
- Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice
- STAT5 signaling in kisspeptin cells regulates the timing of puberty
Informações sobre o DOI: 10.1016/j.yhbeh.2015.04.004 (Fonte: oaDOI API)
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